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Insomnia Narcolepsy Obstructive sleep apnea Restless legs syndrome Klein-Levin Syndrome* Post-traumatic hypersomnia
*recurring periods of excessive amounts of sleeping and eating. At the onset of an episode the patient becomes drowsy and sleeps for most of the day and night (hypersomnolence), waking only to eat or go to the bathroom.
Night terrors Bruxism (tooth grinding) Sleepwalking (somnambulism) Sleeptalking (somniloquy) Exploding head syndrome!
Commentary - Watson
et al 2006 - 50% risk of having insomnia due to genetics. Diathesis-stress model environmental stressors may trigger the disorder Perpetuating factors being tense before going to bed because of previous sleep problems
Sleep Apnoea and other parasomnias (e.g bruxism, sleep walking)- disrupt sleep and increase
likelihood of insomnia
Research Complications
Synoptic point Chronic insomnia highly complex Lots of causes of insomnia stress, depression, poor sleep hygiene, age, gender e.t.c Unlikely to be explained by one factor Therefore - Difcult to draw conclusions
Attribution Theory
One cause of Primary Insomnia is a persons belief that they are going to have diculty sleeping. Self fulfilling tense before sleep Attribute sleep problems to insomnia Treatment Train them to be convinced the source of problem lies elsewhere Storms and Nisbett 1970 insomniacs given a pill half told it would stimulate them and the other half it would sedate them. Those expecting arousal went to sleep faster because they attributed their arousal to the pill and actually relaxed!!
Now drink some caffeine!! Wait 15mins and try again!! What do you predict will happen?
Treatment of Insomnia
Read p 17 Create a problem page to give advice on why they may be suffering from insomnia and how to help it.
HLA - linked to mutation of the immune system - more of one type of HLA
found in narcoleptic patients. Not found in all narcoleptics and also common in general population so cant be only explanation
affecting 20% children and less than 3% adults. Only occurs during NREM/SWS and related to night terrors. Sleep walker not conscious and later has no memory of events during sleep walking
arousal incomplete. Likely that this is genetic. Diathesis - stress model ts. 50% concordance in MZ twins, 10x more likely to SW if rst degree relatives have disorder.
conditions, hormonal changes during puberty and menstruation have all been shown to increase likelihood of sleep walking - the stress part of the Diathesis-stress model
they have more SWS than adults. Oliviero (2008) suggests that the system that inhibits motor activity in SWS is underdeveloped in some children and adults - evident in sleep walkers.
In groups, you will choose one category of disorder to research. You must produce a poster on the category, giving detailed examples, explanations and treatments. You must consider behavioural / psychotherapeutic treatments, management and drug treatments. AS WELL AS A POSTER, YOU MUST ALSO PREPARE AND PRESENT A 5 MINUTE POWERPOINT FOR THE REST OF THE GROUP ON THE CATEGORY YOU CHOSE
Homework
Finish presentation and/or Question 6 p19 600 word essay Outline and evaluate explanations of two or more sleep disorders The narcolepsy powerpoint on the blog is particularly good for essay prep (not done by me. Mores the pity!) Produce some revision materials for the sleep topic e.g. cards, mind maps, notes, game, quiz bring in on Friday
Outline and evaluate explanations of two or more sleep disorders You will be divided into pairs/3s
We will then put these paragraphs together to make a super essay!!!
Pair 1 will prepare an outline of Narcolepsy Pair 2 will prepare an evaluation of Narcolepsy Pair 3 will prepare an outline on sleep walking Pair 4 will prepare an evaluation of sleep walking Pair 5 will prepare synoptic points relating to sleep disorders Pair 6 will prepare an outline of another disorder 23 e.g. Obstructive Sleep Apnoea or Night Terrors