Disease caused by an infectious agent that are transmitted directly
or indirectly to a well person through an agency, vector or inanimate object CONTAGO!" D"#A"# Disease that is easily transmitted from one person to another N$#CTO!" D"#A"# Disease transmitted by direct inoculation through a brea% in the s%in N$#CTON &#ntry and multiplication of an infectious agent into the tissue of the host N$#"TATON & 'odgement and development of arthropods on the surface of the body A"#("" & Absence of disease ) producing microorganisms "#("" & The presence of infection *#DCA' A"#("" -(ractices designed to reduce the number and transfer of pathogens -Clean techni+ue "!,GCA' A"#("" -(ractices that render and %eep objects and areas free from microorganisms -Sterile techni+ue CA,,#, ) an individual who harbors the organism and is capable of transmitting it without showing manifestations of the disease CA"# ) a person who is infected and manifesting the signs and symptoms of the disease "!"(#CT ) a person whose medical history and signs and symptoms suggest that such person is suffering from that particular disease CONTACT ) any person who had been in close association with an infected person -O"T & A person, animal or plant which harbors and provides nourishment for a parasite ,#"#,.O, & Natural habitat for the growth, multiplication and reproduction of microorganism "O'ATON & The separation of persons with communicable diseases from other persons /!A,ANTN# & The limitation of the freedom of movement of persons e0posed to communicable diseases "T#,'1ATON ) the process by which all microorganisms including their spores are destroyed D"N$#CTON ) the process by which pathogens but not their spores are destroyed from inanimate objects C'#ANNG ) the physical removal of visible dirt and debris by washing contaminated surfaces CONC!,,#NT & Done immediately after the discharge of infectious materials 2 secretions T#,*NA' & Applied when the patient is no longer the source of infection 3ACT#,CDA' & A chemical that %ills microorganisms 3ACT#,O"TATC & An agent that prevents bacterial multiplication but does not %ill microorganisms C-AN O$ N$#CTON N$#CTO!" AG#NT Any microorganism capable of producing a disease ,#"#,.O, #nvironment or object on which an organism can survive and multiply (O,TA' O$ #4T The venue or way in which the organism leaves the reservoir *OD# O$ T,AN"*""ON The means by which the infectious agent passes from the portal of e0it from the reservoir to the susceptible host (O,TA' O$ #NT,5 (ermits the organism to gain entrance into the host "!"C#(T3'# -O"T A person at ris% for infection, whose defense mechanisms are unable to withstand invasion of pathogens "TAG#" O$ T-# N$#CTO!" (,OC#"" ncubation (eriod ) ac+uisition of pathogen to the onset of signs and symptoms (rodromal (eriod ) patient feels 6bad7 but not yet e0periencing actual symptoms of the disease (eriod of llness ) onset of typical or specific signs and symptoms of a disease Convalescent (eriod ) signs and symptoms start to abate and client returns to normal health *OD# O$ T,AN"*""ON CONTACT T,AN"*""ON Direct contact ) involves immediate and direct transfer from person&to&person 8body surface&to&body surface9 ndirect contact ) occurs when a susceptible host is e0posed to a contaminated object D,O('#T T,AN"*""ON Occurs when the mucous membrane of the nose, mouth or conjunctiva are e0posed to secretions of an infected person within a distance of three feet .#-C'# T,AN"*""ON Transfer of microorganisms by way of vehicles or contaminated items that transmit pathogens A,3O,N# T,AN"*""ON Occurs when fine particles are suspended in the air for a long time or when dust particles contain pathogens .#CTO,&3O,N# T,AN"*""ON Transmitted by biologic vectors li%e rats, snails and mos+uitoes T5(#" O$ **!N1ATON ACT.# ) antibodies produced by the body NAT!,A' ) antibodies are formed in the presence of active infection in the body: lifelong A,T$CA' ) antigens are administered to stimulate antibody production (A"".# ) antibodies are produced by another source NAT!,A' ) transferred from mother to newborn through placenta or colostrum A,T$CA' ) immune serum 8antibody9 from an animal or human is injected to a person "#.#N CAT#GO,#" O$ "O'ATON "T,CT& prevent highly contagious or virulent infections #0ample; chic%enpo0, herpes <oster CONTACT ) spread primarily by close or direct contact #0ample; scabies, herpes simple0 ,#"(,ATO,5 ) prevent transmission of infectious distances over short distances through the air CD-Bucud 1 #0ample; measles, mumps, meningitis T!3#,C!'O"" ) indicated for patients with positive smear or chest 0&ray which strongly suggests tuberculosis #NT#,C ) prevent transmission through direct contact with feces #0ample; poliomyelitis, typhoid fever D,ANAG# ) prevent transmission by direct or indirect contact with purulent materials or discharge #0= 3urns !N.#,"A' ) prevent transmission of blood and body&fluid borne pathogens #0ample; AD", -epatitis 3 CENTRAL NERVOUS SYSTEM MENINGO- COCCEMIA MENINGITIS ENCEPHALITIS MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD MODE OF TRANSMISSION - Inflammation of the brain - Inflammation of the meninge ! A"#te infe"tion of the bloo$tream an$ $e%elo&ing %a"#liti - Arbo%ir#e - Stre&to"o""# - Sta&h'lo"o""# - Pne#mo"o""# - T#ber"le ba"ill# - Neieria meningiti$e (!)( $a' )!)* $a' +!, $a' Bite of infe"te$ mo-#ito Re&irator' $ro&let "GN" AND "5*(TO*" O$ #NC#(-A'T" .ir# enter ne#ral "ell Peri%a"#lar "ongetion Dir#&tion in "ell#lar f#n"tioning Inflammator' rea"tion Letharg' Con%#lion Sei/#re 0ea$a"he Photo&hobia .omiting Stiff ne"1 Fe%er Sore throat "GN" AND "5*(TO*" O$ *#NNGT" T-,## "GN" O$ *#NNG#A' ,,TATON O("T-OTON!" "tate of severe hypere0tension and spasticity in which an individual>s head, nec% and spinal column enter into a complete arching position 3,!D1N"?>" "GN (lace the patient in a dorsal recumbent position and then put hands behind the patient>s nec% and bend it forward= f the patient fle0es the hips and %nees in response to the manipulation, positive for meningitis ?#,NG>" "GN (lace the patient in a supine position, fle0 his leg at the hip and %nee then straighten the %nee: pain and resistance indicates meningitis "GN" AND "5*(TO*" O$ *#NNGOCOCC#*A URTI2 "o#gh3 ore throat3 fe%er3 hea$a"he3 na#ea an$ %omiting .a"#liti2 &ete"hial rah in the tr#n1 an$ e4tremitie DIC Mi"ro! thromboi P#r&#ra 0'&otenion Sho"1 Death MENINGO- COCCEMIA MENINGITIS ENCEPHALITIS SIGNS AND S5MPTOMS INCIDENCE (!)* 'ear ol$ 6 ( 'ear ol$ 7 month8( 'ear ol$ Stiff ne"1 Photo&hobia Letharg' Con%#lion N#"hal rigi$it' O&ithoton# Br#$/in1i9 :ernig9 ign .a"#liti ;aterho#e! Fri$eri"hen 'n$rome Pete"hiae <ith the $e%elo&ment of hemorrhage DAGNO"TC #4A* nformed consent #mpty bowel and bladder $etal, shrimp or 6C7 position "pinal canal, subarachnoid space between '@&'A or 'A& 'B After; bedrest $lat on bed to prevent spinal headache MENINGO- COCCEMIA MENINGITIS ENCEPHALITIS TREATMENT MODALITIES PRE.ENTION )= >a&anee en"e&haliti .A? )= 0iB %a""ine De4amethaone Mannitol Anti"on%#lant Anti&'reti" Ceftria4one Peni"illin Chloram&heni"ol Rifam&i"in Ci&roflo4a"in CD-Bucud 2 MENINGO- COCCEMIA MENINGITIS ENCEPHALITIS NURSING MANAGEMENT )= Comfort2 -#iet3 <ell!%entilate$ room @= S1in "are2 "leaning bath3 "hange in &oition += Eliminate mo-#ito bree$ing ite2 CULE? mo-#ito )= Re&irator' iolation @,!A@ ho#r after onet of antibioti" thera&' @= Room &rote"te$ againt bright light += Safet'2 i$e!l'ing &oition an$ raie$ i$e rail )= Si$e boar$ @= Cloe "onta"t 0 8 o#e I 8 nfe"te$ &eron 1iing S 8 ame $a'"are "enter S 8 hare mo#th intr#ment += Antibioti" a &ro&h'la4i TETANUS RABIES POLIOMYELITIS MAIN PROBLEM ETIOLOGIC AGENT A"#te infe"tion of the CNS 8 m#"le &am3 &arei an$ &aral'i A"#te %iral $ieae of the CNS 8 b' ali%a of infe"te$ animal A"#te infe"tio# $ieae <ith 'temi" ne#rom#"#lar effe"t Legio $ebilitan Rhab$o%ir# B#llet!ha&e$ Affinit' to CNS :ille$ b' #nlight3 U. light3 formalin Reitant to antibioti" Clotri$i#m tetani Anaerobi" Gram &oiti%e Dr#mti"1 a&&earan"e TETANUS RABIES POLIOMYELITIS INCUBATION PERIOD MODE OF TRANSMISSION A!@) $a' @!B <ee1 A$#lt2 + $a'!+ <ee1 Ditan"e of bite to brain ! Dire"t "onta"t <ith infe"te$ fe"e Bite of an infe"te$ animal Dire"t ino"#lation thro#gh a bro1en 1in E4teni%ene of the bite Reitan"e of the hot Neonate2 +!+* $a' ! Dire"t "onta"t <ith re&irator' e"retion ! In$ire"t <ith oile$ linen an$ arti"le TETANUS RABIES POLIOMYELITIS SIGNS AND S5MPTOMS )= Aborti%e t'&e )= Pro$romal C in%aion &hae R 8 i# ar$oni"# += Paral'ti" t'&e @= Pre!&aral'ti" or meningeti" t'&e += Terminal C &aral'ti" t'&e @= E4"itement C ne#rologi"al &hae O 8 &itothon# T 8 rim# C 8 on%#lion 0 8 ea$a"he I 8 rritabilit' L 8 ar'ngeal &am POLIO ABORTIVE TYPE Does not invade the CN" -eadache "ore throat ,ecovery within CD hours and the disease passes by unnoticed PRE-PARALYTIC OR MENINGETIC TYPE "light involvement of the CN" (ain and spasm of muscles Transient paresis 8E9 (andy>s test 8increased protein in the C"$9 PARALYTIC TYPE CN" involvement $laccid paralysis Asymmetric Affects lower e0tremities !rine retention and constipation 8E9 -O5N#>" "GN 8when in supine position, head will fall bac% when shoulders are elevated9 RABIES PRODROMAL/INVASION PHASE $ever Anore0ia "ore throat (ain and tingling at the site of bite Difficulty swallowing EXCITEMENT OR NEUROLOGICAL PHASE -ydrophobia 8laryngospasm9 Aerophobia 8bronchospasm9 Delirium *aniacal behavior Drooling TERMINAL OR PARALYTIC PHASE (atient becomes unconscious 'oss of urine and bowel control (rogressive paralysis Death TETANUS RABIES POLIOMYELITIS COMPLICATION ISOLATION PRECAUTION DIAGNOSTIC PROCEDURES Paral'i of re&irator' m#"le RESPIRATOR5 FAILURE DEAT0 )= Stool "#lt#re )= Throat <ahing )= Bloo$ e4am Enteri" iolation Re&irator' iolation @= CSF "#lt#re @= Flo#re"ent rabie antibo$' DFRAE += Negri bo$ie TETANUS RABIES POLIOMYELITIS TREATMENT MODALITIES )= Analgei" )= Lo"al treatment of <o#n$ )= Tetan# imm#ne glob#lin DTIGE @= Mor&hine += Moit heat a&&li"ation ,= Be$ ret (= Rehabilitation @= A"ti%e imm#ni/ation L'a%a" Imo%a4 Antirabie %a4 @= Pai%e imm#ni/ation @= Tetan# antito4in DTATE += Peni"illin G ,= Tetra"'"line (= Dia/e&am 7= Phenobarbital A= Tra"heotom' B= NGT fee$ing CD-Bucud 3 TETANUS RABIES POLIOMYELITIS NURSING MANAGEMENT )= Enteri" iolation )= Iolation )= A$e-#ate air<a' @= Pro&er $i&oal of e"retion += Moit hot &a"1 ,= Firm C nonagging be$ (= S#itable bo$' alignment 7= Comfort an$ afet' @= O&tim#m "omfort += Retf#l en%ironment ,= Emotional #&&ort (= Con"#rrent an$ terminal $iinfe"tion @= F#iet3 emi!$ar1 en%ironment += A%oi$ #$$en tim#li an$ light TETANUS RABIES POLIOMYELITIS PRE.ENTION Sal1 %a""ine - Inactivated polio vaccine - Intramuscular )= If the $og i health' )= Ae&ti" han$ling of #mbili"al "or$ Sabin %a""ine - Oral polio vaccine - Per orem @= If the $og $ie or ho< ign #ggeti%e of rabie += If $og i not a%ailable for ober%ation ,= 0a%e $ometi" $og + month to ) 'ear ol$ imm#ni/e$ @= Tetan# to4oi$ imm#ni/ation += Antibioti" &ro&h'la4i - Penicillin - Erythromycin ! Tetracycline RESPIRATORY SYSTEM SARS BIRD FLU MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD MODE OF TRANSMISSION Fl# infe"tion in bir$ that affe"t h#man A ne< t'&e of at'&i"al &ne#monia that infe"t the l#ng A%ian infl#en/a %ir#3 0(N) Corona %ir# +!( $a' @!B $a' Inhalation of fe"e an$ $i"harge of an infe"te$ bir$ Re&irator' $ro&let SARS BIRD FLU SIGNS AND S5MPTOMS Bo$' <ea1ne or m#"le &ain 0igh fe%er G+B9Celi# Chill Co#gh Diffi"#lt' breathing E&io$e of ore throat Fe%er SARS BIRD FLU COMPLICATIONS Se%ere %iral &ne#monia A"#te re&irator' $itre 'n$rome 0'&o4emia Fl#i$ a""#m#lation in al%eolar a" Se%ere breathing $iffi"#ltie M#lti&le organ fail#re DEAT0 Se%ere %iral &ne#monia Re&irator' fail#re SARS BIRD FLU TREATMENT MODALITIES - Generi" fl# $r#g )= No $efiniti%e treatment for SARS )= Amanta$ineCRimanta$ine ! 0(N) $e%elo&e$ reitan"e @= Oeltami%ir DTAMIFLUE Hanama%ir DRELENHAE ! Primar' treatment - ;ithin @ $a' at onet of 'm&tom - )(* mg BID 4 @ $a' @= Anti%iral $r#g Dnormall' #e$ to treat AIDSE ! RIBA.IRIN += Corti"oteroi$ SARS BIRD FLU PRE.ENTION )=C#lling 8 1illing of i"1 or e4&oe$ bir$ )=F#arantine @= Banning of im&ortation of bir$ DE4e"#ti%e or$er I @B*E += Coo1 "hi"1en thoro#ghl' @= Iolation += ;0O alert on SARS DMar"h )@3 @**+E NURSING MANAGEMENT BIRD FLU WHAT TO DO WITH A PERSON SUSPECTED TO HAVE BIRD FLU solation $ace mask on the patient Caregiver; use a fac mask and ! "#""$s/"$asss Distance of % m&' from the patient Transport the patient to a DOH 'f''a$ (#s)*&a$ ,#$#,,A' -O"(TA'" Na&*#+a$ Rf''a$ C+&' ) ,esearch nstitute for Tropical *edicine ,RITM- 8Alabang, *untinlupa9 L./#+ ) Sa+ La/a'# H#s)*&a$ 8/uiricada "t=, "ta= Cru<, *anila9 V*sa!as ) V*c+& S#&&# Mm#'*a$ M0*ca$ H#s)*&a$ 8Cebu City9 M*+0a+a# ) Da1a# M0*ca$ C+&' 83ajada, Davao City9 S A R S "!"(#CT CA"# F= A person presenting after F November DGGD with a history of; H*"( f1' 234 5C AND C#."( #' 6'a&(*+" 0*ff*c.$&! AND CD-Bucud 4 One or more of the following e0posures during the %5 0a!s prior to the onset of symptoms; C$#s c#+&ac& , with a person who is a suspect or probable case of "A," H*s&#'! #f &'a1$ , to an area with recent local transmission of "A," Rs*0*+" in an area with recent local transmission of "A," D= A person with an .+7)$a*+0 ac.& 's)*'a&#'! *$$+ss resulting in 0a&( after F November DGGD, but on whom +# a.&#)s! has been performed ; AND One or more of the following e0posures during the FG days prior to the onset of symptoms; C$#s c#+&ac& , with a person who is a suspect or probable case of "A," H*s&#'! #f &'a1$ , to an area with recent local transmission of "A," Rs*0*+" in an area with recent local transmission of "A," (,O3A3'# CA"# F= A suspect case with 'a0*#"'a)(*c 1*0+c of infiltrates consistent with pneumonia or respiratory distress syndrome on Chest 0&ray= D= A suspect case of "A," that is positive for SARS c#'#+a1*'.s by one or more assa!s8 @= A suspect case with a.&#)s! f*+0*+"s consistent with the pathology of "A," without an identifiable cause= PERTUSSIS DIPHTHERIA MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD MODE OF TRANSMISSION A"#te ba"terial $ieae "hara"teri/e$ b' the elaboration of an e4oto4in Re&eate$ atta"1 of &amo$i" "o#ghing Cor'neba"teri#m$i&htheriae or :leb!Loeffler ba"ill# Bor$etella &ert#i @!( $a' A!), $a' )= Re&irator' $ro&let 2. Dire"t "onta"t <ith re&irator' e"retion += In$ire"t "onta"t <ith arti"le
PERTUSSIS DIPHTHERIA SIGNS AND S5MPTOMS T'&e2 )=Naal @=Tonilo&har'ngeal +=Lar'ngeal ,=;o#n$ or "#taneo# Stage2 )= Catarrhal @= Paro4'mal += Con%ale"ent
NA"A' D(-T-#,A B$##0! 0*sc(a'" f'#m &( +#s E7c#'*a&0 +a's a+0 .))' $*) TON"'O(-A,5NG#A' D(-T-#,A L#9 "'a0 f1' S#' &('#a& B.$$-+ck a))a'a+c Ps.0#mm6'a+- Group of pale yellow membrane over tonsils and at the bac% of the throat as an inflammatory response to a powerful necroti<ing to0ins 'A,5NG#A' D(-T-#,A H#a's+ss C'#.)! c#."( A)(#+*a Mm6'a+ $*+*+" &(*ck+s a*'9a! #6s&'.c&*#+ S.ff#ca&*#+: c!a+#s*s #' 0a&( HO!ND O, C!TAN#O!" D(-T-#,A Y$$#9 s)#&s #' s#'s *+ &( sk*+ PERTUSSIS CATA,,-A' "TAG# Las&s f#' % &# ; 9ks M#s& c#mm.+*ca6$ s&a" B"*+s 9*&( 's)*'a&#'! *+fc&*#+: s+/*+": c#."( a+0 f1' C#."( 6c#ms m#' f'<.+& a& +*"(& (A,O45"*A' "TAG# Las&s f#' = &# > 9ks A.'a? s+/*+": &*ck$*+": *&c(*+" #f &('#a& C#."(: 7)$#s*1 #.&6.'s& +0*+" *+ @9(##)A M.c.s *s &(*ck: +0s *+ 1#m*&*+" Bc#ms c!a+#&*c W*&( )'#f.s s9a&*+": *+1#$.+&a'! .'*+a&*#+ a+0 7(a.s&*#+ CON.A'#"C#NT "TAG# E+0 #f =&(->&( 9k Dc'as *+ )a'#7!sms PERTUSSIS DIPHTHERIA DIAGNOSTIC PROCEDURES SC0IC:9S TESTS CBC 8 in"reae in l'm&ho"'te ! S#"e&tibilit' an$ imm#nit' to $i&htheria -ID of $il#te $i&htheria to4in D*=) ""E DJE lo"al "ir"#m"ribe$ area of re$ne3 )!+ "m MALONE59S TEST !Determine h'&ereniti%it' to $i&htheria anti!to4in -ID of *=) "" fl#i$ to4oi$ -DJE area of er'thema in @, ho#r
PERTUSSIS DIPHTHERIA COMPLICATIONS To4in in the bloo$tream M'o"ar$iti De&igatri" or "het &ainE Peri&heral &aral'i Dtingling3 n#mbne3 &areiE Bron"ho! &ne#monia Dfe%er3 "o#ghE 0eart fail#re De"reae$ in re&irator' rate Re&irat or' arret DEAT0 Con%#lion Dbrain $amage from a&h'4iaE Otiti me$ia Din%a$ing organimE Bron"ho&ne#monia Dmot $angero# "om&li"ationE PERTUSSIS DIPHTHERIA TREATMENT MODALITIES )= Di&htheria anti!to4in )= Er'throm'"in 8 $r#g of "hoi"e - Requires skin testing - Early administration aimed at neutralizing the toin present in the circulation !e"ore it is a!sor!ed !y the tissues @= Antibioti" thera&' - Penicillin # - Erythromycin @= Am&i"illin 8 if reitant to er'throm'"in += Betamethaone D"orti"oteroi$E 8 $e"reae e%erit' an$ length of &aro4'm ,= Alb#terol Dbron"ho$ilatorE
CD-Bucud 5 PERTUSSIS DIPHTHERIA NURSING MANAGEMENT )= Iolation2 ), $a' D#ntil @!+ "#lt#re3 @, ho#r a&artE )= Iolation2 ,!7 <ee1 from onet of illne @= Be$ret for @ <ee1 += Care for noe an$ throat Dgentle <abbingE ,= I"e "ollar D$e"reae &ain of ore throatE (= Diet Doft foo$3 mall fre-#ent fee$ingE @= S#&&orti%e mea#re Dbe$ret3 a%oi$ e4"itement3 $#t3 mo1e an$ <arm bathE += Safet' D$#ring &aro4'm3 &atient ho#l$ not be left aloneE ,= S#"tioning D1e&t at be$i$e for emergen"' #eE MUMPS MAIN PROBLEM An acute contagious disease, with swelling of one or both of the parotid glands ETIOLOGIC AGENT $ilterable virus of paramy0ovirus group INCUBATION PERIOD FD&DI days MODE OF TRANSMISSION ,espiratory droplets PERIOD OF COMMUNICABILITY I days before and J days after onset of parotid swelling SIGNS AND SYMPTOMS (,OD,O*A' (-A"# $&ever 8low grade9 -&eadache *&alaise (A,OTT" $&ace pain #&arache "&welling of the parotid glands CO*('CATON" Orchitis ) the most notorious complication of mumps Oophoritis ) manifested by pain and tenderness of the abdomen CN" involvement ) manifested by headache, stiff nec%, delirium, double vision Deafness as a result of mumps N!,"NG *ANAG#*#NT F= (revent complications "crotum supported by suspensory !se of sedatives to relieve pain Treatment; oral dose of @GG&AGG mg cortisone followed by FGG mg every I hours Nic% in the membrane D= Diet & "oft or li+uid diet & "our foods or fruit juices are disli%ed @= ,espiratory isolation A= Comfort; ice collar or cold applications over the parotid glands may relieve pain B= $ever; aspirin, tepid sponge bath I= Concurrent disinfection; all materials contaminated by these secretions should be cleansed by boiling C= Terminal disinfection; room should be aired for si0 to eight hours GASTROINTESTINAL TRACT SHIGELLOSIS AMOEBIASIS MAIN PROBLEM ETIOLOGIC AGENT Proto/oal infe"tion of the large intetine A"#te infe"tion of the lining of the mall intetine Entamoeba hitol'ti"a Shigella gro#& ! Pre%alent in area <ith ill anitation -A"-#ire$ b' <allo<ing ! Tro&ho/oite2 %egetati%e form ! C't2 infe"ti%e tage )= Shigella fleneri 8 mot "ommon in the Phili&&ine @= Shigella "onnei += Shigella bo'$ii ,= Shigella $'enterae 8 mot infe"tio# t'&e
SHIGELLOSIS AMOEBIASIS NURSING MANAGEMENT )= Enteri" iolation @= Boil <ater for $rin1ing += 0an$<ahing ,= Se4#al a"ti%it' (= A%oi$ eating #n"oo1e$ leaf' %egetable CD-Bucud 6 TYPHOID FEVER CHOLERA MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD MODE OF TRANSMISSION A"#te ba"terial $ieae of the GIT "hara"teri/e$ b' &rof#e e"retor' $iarrhea An infe"tion affe"ting the Pe'er9 &at"he of the mall intetine .ibrio "holerae Salmonella t'&hi ) to + $a' ) to + <ee1 )= Fe"al!oral tranmiion @= ( F9 TYPHOID FEVER CHOLERA SIGNS AND S5MPTOMS Ri"e!<ater tool Fe%er Dla$$er!li1eE Ab$ominal "ram& .omiting Intra%a"#lar Deh'$ration Sho"1 Roe &ot Diarrhea T5P0OID STATE Sor$e S#b#lt# Ten$in#m Coma %igil Car&hologia TYPHOID FEVER CHOLERA TREATMENT MODALITIES )= La"tate$ Ringer9 ol#tion )= Chloram&heni"ol 8 $r#g of "hoi"e @= Oral reh'$ration thera&' += Antibioti" thera&' - Tetra"'"line 8 $r#g of "hoi"e - Cotrimo4a/ole - Chloram&heni"ol @= Am&i"illinC Amo4i"illin 8 for t'&hoi$ "arrier += Cotrimo4a/ole 8 for e%ere "ae <ith rela&e TYPHOID FEVER CHOLERA NURSING MANAGEMENT )= Maintain an$ retore the fl#i$ an$ ele"trol'te balan"e @= Enteri" iolation += Sanitar' $i&oal of e4"reta ,= A$e-#ate &ro%iion of afe $rin1ing <ater (= Goo$ &eronal h'giene INTEGUMENTARY SYSTEM HERPES BOSTER CHICCENPOX MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD MODE OF TRANSMISSION A highl' "ontagio# $ieae "hara"teri/e$ b' %ei"#lar er#&tion on the 1in an$ m#"o# membrane An a"#te %iral infe"tion of the enor' ner%e .ari"ella /oter %ir# )*!@) $a' )+!)A $a' )= Dro&let metho$ @= Dire"t "onta"t += In$ire"t "onta"t HERPES BOSTER CHICCENPOX SIGNS AND S5MPTOMS PERIOD OF COMMUNICABILIT5 One $a' before er#&tion of ) t leion an$ fi%e $a' after a&&earan"e of lat "ro& One $a' before er#&tion of ) t rah an$ fi%e to i4 $a' after the lat "r#t PRODROMAL PERIOD - Fever $lo%-grade& - 0eadache - Malaise HERPES BOSTER CHICCENPOX SIGNS AND S5MPTOMS Rashes ' (entri"ugal distri!ution Rahe -Unilateral3 ban$!li1e $itrib#tion Rash stages' macule papule vesicle pustule crust Pruritus -Dermatomal ! Er'themato# bae ! .ei"#lar3 &#t#lar or "r#ting Regional l'm&ha$eno&ath' Pr#rit# Pain 8 tabbing or b#rning HERPES BOSTER CHICCENPOX COMPLICATIONS SCARRING ) most common complication* associated %ith staphylococcal or streptococcal in"ections "rom scratching NECROTIHING FASCIITIS ) most severe complication RE5E S5NDROME ) a!normal accumulation o" "at in the liver plus increase o" pressure in the !rain resulting to coma+ there"ore leading to ,E-./ RAMSA5!0UNT S5NDROME - Involvement o" Involvement o" the "acial nerve in herpes zoster the "acial nerve in herpes zoster %ith "acial paralysis+ hearing %ith "acial paralysis+ hearing loss+ loss o" taste in hal" o" the loss+ loss o" taste in hal" o" the tongue tongue GASSERIAN GANGLIONITIS ) Involvement o" the optic nerve resulting to corneal anesthesia ENCEP0ALITIS ) acute in"lammatory condition o" the !rain CD-Bucud 7 HERPES BOSTER CHICCENPOX TREATMENT MODALITIES )= Antihitamine 8 'm&tomati" relief of it"hing E4= Di&henh'$ramine DBena$r'lE @= Analgei" an$ anti&'reti" E4= A"etamino&hen += Anti%iral agent 8 for &atient to e4&erien"e le &ain an$ fater reol#tion of leion <hen #e$ <ithin ,B ho#r of rah onet E4= A"'"lo%ir DHo%ira4E ,= Corti"oteroi$ 8 anti! inflammator' an$ $e"reae$ &ain E4= Pre$nione HERPES BOSTER CHICCENPOX NURSING MANAGEMENT Strict isolation Prevent secondary in"ection $cut "ingernails short+ %ear mittens& Eliminate itching' calamine lotions+ %arm !aths+ !aking soda paste Encourage not going to school' usually 0 days Disin"ection o" clothes and linen %ith nasopharyngeal discharges !y sunlight or !oiling GERMAN MEASLES MEASLES MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD MODE OF TRANSMISSION A "ontagio# e4anthemato# $ieae <ith "hief 'm&tom to the #&&er re&irator' tra"t A benign "omm#ni"able e4anthemato# $ieae "a#e$ b' r#bella %ir# )= Dro&let metho$ R#bella %ir# )*!)@ $a' ),!@) $a' Filterable %ir# of &aram'4o%iri$ae @= Dire"t "onta"t <ith re&irator' $i"harge += In$ire"t <ith oile$ linen an$ arti"le GERMAN MEASLES MEASLES PERIOD OF COMMUNICABILIT5 SIGNS AND S5MPTOMS , $a' before an$ ( $a' after the a&&earan"e of rahe One <ee1 before an$ fo#r $a' after the a&&earan"e of rahe PRE!ERUPTI.E STAGE PRE!ERUPTI.E STAGE Cough Coryza Con1unctivitis Fever $high-grade& Photopho!ia Fever 0eadache Malaise Coryza Con1unctivitis COPLICDS SPOT ,R.6#$a- & 3luish white spots surrounded by a red halo & Appear on the buccal mucosa opposite the premolar teeth FORCHEIMERDS SPOTS ,R.6$$a- & small, red lesions & "oft palate to mucus membrane GERMAN MEASLES MEASLES SIGNS AND S5MPTOMS @= ERUPTI.E STAGE ERUPTI.E STAGE Rahe - Ele%ate$ &a&#le - Begin on the fa"e and !ehind the ears - 2pread to trunk and etremities (olor' Dar1 re$ 8 &#r&lih h#e 8 'ello< bro<n += Stage of Con%ale"en"e - De-#amation - Rahe fa$e from the fa"e $o<n<ar$ )= Rah - &in1ih+ maculopapular - Begin on the fa"e - 2pread to trunk or lim!s ! No &igmentation or $e-#amation @= Poterior a#ri"#lar an$ #bo""i&ital l'm&ha$eno&ath' GERMAN MEASLES MEASLES COMPLICATIONS Pneumonia 3. Encephalitis Otitis media Severe diarrhea $leading to dehydration& Encephalitis @= Congenital r#bella 'n$rome - 2pontaneous a!ortion - Intrauterine gro%th retardation $I4#R& - .hrom!ocytopenia purpura Kbl#eberr' m#ffin 1inL - (le"t lip+ cle"t palate+ clu! "oot - /eart de"ects $PDA+ 52,& - Eye de"ects $Catara"t+ glaucoma& - Ear de"ects $,ea"ness& - 6eurologic $mi"ro"e&hal'+ mental retardation+ !ehavioral distur!ances GERMAN MEASLES MEASLES TREATMENT MODALITIES 3. 5itamin - ) helps prevent eye damage and !lindness 3. -spirin ) help reduce in"lammation and "ever 2. -ntipyretics ) "or "ever 7. Penicillin ) given only %hen secondary in"ection sets in GERMAN MEASLES MEASLES NURSING MANAGEMENT )= Dar1ene$ room to relie%e &hoto&hobia @= Diet2 ho#l$ be li-#i$ b#t no#rihing += ;arm aline ol#tion for e'e to relie%e e'e irritation ,= For fe%er2 te&i$ &onge bath an$ anti! &'reti" (= S1in "are2 $#ring er#&ti%e tage3 oa& i omitte$M bi"arbonate of o$a in <ater or lotion to relie%e it"hine 7= Pre%ent &rea$ of infe"tion2 re&irator' iolation SCABIES MAIN PROBLEM nfestation of the s%in produced by the burrowing action of a parasite mite resulting in s%in irritation and formation of vesicles and pustules ETIOLOGIC AGENT "arcoptes scabiei CD-Bucud 8 INCUBATION PERIOD Hithin DA hours MODE OF TRANSMISSION Direct contact ndirect contact Sarcoptes scabiei %8 Y$$#9*s( 9(*& *+ c#$#' ;8 Ba'$! s+ 6! &( .+a*00 ! 38 Fma$ )a'as*& 6.''#9s 6+a&( &( )*0'm*s &# $a! ""s =8 Ma$s a' sma$$' a+0 's*0 #+ &( s.'fac #f &( sk*+ SIGNS AND SYMPTOMS T hin, pencil&mar% lines on the s%in tching, especially at night ,ashes and abrasions on the s%in PRIMARY LESIONS NODULAR LESIONS SECONDARY LESIONS TREATMENT MODALITIES "CA3CD# ; #ura0 ointment 8Crotamiton9 (#DC!'CD# ; ?well lotion 8Gamma 3en<ene -e0achloride9 ) contraindicated in young children and pregnant women Topical steroids -ydrogen pero0ide ; cleanliness of wound 'indane 'otion NURSING MANAGEMENT Apply cream at bedtime, from nec% to toes nstruct patient to avoid bathing for K to FD hours Dry&clean or boil bedclothes ,eport any s%in irritation $amily members and close contact treatment Good handwashing Terminal disinfection SEXUALLY TRANSMITTED DISEASES SYPHILIS AIDS MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD Final an$ mot erio# tage of 0I. $ieae3 <hi"h "a#e e%ere $amage to the imm#ne 'tem Infe"tio# $ieae "a#e$ b' a &iro"hete Retro%ir# 8 0#man T!"ell l'm&hotro&i" %ir# III D0TL.!+E Tre&onema &alli$#m + to 7 month to B to )* 'ear )*!N* $a' SYPHILIS AIDS MODE OF TRANSMISSION Seual contact ) oral+ anal or vaginal se Blood trans"usion Mother-to-child Indirect contact through soiled articles SYPHILIS AIDS SIGNS AND S5MPTOMS OPPORTUNISTIC INFECTIONS )= Pne#mo"'ti "arinni &ne#monia @= Oral "an$i$iai += To4o&lamoi ,= A"#teC"hroni" $iarrhea (= P#lmonar' t#ber"#loi MALIGNANCIES )= :a&oi9 ar"oma @= Non!0o$g1in9 l'm&homa SYPHILIS AIDS SIGNS AND S5MPTOMS )= PRIMAR5 S5P0ILIS - C0ANCRE2 mall3 &ainle3 &im&le!li1e #l"eration on the &eni3 labia maOora3 minora an$ li& - Ma' er#&t in the genitalia3 an#3 ni&&le3 tonil or e'eli$ ! L'm&ha$eno&ath' SYPHILIS AIDS SIGNS AND S5MPTOMS @= SECONDAR5 S5P0ILIS - S1in rah - M#"o# &at"he - 0air lo - COND5LOMATA LATA2 "oale"ing &a&#le <hi"h form a gra'!<hite &la-#e fre-#entl' in 1in fol$ SYPHILIS AIDS SIGNS AND S5MPTOMS += TERTIAR5 S5P0ILIS ! ) to )* 'ear after infe"tion ! A&&ear on the 1in3 bone3 m#"# membrane3 URT3 li%er an$ toma"h - GUMMA2 "hroni"3 #&erfi"ial no$#le or $ee& gran#lomato# leion that i olitar'3 &ainle3 in$#rate$ CD-Bucud 9 SYPHILIS AIDS DIAGNOSTIC PROCEDURES )=ELISA )=Dar1 Fiel$ Ill#mination tet @= ;etern blot += RIPA ,= PCR @= Flo#re"ent Tre&onemal Antibo$' Abor&tion Tet += .DRL SYPHILIS AIDS TREATMENT MODALITIES )= Anti%iral - 2horten the clinical course+ prevent complications+ prevent development o" latency+ decrease transmission - Eample' Hi$o%#$ine DRetro%irE )= Peni"illin G Ben/athine - Dieae 6 ) 'ear2 @=, M #nit on"e in t<o inOe"tion ite - Dieae G ) 'ear2 @=, M #nit in @ inOe"tion ite 4 + $oe @= Do4'"'"line 8 if allergi" to &eni"illin += Tetra"'"line - if allergi" to &eni"illin - Contrain$i"ate$ for ®nant <omen GONORRHEA CHLAMYDIA MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD MODE OF TRANSMISSION P#r#lent inflammation of m#"o# membrane #rfa"e Se4#all' tranmitte$ $ieae "a#e$ b' a ba"teria Chlam'$ia tra"homati Neieria gonorrhea @!+ <ee1 DmaleE @!)* $a' Se4#al "onta"t2 Oral3 %aginal or anal e4 A'm&tomati" DfemaleE GONORRHEA CHLAMYDIA SIGNS AND S5MPTOMS ;omen Ab$ominal or &el%i" &ain Blee$ing after inter"o#re an$ in!bet<een mene Un##al %aginal $i"harge ;omen Blee$ing after inter"o#re B#rning enation $#ring #rination 5ello< or bloo$' %aginal $i"harge Men B#rning <ith #rination S<ollen3 &ainf#l teti"le Di"harge from the &eni ;hite3 'ello< or green &# "rom the penis GONORRHEA CHLAMYDIA COMPLICATIONS ;omen Pel%i" inflammator' $ieae E"to&i" ®nan"' Sterilit' Men E&i$i$'miti Sterilit' Ne<born ConO#n"ti%iti Otiti me$ia Pne#monia Ne<born Gono"o""al o&hthalmia GONORRHEA CHLAMYDIA TREATMENT MODALITIES )= A/ithrom'"in DHithroma4E )= Cefi4ime - Dr#g of "hoi"e !ecause o" single-dose treatment e""ectiveness and lo%er cost 2. Do4'"'"line - Se"on$ar' $r#g of "hoi"e ! Dr#g of "hoi"e !ecause o" oral e""icacy+ single dose 2. (ipro"loacin 7. (e"triaone 8. Erythromycin HERPES SIMPLEX CANDIDIASIS MAIN PROBLEM ETIOLOGIC AGENT INCUBATION PERIOD Mil$ #&erfi"ial f#ngal infe"tion A %iral $ieae "hara"teri/e$ b' the a&&earan"e of ore an$ bliter on the 1in Can$i$a albi"an 0er&e im&le4 %ir# t'&e ) an$ @ @!+ <ee1 @!)@ $a' HERPES SIMPLEX CANDIDIASIS MODE OF TRANSMISSION )= Rie in gl#"oe a in $iabete mellit# @= Lo<ere$ bo$' reitan"e a in "an"er += In"reae in etrogen le%el in ®nant <omen ,= Broa$!&e"tr#m antibioti" are #e$ T5PE ) - Re&irator' $ro&let - Dire"t e4&o#re to infe"te$ ali%a - :iing an$ haring #tenil T5PE @ - Se4#al or genital "onta"t SIGNS AND SYMPTOMS ,Ca+0*0*as*s- ONYCHOMYCOSIS ,ed, swollen dar%ened nailbeds (urulent discharge "eparation of pruritic nails from nailbeds DIAPER RASH "caly, erythematous, papular rash Covered with e0udates CD-Bucud 10 Appears below the breasts, between fingers, a0illa, groin and umbilicus THRUSH Cream&colored or bluish&white patches on the tongue, mouth or pharyn0 3loody engorgement when scraped MONILIASIS Hhite or yellow discharge (ruritus 'ocal e0coriation Hhite or gray raised patches on vaginal walls with local inflammation HERPES SIMPLEX CANDIDIASIS TREATMENT MODALITIES )= Antif#ngal - Fl#"ona/ole DDifl#"anE - :eto"ona/ole DNi/oralE - Imi$a/ole DN'tatinE - Ue$ for oral thr#h - ,B ho#r #ntil 'm&tom $ia&&ear - Cotrimo4a/ole )= Anti%iral ! A"'"lo%ir DHo%ira4E CD-Bucud 11 VECTOR-BORNE DISEASES MALARIA DENGUE MAIN PROBLEM ETIOLOGIC AGENT -n acute "e!rile disease -n acute and chronic parasitic disease .he most common ar!oviral illness transmitted glo!ally .he most deadly vector-!orne disease in the %orld ,engue virus types 3+ 2+ 7 and 8 (hikungunya virus O9nyong9nyong virus :est 6ile virus Plasmodium "alciparum Plasmodium viva Plasmodium ovale Plasmodium malariae MALARIA DENGUE INCUBATION PERIOD MODE OF TRANSMISSION P. ;alciparum ) 32 days P. 5iva ) 38 days P. Ovale ) 38 days P. <alariae ) 7= days 7-38 days >ite o" an in"ected mosquito >lood trans"usion+ contaminated syringe or needle .rans-placentally MALARIA DENGUE .ECTOR Ae$e aeg'&ti DAe$e albo&i"t#E :hite stripes on the !ack and legs $.iger mosquito& ,ay !iting $2 hours a"ter sunrise and 2 hours !e"ore sunset& >reeds on clear stagnant %ater 4r!an-!ased Ano&hele fla%irotri >ro%n in color 6ight !iting $? P<-7 -<& >reeds on clear+ "lo%ing and shaded streams Rural-!ased MALARIA DENGUE SIGNS AND S5MPTOMS FE.ER C0ILLS PROFUSE S;EATING FE.ER 0EADAC0E MALAISE RAS0 EPISODES OF BLEEDING MALARIA DENGUE DIAGNOSTIC PROCEDURES )= TORNIFUET TEST - 2creening test "or dengue - - test "or the tendency "or !lood capillaries to !reak do%n or produce petechial hemorrhage - Per"ormed !y eamining the skin o" the "orearms a"ter the arm veins have !een occluded "or @ minutes - .o detect unusual capillary "ragility )= CLINICAL DIAGNOSIS - >ased on triad symptoms+ @=A accuracy @= PLATELET COUNT - (on"irmatory test "or dengue - ,ecreased count is con"irmatory @= BLOOD SMEAR - ,e"initive diagnosis o" in"ection is !ased on demonstration o" malaria &araite in !lood "ilm += RAPID DIAGNOSTIC TEST - 4ses immunochromatographic methods to detect Plasmodium- speci"ic antigen - .akes a!out 0 to 3@ minutes - 2ensitivity and speci"icity B ?=A MALARIA DENGUE TREATMENT MODALITIES )= Analgei" an$ anti&'reti" ! a"etamino&hen @= .ol#me e4&an$er - Ue$ in the treatment of intra%a"#lar %ol#me $efi"it - E4am&le2 La"tate$ Ringer += Bloo$ tranf#ion 8 for e%ere blee$ing ,= O4'gen thera&' (= Se$ati%e )= Chloro-#ine @= Prima-#ine += P'rimethamine ,= S#lfa$o4ine (= F#inine 7= F#ini$ine LEPTOSPIROSIS SCHISTOSOMIASIS MAIN PROBLEM ETIOLOGIC AGENT A lo<l' &rogrei%e $ieae "a#e$ b' a bloo$ fl#1e A /oonoti" infe"tio# $ieae )= SC0ISTOSOMA >APONICUM - Intestinal tract+ endemic in the Philippines @= SC0ISTOSOMA MANSONI - -"rica += SC0ISTOSOMA 0AEMATOBIUM - <iddle East countries like Iran and Iraq Le&to&ira interrogan LEPTOSPIROSIS SCHISTOSOMIASIS INCUBATION PERIOD MODE OF TRANSMISSION At leat @ month A to )N $a' Ingetion S1in &enetration Conta"t <ith the 1in CD-Bucud 12 LEPTOSPIROSIS SCHISTOSOMIASIS .ECTOR On"omelania -#a$rai )= Thri%e in freh <ater tream @= Cling to grae an$ lea%e += Greenih bro<n in "olor ,= Si/e i a big a the mallet grain of &ala' LEPTOSPIROSIS SCHISTOSOMIASIS SIGNS AND S5MPTOMS ACUTE STAGE )= Cer"arial $ermatiti D<immer9 it"hE @= :ata'ama 'n$rome C ! o#gh 0 8 ea$a"he an$ fe%er A 8 nore4ia an$ letharg' R 8 ah M ! 'algia Se&ti" or Le&to&iremi" Stage F 8 e%er Dremittent 0 8 ea$a"he M8 'algia N 8 a#ea . 8 omiting C 8 o#gh C 8 het &ain LEPTOSPIROSIS SCHISTOSOMIASIS SIGNS AND S5MPTOMS C0RONIC STAGE )= 0e&ati"2 &ain3 ab$ominal $itenion3 hematemei3 melena @= Intetinal2 fatig#e3 ab$ominal &ain3 $'enter' += Urinar'2 $'#ria3 #rinar' fre-#en"'3 hemat#ria ,= Car$io&#lmonar'2 &al&itation3 $'&nea on e4ertion (= CNS2 ei/#re3 hea$a"he3 ba"1 &ain an$ &aretheia Imm#ne or To4i" Stage ! Lat for , to +* $a' ! Iriti3 hea$a"he3 meningeal manifetation ! Olig#ria3 an#ria <ith renal fail#re ! Sho"13 "oma an$ "ongeti%e heart fail#re LEPTOSPIROSIS SCHISTOSOMIASIS DIAGNOSTIC PROCEDURES )= Fe"al'i @= :ato!:at/ Te"hni-#e += Cer"#m o%a &re"i&itin tet DCOPTE ! Confirmator' tet for "hitoomiai LEPTOSPIROSIS SCHISTOSOMIASIS TREATMENT MODALITIES )= Pra/i-#antel DBiltri"i$eE ! Ta1en for 7 month ! ) tablet BID for + month ! ) tablet OD for + month ) t line $r#g )= Peni"illin G 8 $r#g of "hoi"e @= Do4'"'"line @ n$ line $r#g += Am&i"illin ,= Amo4i"illin FILARIASIS MAIN PROBLEM A parasitic disease caused by an African eye worm ETIOLOGIC AGENT Huchereria bancrofti 3rugia malayi 3rugia timori INCUBATION PERIOD K to FI months MODE OF TRANSMISSION (erson&to&person by mos+uito bites AC!T# "TAG# L!m)(a0+*&*s 8inflammation of lymph nodes9 L!m)(a+"*&*s 8inflammation of lymph vessels9 *ale genitalia affected leading to funiculitis, epididymitis and orchitis 8redness, painful and tender scrotum9 C-,ONC "TAG# Develop FG&FB years from onset of first attac% H!0'#c$ 8swelling of the scrotum9 L!m)(0ma 8temporary swelling of the upper and lower e0tremities9 E$)(a+&*as*s 8enlargement and thic%ening of the s%in of the upper and lower e0tremities, scrotum and breast 'A3O,ATO,5 #4A*NATON" N#c&.'+a$ 6$##0 7am*+a&*#+ ,NBE- ) ta%en at patient>s residence2hospital after K(* Imm.+#c('#ma&#"'a)(*c &s& ,ICT- ) rapid assessment method: an antigen test done at daytime T,#AT*#NT D*&(!$ca'6ama/*+ C*&'a& ,DEC- #' HETRABAN ) an individual treatment %ills almost all microfilaria and a good proportion of adult worms= (,#.#NTON AND CONT,O' *easures aimed to control vectors E+1*'#+m+&a$ sa+*&a&*#+ such as proper drainage and cleanliness of surroundings S)'a!*+" with insecticides (,#.#NTON AND CONT,O' *easures aimed to protect *+0*1*0.a$s a+0 fam*$*s? !se of m#s<.*&# +&s !se of $#+" s$1s, $#+" )a+&s and s#cks Application of *+sc& ')$$a+&s Sc'+*+" of houses CD-Bucud 13