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Welcome Otolaryngology Staff Tips and Resources MCQ Pre-Test The ENT History The head and neck exam Important ENT Topics
Otolaryngology Staff
Victoria Dr. D. MacRae - Peds/Otology Dr. M. Husein - Peds Dr. J. Yoo - H&N Dr. K. Fung - H&N Dr. J. Franklin H&N Dr. H. Lampe - H&N Dr. S. Sukerman - General University Dr. L. Parnes - Otology St. Josephs Dr. E. Wright - Rhinosinology Dr. V. Janzen - Rhinosinology Dr. C. Moore - Facial Plastics Dr. R. Ruby - Otology
Goal is to gain exposure to the breadth of ENT, not mastery of every subject Your personal objective should be to gain proficiency with ENT history and exam, and familiarity with a range of Primary Care ENT topics Review your 1st and 2nd year lecture notes, ENT for Primary Care text, emedicine, mdconsult Try to review appropriate lecture notes prior to related clinic/OR
Briefer than the Internal Medicine history (think surgical) Key points (especially important in ENT):
Smoking/alcohol history Numbers of infections (e.g. ear, sinus, throat) in last 1, 2, 3 yrs
Ears: hearing loss, pain, d/c, tinnitus, vertigo Previous ENT surgeries
As with other specialties, the head and neck exam is to be used to supplement clinical information acquired from a detailed history Have an approachbe effective and make sure it can be replicated so as not to miss findings Most of all, practice, practice, practice
You wont know what is normal until you see it many times
Nasal:
Hearing loss
Otalgia
Drugs:
Otorrhea
Ototoxic agents
Tinnitus Vertigo
Family History
Noise exposure
Examples of Abnormalities...
Tympanosclerosis
Osteoma
Otitis Externa
Cholesteatoma
Disease affecting outer/middle ear Disease affecting cochlea or CN VIII Disease involving both middle & inner ear
2.
3.
Response to questions during history Response to a whispered voice Tuning fork air/bone conduction
Weber
No Lateralization
Lateralization to ear with loss Bone conduction better than air conduction (Rinne negative)
Lateralization to better hearing ear Air Conduction better than bone conduction
Rinne
Bone conduction line Air conduction line Air-bone gap = conductive hearing loss Depressed bone conduction line = sensorineural loss
Sinuses
Facial pain Dental pain Hearing loss Post nasal drip Olfaction Congestion Discharge
Sinusitis
Examples - Polyps
Septal Perforation
Nasopharyngeal Carcinoma
Hypopharynx/Larynx
Swallowing:
Dysphagia
Solids vs liquids
Inspect teeth
symmetry, alignment of trachea, fullness, masses, webbing, skin folds, jugular vein distribution, carotid artery prominence
tracheal position, tracheal tug, movt hyoid bone and cartilages with swallowing
The NECK
The Larynx
Indirect laryngoscopy
hold pts tongue wrapped in guaze with one hand hold mirror in other hand against soft palate assess vocal cord mobility, lesions in region posterior pharyngeal wall, posterior cricoid region, piriform recesses vocal cord mobility and appearance arytenoid mucosa/cartilages, aryepiglottic folds epiglottis, valleculae, base of tongue
Direct laryngoscopy
Leukoplakia
Examples - Hypopharynx/Larynx
Foreign Body (Fish Bone)
Vocal Cords
Cyst Papilloma
Nodule Polyp
Leukoplakia
Hypopharynx/Supraglottis
Pyriform Sinus Ca
Epiglottitis
Cranial Nerves
Examine cranial nerves II - XII Consider screening neurological exam in dizzy patients:
Mental Status Cranial Nerves Gross Motor Gross Sensory Reflexes Cerebellar Tests (Rhomberg, finger-to-nose, heel-shin, rapid alternating hand movements)
Otitis Media and Otitis Externa Tinnitus and Hearing Loss Vertigo Facial Paralysis Epistaxis Acute and Chronic Sinusitis Obstructive Sleep Apnea Cancers of the Head and Neck
Otitis Media
Otitis Media
Most common disease diagnosed by clinicians Incidence rapidly increasing each year, almost 90% of kids have at least one bout by their 2nd b-day Presentation: fever, pain, irritability (in kids) also conductive HL, behavioural changes, otorrhea, anorexia, Organisms: Strep. Pneumoniae (40%) Haemophilus influenzae (25%) Moraxella catarrhalis (12%) Risk Factors: day-care, passive smoking, family history, non-breast fed, no vaccine
Otitis Media
Why?
antbx 7-10 day regime vs 6-8 weeks role of tympanostomy tubes +/- adenotonsillectomy (see Bluestone figures)
When to refer
Features of high risk: neonate, immunocompromised state (diabetes, HIV, neutropenia) Symptoms of intracranial pathology:
pain (retroorbital, mastoid), vertigo, SNHL, displaced pinna, cranial nerve 6,7,8
Otitis Externa
Otitis Externa
Presentation: otalgia, fullness, pruritis, hearing loss Etiology: Otitis media, water exposure, canal trauma Organisms: pseudomonas, proteus, Staph, fungal Treatment:
Debridement ototopical agents (Ciprodex, Garasone, Sofracort) 3-7 days PO antibiotics if severe (cellulitis/nodes) analgesics water precautions, pt education
Tinnitus
Tinnitus - DDx
Presbycuisis - age-related sensorineural loss Cardiovascular dz - pulsatile Menieres - assocd w/ episodic vertigo, aural fullness, hearing loss Brain neoplasm - esp CPA tumors Trauma/noise - assocd w/ temporary hearing loss Psychosocial Dz - aural hallucinations, esp. Schiz Drug-induced - ASA most common, usually highpitched, reversible Otosclerosis - otospongiosis of cochlea, labyrinth Multiple Sclerosis
Hearing Loss
Hearing Loss
involves inner ear (i.e. cochlea), acoustic nerve, and/or central auditory pathways
Conductive
External Ear
Sensorineural
congenital atresia cerumen foreign body malformations infections neoplasms congenital effusions (serous OM) acute OM neoplasms otoclerosis TM perforation ossicular discontinuity tympanoscerosis otosclerosis ossicular fixation mastoiditis
congenital acquired
Middle Ear
presbycuisis noise-induced HL head trauma drug toxicity Menieres sudden SNHL tumor perilymphatic leak CNS disease (e.g. MS) labyrinthitis
Vertigo
Vertigo
false perception of movement important Qs: onset, duration, frequency, associated ear symptoms, positional triggers, hx ear dz/head trauma ENT exam, plus Hallpike maneuver, CN+cerebellar testing
Menieres Dz
episodes lasting mins-hrs roaring tinnitus, aural fullness, low-pitched hearing loss
most common cause episodes lasting secs triggered by head movt +/- hx injury, infection
Labyrinthitis/Vestibular Neuronitis
Central
assocd other neuro S+S +/- LOC vascular temporal lobe cerebellar
Facial Paralysis
Recall/review anatomy of the facial nerve; its intra-extracranial components History: onset, duration, rate of progression, recurrence (Bells , MR syndrome) Associated symptoms: numbness middle and lower face, otalgia, hyperacusis, diminished tearing, taste alteration Bells; intense ear pain and vesicular eruption HZ infection Complete Head and Neck exam/ CN assessment, palpation of parotid gland and neck Facial palsy; complete vs incomplete, segmental vs uniform involvt, unilateral vs bilateral (<1%)
CBC with diff and ESR Serum antibody tests; serum ANA and RF Electrophysiologic tests
nerve excitability test (NET) maximal stimulation test (MST) Electroneurography (ENoG) Electromyography (EMG)
Rapid onset palsy, minimal assoc symptoms, spontaneous recovery 1/3 pts develop only paresis, 95% total recovery 2/3 complete paralysis, facial tone/movt 85% in 3 wks; expect 3-6 months The longer the delay in recovery, the greater the liklihood of adverse sequelae ? HSV evidence for etiology
Treatment must be initiated promptly for maximal efficiency delay of > 3 days decreases efficiency Medical tx:
Prednisone 1mg/kg/day for 7-10 days Acyclovir 400mg po 5 times daily for 7 days
Surgical Tx:
Decompression (>90% degen on ENoG w/in 14 days onset + no voluntary motor unit potentials EMG)
Eye Care
avoid vents, liberal use of ophthalmic lubricants, shielded glasses Potential gold weight implants, canthoplasty, tarsorrhaphy for long term
Epistaxis
Epistaxis
Very common 60% incidence through ones life 10% seek medical attention; 6-10% ENT consult
Seasonal incidence Winter > Summer POTENTIALLY LIFE THREATENING Etiology consider local and systemic factors Site of bleed Anterior 90% Posterior 10%
History: side, duration, amount, temporal pattern, trauma PMHx: liver disease, coag d/o, family hx, HTN, previous epistaxis, nutrition Medications: ASA, NSAIDS, warfarin, heparin, chloramphenicol, dipyridamole Examination: - ABCs and vitals (orthostatics) - General exam (purpura, petechiae) - Nasal exam (head light, suction, decongest, determine bleeding site)
Reassure patient IV hydration depending on extent of bleed control HTN Bloodwork CBC, INR/PTT, Group and Cross Treatment - depends on etiology - those with systemic factors, conservative, noncauterizing, cartilage-sparing techniques for initial therapy correct coags, d/c meds
Anterior : localize bleed - silver-nitrate cautery - surgicel/oxycel (cellulose),gelfoam (gelatin) - anterior packing (merocel vs impreg guaze) - PO antibiotics with packing (TSS) Posterior: difficult to see etiology - posterior packing (foley/rockets/formal pack) - embolization - IMAX , ethmoid ligation - endoscopic cauterization Consider ENT referral if posterior pack required
Sinusitis
Sinusitis
Inflammation of mucosal lining of the sinuses Pathophysiology: patency of ostia function of cilia quality of nasal secretions Predisposing factors: local, regional, systemic Be aware of complications very serious GET CULTURE for diagnosis Treat for at least 10 days 3 weeks to prevent relapse
Sinusitis - Classification
Rhinosinusitis classified according to 5 axes: clinical presentation: acute, subacute, chronic sinus involved: ethmoids, maxillary, frontal, sphenoidpansinusitis causative organism: bacterial, viral, fungal, protozoan presence of complication: extrasinus extension modifying or aggravating factors: immunosuppression, diabetes, malnutrition, NG tube, IgG deficiency
Sinusitis - bacterial
Acute lasts 1 day 4 weeks - management antbx for at least 7 days post-sx - surgery rarely necessary complications Subacute lingers 4 weeks 3 months - inflammation still reversible med. managet Chronic - persisted disease > 3 months - generally irreversible damage to sinus drainage - surgical managet
Sinusitis
Viral sinusitis:
follows viral URI damage cilia from cilia ciliotoxins predisposes to bacterial sinusitis noninvasive (mycetoma, AFS) invasive ( fulminant FS, indolent) orbital intracranial need aggressive medical AND surgical tx
Fungal sinusitis:
Complications of sinusitis
repeated reductions/cessations in airflow, w/ apnea index >=5, respiratory disturbance index (RDI) of at least 10 on polysomnograph central apnea: absence of airflow assocd w/ lack of inspiratory effort Snoring: 28% of women, 44% of men aged 30-60 OSA: 9% of women, 24% of men (RDIs of 5 or higher)
OSA: Pathophysiology
tongue contacts the soft palate and posterior pharyngeal wall in the presence of lateral collapse of the pharynx, thus generating occlusion risk factors: obesity, redundant tissue in the neck, retrognathia, craniofacial anomalies Alcohol and other sedating medications may contribute
OSA Management
Conservative Measures: weight loss, avoid sedatives, sleep on side Continuous Positive Airway Pressure (CPAP) Oral Appliance Surgery in select patients: Uvulopalatopharyngoplasty, septoplasty
6-8 % of all malignancies in the body historically M>F but ing in women due to smoking 90% Squamous Cell Ca
Nose/Sinuses: asian descent, hardwood dust, nickel, chromium Lip: UV exposure, poor oral hygiene, smoking/EtOH Salivary Gland: smaller gland, risk malignant Oral Cavity: smoking, EtOH, poor oral hygiene, chronic dental irritation, betel nut chewing Pharynx: smoking, EtOH Thyroid: family history, radiation exposure
Peritonsillar Abscess
Common complication of tonsillitis in adolescents and young adults Symptoms: trismus, painful swelling in throat, dysphagia, odynophagia, fever, otalgia, hot potato voice Classic findings:
unilateral swelling peritonsillar region with bulging soft palate Deviation of midline of palate and uvula to contralateral side
Peritonsillar abscess
Management:
Clindamycin 300mg QID x 7 days + analgesics Needle aspiration and I&D (effective >90%) - risk of recurrence 10-15% - pts younger than 40 yrs with hx of recurrent tonsillitis @ greatest risk >2 bouts of peritonsillar abscess candidate for tonsillectomy Inability to swallow fluids, poor airway, immunosuppression, young patients may be factors for admission Tonsillectomy for some surgeons
Can present as a life-threatening hypoxemia and hypercapnia First priority is to establish airway; dont forget about the nasopharyngeal a/w Signs: inspiratory stridor (decreased intraluminal pressure compared to atmospheric pressure Bernouille principle Most important step in initial evaluation is determining whether an airway needs to be established immediately
Considerations
? Immediate a/w ? Cancer in upper a/w ? Infection ? site RLN injury VC paralysis Post. Glottic closure or subglottic scar tissue angioedema Obstn @ glottic level
Main points in hx: timing, age, PMHx, other systemic d/o, ability to sleep lying down Physical exam: pt may need antihistamines, epinephrine, steroids, antbx during dx evaluation Pulse oximetry demonstrates end-point obstn, no info during progression Hypercapnia, acidosis early signs of hypoventilation Agitation, cyanosis, resp effort on inspection Nasal flaring, neck retractions, accessory muscle use signs of fatigue; listen to chest for symmetry/noises
Complete head and neck exam: nose, oral cavity, larynx highlight exam Radiology: may not be time for soft tissue lateral views, generally not great aid to dx
MISI BOVO Malignant tumours (SCC, Adenoid cystic of trachea, thyroid Ca) Infections (Epiglottitis, supraglottitis, Tracheitis, cellulitis FOM
Lugwigs, Retropharyngeal abscess)
Subglottic stenosis (hemangioma, intubation) Inflammatory (GERD larygospasm, Angioedema) Benign tumours (recurrent papillomas, chondromas, lipomas,
fibromas) Body (Foreign)
Other Vocal Cord lesions (polyps, glottic webs) Vocal cord paralysis (recurrent nerve injury, systemic neurologic
disorder, idiopathic)
Angioedema
Presentation: acute painless mucosal edema - face, lips, tongue, larynx - airway obstruction 20% Etiology ACE Inhibitor sensitivity most common - see chart Treatment aggressive - high humidity oxygen, epinephrine, antihistamines, steroids - secure airway (observe, ET Tube, tracheotomy) - D/C ACE inhibitors and Med consult (HTN)
Blunt and penetrating trauma MVA, fall Three types : longitudinal, transverse, mixed Longitudinal: most common 70-80% - facial nerve injury 10-20% - ruptured TM, hemotympanum, CSF leak - persistent conductive HL (ossicular chain) Transverse: # usually involves bony labyrinth - profound SNHL - facial nerve injury (~ 50%) - CSF otorrhea/rhinorrhea, meningitis
Management: - trauma protocol ABCs, C-spine - Ear exam - Assess facial nerve early (immediate vs delayed) - Assess hearing Audiogram, tuning forks - Radiology Head CT (brain injury) + CT temporal bone windows
Treatment: immediate facial nerve paralysis OR to repair delayed FN paralysis observe, steroids, eye protection CSF leak conservative bed rest, >90% resolve in two weeks SNHL hearing aid conductive HL ossicular reconstruction vertigo tx symptomatically, Serc, Meclizine, PT
Nasal Fracture
Very common; most common facial fracture High index of suspicion for fracture - mechanism, appearance, epistaxis, obstruction Examine entire face (nose, orbit, zygoma, mandible) - instability, mobility, crepitation - septal hematoma, lacerations Facial x-rays variable reliability CT face indicated if other fractures present ENT REFERRAL - < 5 days for closed reduction - > 12 days for septorhinoplasty
Hearing Loss
sudden, usually unilateral no trauma history rapidly progressive (<3 days) Etiology Uncertain - Viral (30-50% assoc viral URTI) - see chart Associated Symptoms Aural fullness, tinnitus, vertigo
Sudden SNHL
Diagnostics: 90% no etiology found - normal P/E - Audiogram, ABR, Otoacoustic emission - Lab tests (see chart) - possible MRI with gadolinium (1-3% AN) Management: 2/3 recover spontaneously - Antiinflammatory steroids - vasodilators carbogen, histamine, papaverine - rheologic agents LMW dextrans, heparin - antivirals/diuretics/triiodobenzoic acid deriv - surgery Bottom line: EARLY REFERRAL