Professional Documents
Culture Documents
I.
di"retics, digitalis
VII.
II.
1. PERICARDITIS
III.
Occurs when much pressure is generated within the left ventricle to be generated to the aorta resulting to backflow of blood to the left atrium &ress"re is re#lected back to the p"lmonary veins and arteries Manifestations: asymptomatic "ntil cardiac o"tp"t #alls, m"rm"rs, atrial #ibrillation, p"lmonary mani#estations Management: restrict physical activities, restrict sodi"m, di"retics, digitalis
ac"te or chronic in#lammation o# the pericardi"m Assessment: precordial pain pain (inspiration, co"ghing ) s'allo'ing pain 'orse 'hen s"pine pericardial #riction r"b #ever ) chills elevated *(! ct+ Management: &osition: side lying, high ,o'ler-s, "pright ) leaning #or'ard Admin+ analgesic, corticosteroids, $.AID-s Avoid aspirin ) anticoag"lants Antibiotics Di"retics ) digoxin /onitor #or complications: !ardiac Tamponade
2. MYOCARDITIS
IV.
AORTIC STENOSIS
Caused by calcification of the valve and stiffening of the aortic valve Manifestations: initially asymptomatic, angina pectoris, syncope, dyspnea Management: avoid vigoro"s physical activity, antibiotics, digitalis, beta-blockers
V.
AORTIC REGURGITATION
Blood propelled into the aorta propels back to the left ventricle through an incompetent valve Manifestations: initially asymptomatic, palpitations, m"rm"rs, lo' (& Management: same #or aortic stenosis
ac"te 0 chronic in#lammation o# the myocardi"m Etiology: (acterial : staphylococc"s 0 pne"mococcal 1iral : coxsackievir"s 0 m"mps 0 in#l"enza &arasitic : Toxoplasmosis 2adiation 0 3ead /eds: 3ithi"m 0 !ocaine Assessment: #ever pericardial #riction r"b gallop rhythm m"rm"r p"ls"s alternans (reg"lar alternation o# 'eak and strong beats 'itho"t changes in cycle length .0. o# 4, !hest pain Management: &osition: (ed rest 0 sitting "p or leaning #or'ard /onitor p"lse rate ) rhythm Admin+ #or #ever ) pain 3imit activities Admin+ digoxin 0 antidysrhythmics 0 antibiotics as prescribed $.AID-s 0 analgesics 0 salicylates
3. ENDOCARDITIS
VI.
In#lammation # the inner lining o# the heart Assessment: #ever, anorexia, 't loss, #atig"e cardiac m"rm"rs 5ane'ay-s lesions 6ssler-s nodes &etechiae, splinter hemorrhages in nailbeds .plenomegaly Management balance activity '0 rest
antiembolism stockings monitor emboli: o Splenic s"dden abd+ pain radiating to 3 sho"lder 0 rebo"nd tenderness on palpation o Renal #lank pain radiating to groin, hemat"ria poly"ria
'ypertension )nfections
1) DILATED CARDIOMYOPATHY
@s"ally both ventricles dilate, myocardial #ibers degenerate and replaced by #ibrotic tiss"e Associated 'ith in#ections, metabolic problems, ne"rom"sc"lar problems, toxins, pregnancy, connective tiss"e disorders and genetic predisposition similar in heart #ail"re 2est and avoid stress 2estrict sodi"m in diet Di"retics, nitroglycerin, anticoag"lants, antidysrhythmics as ordered !ardiac de#ibrillation Avoid alcohol 4eart transplant
4. RHEUMATIC FEVER/RHD
&ericarditis that #ollo's expos"re o# child to throat and skin in#ection ca"sed by 7ro"p A (-hemolytic organisms 82epeated bo"ts 'ith permanent scarring o# the valves 24D heart failure
Management:
Jones Criteria /a9or /inor TERMS !arditis &olyarthritis !horea :rythema marginat"m ."bc"taneo"s nod"les ,ever Arthralgia :levated :rythrocyte .edimentation 2ate &ositive ! 2eactive &rotein &rolonged &-2 interval Chorea
2) HYPERTROPHIC CARDIOMYOPATHY
$isproportionate thickening of the myocardium that leads to obstruction of blood flow Causes* o genetically transmitted, idiopathic Manifestations* may lead to death ;asymptomatic Management: medications as ordered, avoid alcohol 8."rgery: myotomy
3) RESTRICTIVE CARDIOMYOPATHY
!a"sed by #ibrosis and thickening in the heart that ca"ses the ventricles to lose their ability to stretch /anagement: no speci#ic interventions b"t goals are aimed to diminish heart #ail"re
aneway!s lesion non-tender, small erythemato"s or hemorrhagic mac"les or nod"les in the palms or soles, 'hich are pathognomonic o# in#ective endocarditis Ossler!s nodes pain#"l, red, raised lesions on the #inger p"lps, indicative o# the heart disease s"bac"te bacterial endocarditis+ "rythema marginatum pink rings on the tr"nk and inner s"r#aces o# the arms and legs C Reactive #rotein a plasma protein that increases d"ring in#lammation
Management of RF/RHD Aspirin or steroid Initially &enicillin #or ;< days .econdary &rophylaxis: &enad"r = >-? 'eeks &henobarbital or haloperidol #or chorea Digoxin #or heart #ail"re Di"retics #or heart #ail"re 2est, ade="ate #eeding and #l"id balance
CARDIOMYOPATHY
Unkno'n ca"se > ma9or types $ilated %congestive& cardiomyopathy 'ypertrophic cardiomyopathy Restrictive cardiomyopathy 2isk #actors (lcohol abuse #regnancy
Surgical Management
a.
,.
A"-$&$#" )*
o
."bsternal or &recordial pain choking, heartb"rn, pressing, b"rning, s="eezing+ 2adiating to the le#t sho"lder and "pper arm and may travel do'n to the elbo', 'rist and #ingers 6evine!s sign Aggravated by activity 2elieved by rest
#.
0.
VASODILATORS
n angi +(a!"*
:##ects: $irect rela3ing effect on vascular smooth musle, res"lting in vasodilation o isosorbide dinitrate %)sordil& 9 nitroglycerin o transdermal nitrodisk %patch& 9 1itrol: 1itrobid %Ointment&: (myl 1itrate,
.ETA6 .LOC7ERS
ANGINA PECTORIS
chest pain res"lting #rom myocardial ischemia; a symptom o# an existing disease; no necrosis Transient chest pain ca"sed by )1SU55)C)"1T B6OO$ 56O7 to the myocardium resulting in myocardial ischemia,
:##ects: $ecrease myocardial o3ygen demand by decreasing heart rate: B#: myocardial contractility and calcium output o (tenolol %Tenormin& o #ropanolol %)nderal& o Metoprolol %6opressor& o 1adolol %Corgard& o #indolol %;isken& o "smolol %Brevi2bloc& o Medication*
Types of Angina 1. S"a,($ o triggered by a &2:DI!TA(3: degree o# exertion or emotion (eg+ *alking A< #eet 2. Un!"a,($ o triggered by an @$&2:DI!TA(3: degree o# exertion or emotion 3. P&in2)$"a(/3a&ian" o similar to classic angina b"t longer and /AF 6!!@2 AT 2:.T 4. In"&a#"a,($ o "nresponsive to treatment 4. N #"'&na( o associated 'ith 2:/ sleep d"ring dreaming 6. Angina 0$#',i"'! o occ"rs 'hen the client reclines and lessens 'hen the client sits or stands "p 5. P !"6in/a&#"i n o occ"rs a#ter /I Causes: E!s "3ertion "motion "3posure to cold "3cessive smoking "3cessive eating Assessment: &ain pattern: /ild % moderate
:##ects: o o o
ANTICOAGULANTS
G Heparin Sodium o Inactivates thrombin and other clotting #actors inhibiting conversion o# #ibrinogen #ibrin, there#ore #ibrin clot #ormation is prevented+ Warfarin Sodium Sodium ; !icumarol o Inhibit hepatic synthesis o# 1it+H
1URS)1- )1T"R;"1T)O1S )n $RU- T'"R(#> 1itroglycerine Therapy o (ssume sitting or supine position 'hen taking the dr"g+ o Take a maxim"m o# 0 doses at ? minute interval o -radual change of position o 6##er SO7%sip of water& before giving S6 nitrates o Instr"ct client to avoid drinking alcohol: smoke
6bserve #or .:: 'eadache: flushed face: di//iness: faintness and tachycardia,
Beta2 (drenergic Blockers o (ssess pulse rate be#ore administration o# the dr"g o Administer 'ith #ood o $o not administer #RO#(1O6O6 to clients with asthma and hypoglycemia o 7ive extreme ca"tion in clients 'ith heart #ail"re o 6bserve #or .:: nausea: vomiting: mental depression: mild diarrhea: fatigue and impotence Calcium2Channel Blockers o (ssess 'R and B# o /onitor 'epatic and Renal 5unction o (dminister + hour ac or . hrs pc, (fter meals, #latelet (ggregation )nhibitors o Assess #or signs and symptoms of bleeding o $o not give (S( with coumadin o Observe for (S( to3icity2 T)11)TUS 'eparin Sodium o Assess #or signs and symptoms of bleeding o Antidote: #ROT(M)1" SU65(T" o I# administered .I+, $o not aspirate: and do not massage o Monitor #TT or (#TT levels o @sed #or a ma3imum of . weeks Coumadin o (ssess for signs and symptoms of bleeding o Antidote: ;)T,@ o Monitor #T o Minimi/e green leafy vegetables in the diet, 1ote* .2? minutes without O3ygen supply %brain& causes death 0?2A? minutes without O3ygen supply %heart& causes death
Collaborative Management for Myocardial )nfarction %oals of T&eatment G #revention of further tissue damage an" limitation of infarct si/e G Ma3imi/e myocardial tissue perfusion an" reduce myocardial tissue demands a+ Ac"te .tage: Admin+ prescribed medications : M& '& (& A& G Morphine Sulfate G ')y$en #A*( G (itrates G Aspirin +idocaine (Jylocaine ,eta-.loc/ers (propranolol, timolol 0Throm.olytics (streptokinase, "rokinase Anticoa$ulants (heparin, 'ar#arin0co"madin 'u&sing Management: &romoting Tiss"e 6xygenation and Tiss"e &er#"sion &romoting Ade="ate !ardiac 6"tp"t &romoting !om#ort &romoting rest &romoting Activity &romoting $"trition and :limination &romoting 2elie# o# Anxiety and *ell- (eing ,acilitating 3earning Teaching and !o"nseling Re(a)ilitation *Dis#(a&ge afte& MI+: ;+ Discontin"e smoking A+ Diet >+ *eight 2ed"ction ?+ &rogressive exercise B+ /aintenance /edication C+ 2es"mption o# sex"al activity is 1-2 wee/s #rom discharge D+ .tress /anagement Techni="es Tea#(ing %ui"elines in Resuming Se,ual A#ti-ity: o 2es"me i# able to climb A #lights o# stairs o (e#ore: rest is impt+ 0 avoid large meals 0 'ear loose #itting clothes 0 nitro be#ore sex 0 "s"al environment 0 sex at room temperat"re 0 #oreplay o D"ring: com#ortable position 0 sel#-stim"lation 0 oralgenital 0 avoid anal o 3emale position: side lying or rear entry position o Male #osition : reverse missionary or sitting position
MYOCARDIAL INFARCTION
."dden decrease o# oxygenation d"e to red"ced coronary blood #lo' that res"lts to destr"ction o# myocardial tiss"e in regions o# the heart+ Causes: o Thromb"s o :mboli o Atherosclerosis $o#ation: 6eft anterior descending artery % anterior or septal 'all /I or both Circumfle3 artery% posterior 'all /I or lateral 'all /I Right coronary artery % in#erior 'all /I o Three areas 'hich develop in /I: a+ T wa"e in"ersion (zone o# hypoxia b+ ST ele"ation (zone o# in9"ry c+ #atholo$ic % wa"e (zone o# in#arction $iagnostic Studies +, Total C@ levels ., 6$' 0, (ST A, "CAssessment &ain pattern: o .evere, prolonged cr"shing s"bsternal pain; kni#elike that may radiate to 9a', back ) le#t arm o 3:1I$:-. .I7$ ,ever 6lig"ria Anxiety and Apprehension &allor 0 cyanosis 0 coolness o# ext+0 *eak p"lse Ac"te &"lmonary :dema :levated !H-/(, elavated 3D4, A.T
CARDIAC DYSRHYTHMIAS
Abnormal heart rhythms o#ten detected beca"se o# associated mani#estations o# dizziness, palpitations, and syncope ETIOLOGY Dist"rbance in the > ma9or mechanisms: o Automaticity 6 .in"s Tachycardia 6 .in"s (radycardia 6 &remat"re Atrial !ontraction 6 &remat"re 1entric"lar !ontraction 6 Torsades de &ointes o 6 Conduction ,irst Degree A1 block
6 6 o 6 6 6
.econd Degree A1 block Third Degree A1 block Reentry of impulses Atrial ,l"tter Atrial ,ibrillation 1entric"lar ,ibrillation
Management: 6 !ardioversion, 6 anti-dysrhythmics (digitalis, ="inidine, etc , 6 !alci"m !hannel (lockers, 6 (eta adrenergic blockers ATRIAL FI.RILLATION 6 2apid, chaotic atrial depolarization #rom a reentry disorder 6 ABB2DBB bpm 6 A1 node is bombarded 'ith more imp"lses than it can cond"ct Management: 6 Antidysrhythmics, 6 beta-blockers, 6 De#ibrillation
Clinical Manifestations &alpitations .yncope &allor Diaphoresis Altered mentation 4ypotension .l"ggish !2T Decreased "rine o"tp"t :!7 changes
SINUS DYSRHYTHMIAS
o o SINUS TACHYCARDIA 2apid, reg"lar rhythm at a rate of +BB2+CB bpm 6cc"rs in response to : 6 increased sympathetic stimulation or decreased parasympathetic stimulation, 6 medications (eg. At&opine/ nit&ates/ epinep(&ine+, 6 stress, 6 ca##eine, 6 nicotine, 6 hyperthyroidism, 6 hypercalcemia, 6 #ever, 6 heart #ail"re, 6 #l"id vol"me loss
VENTRICULAR DYSRHYTHMIAS
o o o o &remat"re 1entric"lar !ontraction 1entric"lar Tachycradia 1entric"lar ,ibrillation 1entric"lar Asystole
PREMATURE VENTRICULAR CONTRACTION 6 &remat"re ventric"lar beats associated 'ith /I, acidosis, alcohol, heart #ail"re, !AD, nicotine, hypermetabolic states 6 a cardiac irreg"larity in 'hich the "entricled contracts .efore its anticipated time& 6 Dangero"s Management: 6 lidocaine 6 other antidysrhythmics TORSADES DE POINTES 6 ,orm o# ventric"lar tachycardia in 'hich the %RS comple)es appear to .e constantly chan$in$ 6 Usually results from dru$ to)icity or electrolyte im.alance 6 *t is an emer$ency Management: 6 3idocaine, 6 De#ibrillation, 6 !ardioversion
o Management: 6 aimed at treating the "nderlying ca"se, bed rest, oxygen, 8meds as ordered SINUS .RADYCARDIA o 8.A node #ires less than C< times per min"te o $ormal in some people (eg+ Athletes
Management: 6 treat the "nderlying ca"se, increase the heart rate as appropriate 6 Isoproterenol
ATRIAL DYSRHYTHMIAS
&remat"re Atrial complex (&A! Atrial ,l"tter Atrial ,ibrillation &aroxysmal Atrial Tachycardia
VENTRICULAR FI.RILLATION 6 3i#e-threatening dysrhythmia characterized by rapid, erratic imp"lse #ormation and cond"ction 'hich ca"ses a.rupt cessation of cardiac output0 6 2es"lts #rom severe /I, electroc"tion, digitalis toxicity, electrolyte imbalance Management: 6 De#ibrillation, 6 $a (icarbonate, 6 :pinephrine
PREMATURE ATRIAL CONTRACTION 6 :arly beats arising #rom the atria and interr"pting the normal rhythm 6 Associated 'ith "al"ular disorders, atrial cham.er enlar$ement, stress, fati$ue, heart failure, M*, CA!, pulmonary hpn 6 & 'aves are premat"re and di##erent #rom the normal sin"s & 'ave in appearance, shape and size Management: 6 I"inidine or procainamide
AV n 0$ D*!&-*"-)ia!
o o o ,irst degree heart block .econd degree heart block 6 /obitz type ; 6 /obitz type A Third degree heart block
ATRIAL FLUTTER 6 .ame toother atrial 'ave #ormation ca"sed by rapid reentry in the atria 6 (trial rate ranges from ..B20?B bpm 6 A1 node cannot cond"ct all the atrial imp"lses that bombard it
3*RST !E4REE A8 ,+'C9 6 !elay in the passa$e of impulse from the atria to "entricles usually occurs at the le"el of the A8 node 6 2es"lts #rom !AD, increased vagal stim"lation, congenital anomalies and digitalis administration 6 2e="ires observation and monitoring since it can progress to higher-degree A1 block
SEC'(! !E4REE A8 ,+'C9 6 /ore serio"s and some impulses are .loc/ed while others are not 6 Develops #rom !AD, digitalis toxicity, rhe"matic #ever, viral in#ections and /I 0 types: 6 Mobit/ Type + Block %7enckebach& 6 Mobit/ Type . Block 6 6 6 MOB)TE T>#" + B6OC@ Caused .y a lon$ refractory period that occurs at the A8 node &2 interval lengthens "ntil a & 'ave is not cond"cted Interventions is not re="ired as long as the ventric"lar rate remains ade="ate #or per#"sion
6 6
.acc"lar (an o"tpo"ching o# an artery 'here the medial coat is thinned Dissecting (hematoma in the artery 'all #rom a localized enlargement o# the involved artery
MOB)TE T>#" . B6OC@ 6 +e"el of the .loc/ is .elow the A8 node 6 2e="ires :!7 monitoring Management: a"minist&ation of at&opine/ inse&tion of pa#ema1e&/ 2it((ol"ing #a&"ia# "ep&essant "&ugs TH*R! !E4REE A8 ,+'C9 6 Complete a.sence of conduction of electrical impulses d"e to a block in the A1 node, b"ndle o# 4is or b"ndle branches 6 Danger o# ventric"lar standstill or asystole Management: T, of CHOICE: ;"1TR)CU6(R #(C"M(@"R
Complication: Rupture T&ia" manifestations of &uptu&e: &ain &"lsating mass .hock 6ther mani#estations: .yncope 3ight headedness hypotension Me"i#al Management: (ntihypertensives as ordered: ultrasonography every F months Su&gi#al Management: Resection and graft replacment
CARDIAC TAMPONADE
6 &ericardial e##"sion occ"rs 'hen the space bet+ the parietal ) visceral layers o# the pericardi"m #ill 'ith #l"id+8
Assessment: o ,ec/s triad: distended neck veins 0 m"##led heart so"nds 0 hypotension o !hest pain o !ardiogenic shock o Increased !1& Management: o !!@ #or hemodynamic monitoring o &:2I!A2DI6!:$T:.I. o Admin+ I1 #l"ids as prescribed
HEART FAILURE
6 Causes: a< .< c< d< e Types: *na.ility of the heart to maintain ade;uate circulation to meet the meta.olic needs of the .ody .ec& of an impaired pumpin$ capa.ility& Hyper"olemia Arteriosclerosis Myocardial *nfarction Cardiomyopathies 8al"e disorders Ri$ht-sided CH3 Systemic S)< 6 3ati$ue 6 distended 7u$ular "eins 6 Ascites 6 pittin$ edema 6 Cyanosis 6 hepatome$aly 6 increased peripheral "enous pressure 6 anore)ia = 4* distress 6 polyuria = wt& $ain
ANEURYSMS
6 6 6 &ermanent localized dilation o# an artery that enlarges grad"ally !a"ses: atherosclerosis, congenital mal#ormations, in#ection, connective tiss"e disorders, hypertension !omplications: r"pt"re, press"re on s"rro"nding str"ct"res, thrombosis and embolization
Classification of (neurysms according to* 3ocation 6 1eno"s or arterial 6 Aortic, iliac, etc++ 6 7ross appearance: ,"si#orm (localized dilations o# an artery
+eft-sided CH3 #ulmonary S)< 6 Cardiome$aly 6 .lood tin$ed sputum 6 Cou$h 6 acute pulmo edema 6 E)ertional dyspnea 6 cyanosis 6 'rthopnea 6 wt& +oss Management 6 Rest 6 Hi$h-3owlers or sittin$ 6 !ecrease fluids > (a? 6 Medications:
o o o o o o o o o
!ardiac 7lycosides % (K inotropy 0 (chronotropy digitalis 0 digoxin (3anoxin 0 digitoxin (!rystodigin 0 lanatoside (!edilanid-! Di"retics % 4A6 ) $aK excretion 3oop di"retics % ,"rosemide (3asix Thiazide di"retics % chlorthiazide (Di"ril &otassi"m sparing % spironolactone (Aldactone Inotropics % increases the strength o# contraction s"ch as "opamine *Int&opin+/ "o)utamine *Do)ut&e,+ 1asodilators 2otating To"rni="et #rinciples: o apply 3 tou&ni4uets o inflate #uff 56 mm a)o-e "iastoli# p&essu&e o &otate 4 5 mins. o #(e#1 "istal pulses o &emo-e 5 at a time 7 5 mins. Inte&-al