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Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Skin Anatomy
Epidermis Dermis
Hypodermis
What is a Burn?
An injury to tissue from:
Exposure to flames or hot liquids Contact with hot objects Exposure to caustic chemicals or radiation Contact with an electrical current
Zone of Stasis:
Impairment of blood flow Recovery variable
Zone of Hyperemia:
Prominent vasodilation Usually recovers
Pathophysiology of Burns
Get edema in burned & non-burned skin Burns cause coagulative necrosis
Chemical/Electricity also cause direct injury to cell membranes, in addition to heat transfer
Pathophysiology of Burns
Burns release of inflammatory mediators Increased capillary permeability
Leak proteins into interstitium
Large fluid loss due to fluid shifts & also losses from exposed burned skin Characteristic Ebb and Flow of burns
Ebb: Low metabolism/cardiac output, Temp Flow: hypermetabolism, high cardiac output, hyperglycemia, increased heat produx
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Airway can be an issue with severe burns or inhalational injury (esp. with indoor fire)
Direct injury from heated air/smoke -> edema Edema from inflammatory response to burns Edema from the resuscitation fluids
Give pt oxygen & put on pulse oximetry Progressive hoarseness is a sign of impending airway obstruction Pre-emptively intubate anyone with:
Respiratory distress, inhalational injury, large burns (due to inevitable edema from resusc) Bronchoscopy to help dx inhalational injury
Disability (GCS less than eight -> intubate) Exposure: remove all clothing
Initial Assessment
Burn Resuscitation with Lactated Ringers Figure out burn size by rule of nines or entire palmar surface of pts hand = 1% (palm rule) Parkland formula 4 x Wt(kg) x %TBSA = mL to give in 1 day Half over 1st 8hrs (subtract what was given) Give other Half over next 16 hours In reality, titrate to UOP of 0.5mL/kg/hr in adults and 1mL/kg/hr in children Do not give colloid in first 24 hrs
Severity of a Burn
Depends on: Depth of burn Extent of burn Location of injury Patients age Presence of associated injury or diseases
Depth of a burn
Superficial (1): epidermis (sunburn) Partial-thickness (2): Superficial partial-thickness: papillary dermis Blisters with fluid collection at the interface of the epidermis and dermis. Tissue pink & wet. Deep partial-thickness: reticular dermis Blisters. Tissue molted, dry, decreased sensation. Full-thickness (3): dermis Leathery, firm, insensate. 4th degree: skin, subcutaneous fat, muscle, bone
Third degree
Depth of a Burn
First Degree
Epidermis only Erythematous Hypersensitive Classic sunburn Heals without scar
Depth of a Burn
Second Degree Epidermis + part of dermis Superficial Deep Blisters Edematous and red Very painful Scaring variable
Depth of a Burn
Third Degree Full thickness burn Can involve underlying muscle, tendon, bone Waxy white, leathery brown or charred black Painless Heals with scar
Extent of a Burn
Rule of Nines
Most universal guide for initial estimate Deviates in children due to larger head surface area Palm rule
Robyns Rule of 4s
Management Principles
Stop the Burning Process Universal Precautions Airway Management Breathing Management Circulatory Management Insertion of a Nasogastric Tube Insertion of a Foley Catheter
Management Principles
Relieve Pain Assess Extremity Pulses Regularly Assess for Ventilatory Limitation Provide Emotional Support Suicide Management
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Airway Management
Inhalation Injury
Important determinant of morbidity & mortality Manifests within the first 5 days after injury Present in 20-50% of pts admitted to burn centers Present in 60-70% of pts who die in burn centers
History of Event
Is there a history of unconsciousness? Were there noxious chemicals involved? Did injury occur in closed space?
Colorless, odorless gas Binds to hemoglobin 200 times more than oxygen Most immediate threat to life in survivors with severe inhalation injury Toxicity related directly to percentage of hemoglobin it saturates
Carboxyhemoglobin (%)
0-10 10-30 30-50 50-60 60+
Signs/Symptoms
None Headache Headache, nausea, dizziness, tachycardia CNS dysfunction, coma Death
Signs of CO Poisoning
Cherry red coloration Normal or pale skin with lip coloration Hypoxic with no apparent cyanosis PaO2 is unaffected Essential to determine carboxyhemoglobin levels !
CO Poisoning: Treatment
100% oxygen until carboxyhemoglobin levels less than 15
Increases rate of CO diffusion from 4 hours to 45 minutes
Intubate!!!
Steroids not indicated Prophylactic antibiotics unjustified Circumferential chest burns: escharotomies
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
First half of volume over first 8 hours, second half over following 16 hours
Hypovolemia, decreased CO Increased capillary permeability Crystalloid fluid is keystone, colloid not useful
Monitoring of Resuscitation
Actual volume infused with vary from calculates according to physiologic monitoring Optimal regimen:
minimizes volume & salt loading prevents acute renal failure low incidence of pulmonary & cerebral edema
Monitoring of Resuscitation
Urinary output is a reliable guide to end organ perfusion
Adults: 30-50 ml per hour Children (less than 30 Kg): 1 ml/Kg per hour
Infusion rate should be increased or decreased by 1/3 if u/o falls or exceeds limits by more than 1/3 for 2-3 hours
Monitoring Resuscitation
Blood pressure:
Can be misleading due to progressive edema & vasoconstriction
Heart Rate:
Tachycardia commonly observed
Monitoring Resuscitation
CXR: daily for first 5-7 days
Normal study in first 24 hours does not r/o inhalation injury
ECG:
All electrical injuries Pre-existing cardiovascular disease
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Performing an Escharotomy
Bedside procedure Sterile technique (sharp division or electrocautery) Local anesthesia not required
Control anxiety
Avoid major nerves & vessels Extend incision into subcutaneous fat Incision to be carried across involved joints 2nd incision on contralateral aspect of limb may be required
chemical conjunctivitis
Tar Burns
Contact burns Bitumen is non-toxic Immediate cooling of molten with cold H20 Removal of tar not an emergency Cover with petroleum based product & dressed to emulsify tar
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Electrical Injury
Occurs when electricity is converted to heat as it travels through tissue Divided into:
High voltage greater than 1000 V
Local injury, deep injury, fractures, blunt injuries Risk of rhabdomyolysis, compartment syndrome, cardiac injury
Hands & wrists are common entrance wounds Feet are common exit wounds
Electrical Burns
Extremely difficult to evaluate clinically Greatest tissue damage occurs under and adjacent to contact points, Most significant injury is within deep tissue Edema can compromise circulation Explore & debride necrotic tissue May have to re-explore questionable areas Late complications: cataracts, progressive demyelinating neurologic loss
Lightning Injury
Direct current of >100 000 000 volts and up to 200 000 amps Injury results from:
Direct strike Side flash
Flow of current between person & nearby object
No initial debridement
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Acids
HCl, oxalic, muriatic & sulfuric acids Common in household & swimming pool cleaners
Organic Compounds
Phenols, creosote, petroleum products Contact chemical burns & systemic effects
Chemical Burns
Factors That Determine Severity:
Agent Concentration Volume Duration of contact (delay in treatment) Alkalis generally cause worse damage
Likely to present with swelling & lid spasm Place catheter in lateral sulcus to irrigate
No specific antidote
Acute Tx: copious irrigation with H2O or Zephiran (benzalkonium chloride) Topical calcium gluconate gel or Epsom salts If pain persists, inject 10% Ca gluconate into site Intraarterial and IV infusions with Bier block Hydrofluoric acid: can cause severe hypoCa
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Pediatric Burns
Scald burns most common burn in < 3 years Flame burns most common in children > 3 years Always consider child abuse
Incuffed tube always used Cricothyroidotomy is never indicated Large bore needle placed through cricothyroid membrane may be used in emergency cases
Overview
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
Radiation Injury
Effects reproductive mechanism of certain tissue cells Mature cells suffer less damage Stem cells are more vulnerable to injury Large doses of radiation (> 2000 RAD) may lead to acute mortality
Response
Change in # of leukocytes Severe reduction in leuks, N/V, hair loss, death due to infection
600- 1 000 Destruction of mone marrow, diarrhea, 50% mortality within 1 month
1 000-2 000
2 000+
Beta particles
Positive electrons or negatively charged particles Penetrate approximately 1 cm of tissue
Radiation Burns
Identical in appearance to thermal burns
Treat as you would a non-contaminated burn
Differ from thermal burns from time between exposure and clinical manifestation
SKIN RESPONSE TO RADIATION 200-300 (RADS) 300 1000-2000 2000 Epilation Erythema Transdermal Injury Radionecrosis
Multiple eitiologies
Drugs (penicillins, sulfas, anti-inflammatories) Infection: (staph toxin, HSV, menigococcus, septicemia) Often unknown
TEN Type II
( Stevens-Johnson syndrome) Separation is at the dermal/epidermal junction Adult population High mortality (25-50%)
Treatment:
Limit stimulation of patient V.Fib easily induced Rapid re-warming i9n warm water bath Intubation to administer warm air Central administration of warm Ringers solution
Setting of physical exercise w/o acclimatization Present with temperature>103, no sweating, decreased LOC Tx: rapid cooling until temperature <102 deg If shivering develops, slowly give IV Thorazine DIC frequently reported
Tetanus Immunization
CLINICAL TETANUS-PRONE CLEAN FEATURES WOUNDS WOUNDS Age of wound > 6 Hours <6 Hours Configuration Stellate, avulsion Linear, abrasion Mechanism Missile,crush,heat, Sharp surface cold Signs of Infection Present Absent Devitalized Tissue Present Absent Contaminants Present Absent
Tetanus Immunization
History Of Tetanus Clean Wounds TD1 TIG yes yes yes no no no no no Tetanus-Prone Wounds TD1 TIG yes yes yes no yes yes no no
The End!