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Review ocular emergencies
Trauma Non-traumatic
One estimate in 2000: more than 7500 hospitalizations for the treatment of pediatric eye injuries that resulted in more than $88 million in inpatient charges
Second leading cause of monocular vision loss Second leading cause of ocular surgery in children
In some studies, Up to 60% of pediatric eye injuries occur during sports and recreational events Other studies show that the home has become the more common place for pediatric eye injuries Special consideration: children under age 5
Trauma or treatment can lead to visual deprivation from amblyopia
History
Difficult to examine kids History! History! History!
Mechanism of injury
Blunt trauma Penetrating trauma Associated head injury Chemical exposure Animal bites Foreign body??? Abuse???
History
Coexisting systemic disease Allergy Tetanus status
Examination
First goal: Rule out open globe
Signs: Chemosis; Peaked pupil; Vitreous hemorrhage
Vision!!!
One eye at a time
Red reflex
Lacerations
Eye Lid Lacerations
Lacerations
History: Mechanism of injury
Foreign body possible? Animal bite or scratch?
Referral to ophthalmologist
Lacerations
Subconjunctival Hemorrhage
Bruise of the eye Painless No vision change Mechanism of injury
Trauma Sudden increase of pressure in the chest (coughing, sneezing, vomiting)
Subconjunctival Hemorrhage
If moderate or severe, and other signs of trauma (eyelid bruising, worrisome history)
Concern for more extensive eye injury Refer to ophthalmology or ER
If vision ok & rest of eye looks normal - probably no other ocular injury
If vision loss, irregular pupil & swollen conjunctiva possible globe rupture
Corneal Abrasion
Scratch on the cornea and/or conjunctiva
Fingers, fingernails, blowing debris, paper, contact lens wear, chemical exposure
History (OUCH)
Pain/ foreign body sensation Copius tearing Blepharospasm or inability to open eye
Corneal Abrasion
Exam
Watery discharge Conjunctival injection Topical anesthetic will relieve pain
Rough edge of abraded cornea
Corneal Abrasion
is a defect in the epithelial layer only Typically the rest of the cornea is not damaged VERY painful because of so many nerve endings in the cornea
Corneal Abrasion
Area of corneal defect stains with fluorescein and lights up with cobalt blue light.
Corneal Abrasion
Linear Abrasion
Look for foreign body under eyelid.
Corneal Abrasion
Management
One drop of topical anesthetic
NEVER give a bottle of topical anesthetic drops
Commonly include dust, dirt or metallic slivers Metallic foreign bodies can leave a rust ring in the cornea History
Hammering or working with tools Pain/foreign body sensation Tearing
Exam
Management
Can remove with aid of topical anesthetic and cotton tip applicator May need slit lamp and needle or burr Referral to ophthalmologist
Open Globe
Perforating injury of the cornea and/or sclera which violates the integrity of the globe History: mechanism of injury
Blunt trauma Penetrating trauma Intra-Ocular Foreign body possible
Open Globe
16 YO, 22 caliber shotgun casing, penetrated thru lid and into eye
Open Globe
Exam:
Chemosis Peaked pupil Prolapse of iris or uveal tissue Hyphema Vitreous hemorrhage
Open Globe
Management
Call ophthalmologist Place fox shield, protective shield or Styrofoam cup over eye IV antibiotics broad spectrum coverage Consider CT scan if foreign body suspected Do NOT let the patient eat or drink!!!
Chemical Burn
True ophthalmic emergency!!! From alkaline or acidic substance in contact with eye May be in the form or:
Liquid Solid Powder Mist Vapor
Chemical Burn
Begin Irrigating with normal saline!! History
Alkaline worse than acid Contact with household solutions/cleaners Pain Tearing
Chemical Burn
Severity of the injury depends on
the pH
Alkaline agents (high pH) much more damaging than acidic Denatures proteins and lyses cell membranes
Chemical Burn
Treatment
If history of chemical injury, defer exam and begin STAT lavage with normal saline COPIOUSLY irrigate Continue until neutral pH pH can be tested with litmus paper Prognosis is dependent on number of clock hours of avascular cornea and scleral whitening
Acidic Injury
Corneal abrasion
Alkaline Injury
Damage to epithelium
Alkaline Injury
Hyphema
Blood in the anterior chamber Caused by blunt force trauma
External compression causes tearing of the iris root
Hyphema
Management
Evaluation for associated ocular problems Referral to ophthalmologist
Eye SHIELD (not patch) Elevate head of bed Bed rest for 5 days NO NSAIDS Tylenol OK for comfort May require hospitalization for large hyphema +/- Cycloplegic and topical steroids Watch for increased IOP
Hyphema
Special considerations
Rebleeding possible
First 2-5 days Associated with fibrinolysis May be worse than initial bleed
Hyphema
Special considerations
Spontaneous hyphema no history of trauma Juvenile Xanthogranuloma (JXG)
Children under 2 Red/brown papules
Traumatic Iritis
History
Blunt Trauma Pain, photophobia usually 1 to 2 days after injury
Exam
Slit Lamp Exam shows cell and flare in anterior chamber Perilimbal conjunctival injection, photophobia
Treatment
Refer to Ophthalmology Cycloplegia Steroids Sunglasses
Orbital Fracture
History
Blunt force trauma ball, fist Nausea and vomiting Double vision
Exam
May have periocular edema and ecchymosis Enophthalmos Inability to look up
Orbital Fracture
Management
CT scan better than MRI Referral to orbital surgeon
Orbit
Leukocoria
Literally means white pupil Many disorders can cause leukocoria and all represent a serious threat to vision. Any patient with Leukocoria should be referred to Ophthalmology immediately.
Retinoblastoma
Retinoblastoma
Malignant tumor of the eye originating from the retina Unilateral or bilateral Nearly 100% mortality when untreated, but >95% survival with prompt treatment Suspect with any of the following:
Abnormal red reflex/leukocoria Strabismus Chronic red eye ( conjunctivitis clears in 10 to 14 days)
Congenital Cataract
Congenital Cataract
Opacity in the lens of the eye May be unilateral or bilateral
Inherited (Usually Autosomal Dominant) Metabolic Infectious Traumatic
Requires removal in eyes at risk for amblyopia Urgent referral to Ophthalmology in newborns
Risk of irreversible amblyopia if not removed by 4 to 6 weeks of age
Congenital Glaucoma
Congenital Glaucoma
Elevated intraocular pressure Signs:
Cloudy cornea
Congenital Glaucoma
Bupthalmos or big eye (big eyes or corneal enlargement = rule out glaucoma) Tearing, photophobia and blepharospasm
Corneal Ulcer
Vision and eye threating condition History
Contact lens wear, esp. SLEEPING in contact lenses Red, painful eye
Cellulitis
Infection or inflammation of the eyelid skin and/or orbit The orbital septum is a fibrous membrane that separates the eyelid skin from the deeper structures of the orbit Preseptal cellulitis effects the skin and does not extend beyond the septum into the orbit Orbital cellulitis is infection within the orbit itself, often from sinus disease
Cellulitis
Preseptal cellulitis can often be managed with oral antibiotics and close follow up Orbital cellulitis is much more serious (Ophthalmology involved) Signs:
Proptosis (versus lid swelling) Red, injected eye Limitation of ocular motility Decreased vision (can be signs of optic nerve compromise)
Cellulitis
Treatment
IV Antibiotics CT scan to rule out sinus disease/abscess Management with Ophthalmology to monitor signs of compromise, infectious disease and ENT if sinus disease/abscess
Thank You