-Lifestyle -Hereditary -Diet -Age -Gender Precipitating factors: -Stress -Presence of Pathogens Possible agents Typical Atypical S. Pne!oniae M. Pne!oniae H. "nflen#ae $. pne!onia S. Ares Legionella spp. "n%asion of bacteria or %irs in the body &rganis!s enter the 'espiratory tract (y inspiration or aspiration Acti%ation of defense Mechanis! LEGEND: Pleral effsion $AP DM $o!!on to all S)S* doctor+s order* nrsing !anage!ent D.& Doctor+s order N.M. Nrsing !anage!ent Direct ----- Not direct 62 G,N,'AL ASS,SSM,NT -. y)o* !ale* (P: /-0)/10* (M": /2.-1* Pre !orbid diet: high in carb* high in fats Persistent cogh 3/ !onth4* 5hite phleg!* 364 s!o7ing 38 years / pac7 a day4* 364 alcoholis!* 9a!ily history of: DM type ""* HPN* 'espiratory Diseases* 9ree lance electrician* concilor* 5ith periodic bris7 5al7ing* $:' "!pression: Pne!onia retrogressing* $onco!itant ,:tensi%e PT( highly entertained. Loose effecti%eness of Defense !echanis! Penetration in the lo5er lngs 3Al%eoli4 (acteria 5ill !ltiply $oloni#ation of bacteria in the lngs (acteria 5ill release Da!aging to:ins "nfection in the Lngs "nfla!!atory process ;aso dililation "ncrease blood flo5 Plas!a and $H&N rich Lea7age flid Al%eolar sacs 5ill be filled 5ith flid Decrease o:ygen Lng consolidation Hypo:e!ia Possible $o!plications: "ncrease blood pressre in the Lngs< respiratory arrest "f treated: reco%ery not treated: death Predisposing factors: -age -se: Precipitating factors -increase $(G Altered !etabolis! rt. lifestyle "!paired beta cell fnction Peripheral inslin resistance "ncrease Hepatic Glcose prodction "ncrease beta cell secretion Hyperglyce!ia 3=.>!!)!ol4 (ody atte!pts to !aintain nor!al glcose le%el "ncrease postprandial Glcose le%el Absolte inslin deficiency de to progressi%e beta cell failre Type ""* DM Diagnostic e:a!s: 9(S H(A/$ D.&. Ad!inister inslin 9or H(A/$* 9(S* Lipid profile N.M "nstrct the pt. to a%oid s5eet foods. $linical Manifestation: ,:cessi%e s5eating Trace of sgar in the rine 63 v "ncrease sti!lation of Mast cells "ncrease sti!lation of Goblet cells 'elease of hista!ine "ncrease !cs prodction "ncrease %asclar per!eability "ncrease hydrostatic pressre "ncrease flid shifting in the plera "ncrease flid collection "ncrease co!pression in the lngs "rritation of pleral space S&( and $hest disco!fort "!pair!ent of gas e:change Heart rate increases as the co!pensatory !echanis! of the body Dyspnea 364 5hee#e $hanges in '' and rhyth! of breathing D.&. Gi%e Acetylcystein =00!g p.o. Salbta!ol /neb ?1 N.M. "ndependent: -"nstrct the pt. to increase flid inta7e -!oti%ate pt. to e:pertorate Dependent: -"nstrct the pt. to dilte acetylcystein in half glass of 5ater Dyspnea $apilliary refill in 8 seconds Nasal flaring D.&. Albterol 3Salbta!ol4 /neb ?1 N.M. Ad%ise pt. to 'inse !oth After inhalation "ncrease arterial pressre Peripheral %aso constriction 64 "ncrease blood flo5 in the site "ncrease blood pressre 3Hypertension4 $linical !anifestation: (P of ==0)/.0!!Hg D.&. $atapres >8 !cg)tab* / tab P'N for (P@ /.0)/00 !!Hg 65