Professional Documents
Culture Documents
EDITORS NOTE: This article is the second of six that will be published in 2002 for which a total of up to 6 Category
1 CME credits can be earned. Instructions for how credits can be earned appear on the last page of the Table of Contents.
INTRODUCTION
Good airway management in the pediatric emergency department (ED) can mean the difference between an acceptable outcome and permanent disability or death. Airway management in
the critically ill child is a challenge for several reasons: it occurs
rarely, is often unanticipated, and must be achieved with little time
to plan in children with limited respiratory reserve. Moreover, the
emergency physician must sometimes provide airway management without the benet of fully knowing the extent of the patients injuries or medical conditions. Antecedent conditions (eg,
full stomach, shock, elevated intracranial pressure, and cardiovascular disease) complicate management and may not be immediately evident. As a consequence, the emergency physician must
choose strategies that are likely to succeed with few potential
complications.
Patients in the ED who require urgent or emergent airway management usually present with little advance warning; hence, there is
limited opportunity to mobilize specialized personnel and equipment before the patients arrival. This is different from the operating room where an anesthesiologist faced with a difficult airway can
usually get help from several anesthesiologists and has an array of
airway adjuncts. Elective surgical cases complicated by airway difculty can be managed by canceling surgery until well-planned alternative techniques are available. The emergency physician usually
cannot return at another time with a better strategy.
Optimal outcomes with pediatric airway management require a
thorough understanding of the physiologic, pharmacodynamic, and
anatomic differences between children and adults. Additionally,
good preparation, difficult airway recognition, and familiarity with
back-up plans for airway management are essential. This review is
intended for emergency physicians and, thus, will focus on devices
and techniques easily obtained and used in the ED.
TARGET AUDIENCE
This CME activity is intended for physicians, nurses, nurse practitioners, physician assistants, and paramedics who care for children in respiratory distress or cardiopulmonary arrest. Specialists
including pediatricians, emergency physicians, pediatric emergency physicians, anesthesiologists, and trauma surgeons will nd
this information particularly useful.
LEARNING OBJECTIVES
After completion of this article, the reader will
1. Be able to recognize the difficult airway and make the preparations necessary for the clinical scenario.
2. Be able to discuss the role of neuromuscular blocking agents
and induction drugs in airway management.
3. Be able to describe the management of the uncomplicated airway using manual ventilation with a bag and mask or direct
laryngoscopy.
*Pediatric Anesthesiologist and Intensivist, Departments of Pediatric
Anesthesia and Critical Care, Nemours Childrens Clinic, and Professor and
Chief, Division of Critical Care Medicine, University of Florida, Jacksonville,
Florida.
Address for reprints: Niranjan Kissoon, MBBS, Professor and Chief, Division of Critical Care Medicine, University of Florida Health Sciences
Center, 820 Prudential Drive, Suite 203, Howard Building, Jacksonville,
FL 32207; e-mail: niranjan.kissoon@jax.u.edu
Key Words: Airway management, intubation, emergency resuscitation,
respiratory failure
PREPARATION
The rst step toward success in securing a childs airway is appreciation of the unique physiologic and anatomic differences between
108
TABLE 1
Respiratory physiologic differences between children and adults
Increased respiratory rate
Increased chest wall compliance
Decreased lung elastic recoil (lower lung compliance)
Diminished functional residual capacity
Increased rate of oxygen consumption
109
TABLE 2
and intubation are difficult or impossible. This is particularly relevant to the ED, where the clinician may not have the luxury of a
thorough airway assessment.
Recognition of the Difficult Airway. It is critical to recognize
the difficult airway before using induction agents and neuromuscular blockade, because failure to do so can result in a life-threatening
situation in which ventilation and intubation are impossible. Choosing alternative awake, asleep, or spontaneously breathing techniques for airway management in children with difficult airways can
be the difference between a smooth intubation and a fatal airway
ail.
In the absence of specic anatomic markers of airway abnormality, facial and upper airway trauma, or inammation, it is exceedingly unlikely to be surprised by a difficult airway. A history
of difficult airway in the patient can be identied from Medic-Alert
bracelets or from the caretakers of children with conditions known
to predispose to airway difficulty. A prior history of airway treachery is a red ag to heed, particularly if the difficulties occurred in
the hands of clinicians who are experienced in the airway management of children. Physical ndings that predict airway difficulty
are summarized in Table 3 and are discussed below.
Oropharyngeal Examination. The oropharyngeal examination
is the rst step in assessing the airway. The patients oral cavity is
examined with his or her mouth open and the tongue maximally
protruded. The degree of mouth opening and the size of the tongue
relative to the oral cavity are assessed. Mallampati et al. (5) classied airways on the basis of the degree of visualization of the faucial pillars, soft palate, and uvula. The ability to see a large part of
these structures predicts a high probability of adequate laryngeal
visualization in adults. Whether the Mallampati et al. score can successfully predict the degree of difficulty with endotracheal intubation in small children is not known.
Macroglossia, an absolute or relative enlargement of the tongue
in relation to the oral cavity, is a prominent feature of Down and
Beckwith-Wiedemann syndromes and is associated with airway
difficulty. Inltration or crowding of the tongue and airway structures is commonly seen in mucopolysaccharidosis, morbid obesity,
cystic hygroma, edema, and cellulitis. The presence of a high,
arched palate is also associated with airway difficulty.
TABLE 3
Findings that predict the presence of a difficult airway
Limited mouth opening
Cervical spine immobility (immobilization, trauma, degenerative
processes)
Small mouth
Prominent central incisors
Short mandible
Short neck
Large tongue (relative or absolute)
Obese patients
Laryngeal edema (infection, inhalation thermal injury)
Mandibular, midface, and facial trauma
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April 2002
TABLE 4
Suggested airway cart equipment for the emergency department
Laryngoscope blades of all sizes and styles and Magill forceps
Endotracheal tubes of all sizes, cuffed and uncuffed
CO2 detector (adult and pediatric)
Facemasks (neonate to adult)
All sizes of naso- and oropharyngeal airways
Suction equipment and catheters
Self-inating resuscitation bags
Endotracheal tube guides
Semi-rigid intubating stylet (some hollow variants facilitate jet
ventilation)
Light wand
Flexible beroptic intubation equipment
Emergency nonsurgical ventilation
Transtracheal jet ventilation
Laryngeal mask airway
Tracheoesophageal combitube
Emergency surgical airway access
Cricothyrotomy equipment
Needle (14-gauge catheter over needle, 3.0 ID endotracheal tube
adapter)
Seldinger kits available for children younger than 910 years
The expertise and experience of the ED clinician should dictate the contents of the airway cart.
Adapted from Practice guidelines for management of the difficult airway:
a report by the American Society of Anesthesiologists Task Force on the
Management of the Difficult Airway. Anesthesiology 1993; 78:597602.
children with intracranial hypertension. It is given as an intravenous bolus of 1 to 2 mg/kg 2 to 3 minutes before laryngoscopy,
and it has direct anesthetic properties on the central nervous system
(17). Lidocaine can also be nebulized or sprayed onto airway structures or into the trachea to diminish the patients response to laryngoscopy and intubation. Concentrations of 1%, 2%, and 4% can be
given in volumes calculated to keep the total dose below the toxic
limit of 5 mg/kg.
Opioids and benzodiazepines do not reliably produce unconsciousness unless they are administered in large quantities. In such
doses, they often also produce apnea. As such, these medications
are best used as adjunct therapies for amnesia and analgesia with
other induction drugs. Additionally, these medications may be used
for conscious sedation in the spontaneously breathing patient who
is to be kept awake and responsive during endotracheal intubation.
Opioids produce sedation and blunt the response to noxious airway
stimuli, whereas benzodiazepines induce sedation and amnesia.
After adequate topical anesthesia of the airways, short-acting medications such as midazolam in 0.05 to 0.1 mg/kg increments and
fentanyl in 1 to 2 g/kg increments can be given to render the patient less anxious and more comfortable.
Synergistic respiratory depression results from concomitant administration of opioids and benzodiazepines; however, with careful
titration of these medications, it is possible to avoid respiratory depression. Should respiratory depression or excessive somnolence
occur, these are the only medications for which reliable and rapid
antagonists exist and allow reversal if spontaneous ventilation is
threatened. Slow titration of these agents will allow the clinician to
avoid excessive respiratory depression and the chest wall rigidity
that has been described with the rapid administration of large quantities of fentanyl to children (18). Owing to differences in drug protein binding, central nervous system perfusion and permeability,
and receptor affinity, the risk for respiratory depression and apnea
is greater in neonates (1921).
Anticholinergic medications including glycopyrrolate, scopolamine, and atropine have several applications in the management
of the difficult pediatric airway. They cause tachycardia and drying
of oral secretions, and scopolamine also causes amnesia. Neonates
have a tendency to respond to laryngoscopy or hypoxemia with a
dramatic slowing of the heart rate as a result of parasympathetic
nervous system activation. Because stroke volume cannot be augmented appreciably in neonates, cardiac output is dependent on
heart rates that are neither excessively fast nor slow (22). Neonates
are, therefore, good candidates for premedication with anticholinergic medications, especially when succinylcholine is used. Many
investigators recommend premedication with atropine for all children younger than 10 years (17). Atropine sulfate in an intravenous
dose of 0.02 mg/kg with a minimum dose of 0.1 mg can be given
to prevent bradycardia during airway management. Atropine sulfate can also be given intramuscularly in a dose of 0.04 mg/kg. The
site of intramuscular injection affects the rate of response to atropine, with lingual injection producing faster response than deltoid or vastus lateralis injection (23). Anticholinergic medications,
such as glycopyrrolate (0.01 mg/kg), are given to dry oropharyngeal secretions to optimize visualization during beroptic techniques of airway management. This is optimally given 30 minutes
before airway management is attempted and, therefore, may be of
limited use in emergency airway management. Scopolamine is also
a potent antisialagogue and has the added benet of producing sedation and amnesia.
Defasciculating doses of nondepolarizing neuromuscular blockers (NMBs) are administered to patients for two reasons. Before
111
112
TABLE 5
Selected potential deleterious effects
associated with the use of succinylcholine
Hyperkalemia
Myopathy
History of malignant hyperthermia
Denervating injury or disease process ( 23 d, lasting for 36 mo or
longer)
Recent burns (more than 24 h and less than 6 mo since injuryrisk
believed to decrease with healing)
Crush injuries
Abdominal abscesses
Arrythmias
Increased intracranial pressure
Increased intraocular pressure
Myalgias
Increased intragastric pressure
April 2002
four returns in approximately 17 minutes and can be pharmacologically antagonized with neostigmine if necessary (37). Like succinylcholine, rocuronium can be administered intramuscularly in
the absence of intravenous access (38). In high doses, it sometimes
causes tachycardia. Rocuronium is best used for RSI when there is
a reason to avoid succinylcholine, and the airway is not anticipated
to be difficult. Rocuronium is metabolized and excreted by the liver
and can be antagonized by neostigmine.
SEDATIVE AND INDUCTION DRUGS. Sedative and induction drugs
produce unconsciousness and amnesia and facilitate endotracheal
intubation. A detailed discussion of the pharmacodynamics and
pharmacokinetics of the induction agents is beyond the scope of
this report, but the salient features are reviewed briey. Sodium
thiopental (STP) reliably produces unconsciousness, amnesia, and
apnea in induction doses of 4 to 7 mg/kg intravenously (40).
Lower doses are required in neonates because of decreased protein
binding (41). The clinical effects of STP dissipate rapidly because
of redistribution from the central nervous system to fat and muscle, which explains why a medication with an elimination half-life
of 3 to 10 hours in children is clinically effective for only 20 minutes (42). After repeated doses of STP are administered, saturation
of muscle and fat sites occurs and redistribution no longer takes
place.
Induction doses of STP lower intracranial pressure more than
mean arterial pressure (43). Thus, cerebral perfusion pressure is preserved or improved, whereas the medication decreases the cerebral
rate of oxygen consumption (43). STP is also a potent antiepileptic
drug even in small doses, but it is of short duration because of redistribution. STP is a direct myocardial depressant, causing tachycardia
by a central vagolytic mechanism, venous capacitance vessel pooling of blood, and reduced cardiac output (43). Patients with cardiomyopathy, profound hypovolemia, and insufficient baroreceptor
reexes can experience profound hypotension from customary induction doses of STP. Therefore, if STP is to be used in these circumstances, smaller doses should be titrated cautiously.
Ketamine can be considered to be the opposite of STP with respect to its hemodynamic prole. Its usual intravenous dose of 1 to
2 mg/kg increments produces dissociative analgesia and amnesia.
Intramuscular administration is unpredictable, but an intramuscular
dose of 4 to 6 mg/kg is usually sufficient. Ketamine increases heart
rate, blood pressure, and cardiac output through its stimulation of
central sympathetic nervous system outow (26). It is a direct myocardial depressant (44) and must be used with caution in patients
with depleted catecholamine reserves (severe end-stage shock) in
whom deleterious hemodynamic effects may be observed. As opposed to all of the other induction drugs, respiratory drive is relatively preserved with ketamine (26), although intact laryngeal reexes cannot be presumed to be present. Patients ventilate
spontaneously, which makes this an attractive sedative for procedures in unintubated patients in whom it is preferable to maintain
spontaneous ventilation. However, preservation of spontaneous
ventilation cannot be assured in neonates receiving ketamine or in
older children who are premedicated with large doses of opioid
medications. Ketamine is a potent bronchodilator and may be the
drug of choice for intubation of the severe asthmatic. As a potent
sialogogue, however, concomitant administration of an anticholinergic medication should be considered to prevent copious airway
and salivary secretions. In contrast to STP, the central nervous system effects of ketamine include increased cerebral metabolic rate,
intracranial pressure, and cerebral blood ow (43). As such, it is a
poor choice for the patient with increased intracranial pressure.
Emergence delirium is less common in children than adults. How-
113
airow obstruction during mask ventilation can be bypassed by introducing a laryngeal mask airway (LMA) or Combitube (Kendall,
Manseld, MA) (for larger children). If this is unsuccessful or unavailable, the next step is to proceed immediately to invasive airway
management. For the profoundly hypoxemic patient who is in extremis, the clinician should proceed directly to needle cricothyrotomy in children, because this is the technique that is most likely to be
rapidly successful.
The nal algorithm, the crash airway algorithm (Fig. 1E), provides some recommendations for airway management in the patient
who is critically ill or unstable. It acknowledges that it may not be
necessary to provide pharmacologic assistance for the patient who
is already moribund and unlikely to react to or recall the airway intervention. We advocate immediate initiation of bag-mask ventilation before the rst attempt at endotracheal intubation. This may
restore some physiologic stability and prevent or forestall cardiovascular collapse. Likewise, for the patient with intracranial hypertension, mask ventilation is the most effective in rapidly lowering
intracranial pressure and improving cerebral blood ow. We suggest using STP or etomidate plus lidocaine and a muscle relaxant if
the patient is moribund and intracranial hypertension is present. For
other patients, laryngoscopy and intubation can be performed without pharmacologic assistance.
It should be emphasized that algorithms are helpful; however,
clinical judgment, experience, and expertise should guide the individual approach. Maintaining spontaneous ventilation (if it is physiologically adequate) in the difficult airway scenario is critical. Finally, it is important to recognize when airway interventions are
ineffective. At this point, failure to proceed to another translaryngeal or transtracheal form of support may result in an adverse patient outcome.
The single most valuable asset available to the clinician is prociency at bag-mask ventilation. This is the technique that will buy
time and preserve life while other airway equipment, personnel, and
techniques can be mobilized. Poor technique results in hypoventilation, gastric insufflation with air, and subsequent aspiration of gastric contents. Good technique involves preserving good maskface
seal, inating the chest with the minimal required pressure, maintaining the optimal patency of the upper airway through manipulation of the mandible and cervical spine (when not contraindicated),
and insertion of oropharyngeal or nasopharyngeal airways (when
not contraindicated) to maintain patency of the upper airway. Good
mask technique, positioning, and gentle cricoid pressure are required to help minimize the risks of gastric aspiration. Cricoid pressure must be applied gently (depress the trachea 12 cm) in infants,
because the pliable trachea can be completely occluded by the Sellick maneuver if performed with too much pressure.
In most children, placement of the endotracheal tube under direct
visualization with the RSI technique is all that is needed. All attempts at direct laryngoscopy should be gentle and brief. Failure to
do so will result in further airway bleeding and edema and may exacerbate intracranial hypertension. Indeed, the most common scenario leading to severe injury in lost airway scenarios is progressive
airway compromise as a result of repeated iatrogenic trauma and
failure to abandon a technique that has proven to be ineffective.
The Uncomplicated Pediatric Airway. Patients in the ED
can generally be assumed to have a full stomach. Rapid sequence
induction is an airway strategy that is designed to minimize the risk
of aspiration of gastric contents. In this scenario, the patient is preoxygenated by breathing spontaneously, and induction drugs and
paralytics are injected in rapid sequence. The patient is allowed to
become apneic without ventilating with positive pressure. As soon
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April 2002
B
FIG. 1. A, Global algorithm for pediatric airway management in the emergency department. B, Management of the straightforward pediatric airway. (continued)
115
D
FIG. 1. (Continued ) C, Difficult airway algorithm. Options for endotracheal tube placement while the patient is breathing spontaneously include, but are not
limited to, awake direct laryngoscopy, blind nasal (children 10 y), beroptic methods (Bullard, exible bronchoscope), intubating laryngeal mask airway, light
wand, retrograde techniques. D, Lost airway algorithm. (continued )
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April 2002
E
FIG. 1. (Continued ) E, Crash airway algorithm. Adapted from Walls RW. The emergency airway algorithms. In: Manual of emergency airway management.
Philadelphia: Lippincott Williams & Wilkins, 2000;1626.
tions for accomplishing this include generous topical airway anesthesia followed by awake laryngoscopy, beroptic bronchoscopy,
blind nasotracheal intubation, light wand intubation, and retrograde
techniques. The choice of technique will depend on the preference
and condence level of the practitioner, age and degree of cooperation of the child, and the specic clinical circumstances. It may
also be appropriate to involve other health care professionals
skilled in airway access to assist if time permits.
For patients who are likely to be easy to bag-mask ventilate, but difcult to intubate endotracheally, other approaches are possible. In this
situation, the risk of gastric aspiration during airway manipulations
must be weighed against the increased cooperation gained by administering sedatives and/or paralytic agents. In agitated and uncooperative patients, the risk benet ratio may favor sedation and/or paralysis. The favorable conditions gained may more than offset the risk of
aspiration. In addition to the techniques described previously, the
spontaneously breathing patient can also be taken to the operating
room and intubated while spontaneously inhaling volatile anesthetics.
In the ED there will be patients with difficult airways and limited
physiological reserve who are in need of immediate airway access.
Approaches in such a setting include the quick conrmation that
one can neither mask ventilate nor endotracheally intubate the
patient. If neither can be done, it is appropriate to place a temporizing airway (eg, LMA, Combitube) or proceed directly to an invasive airway technique.
Options for invasive airway management in children in the ED include needle cricothyrotomy and/or placement of a larger cricothyroid tube via the Seldinger technique. Needle cricothyroidotomy is
the easiest and safest technique for temporary ventilatory support in
the ED and should be considered the technique of choice. The technique for gas exchange is dictated by patient age. Bag ventilation or
transtracheal jet ventilation (TTJV) with the pressure regulator set to
a low pounds per square inch (PSI) can be used to temporarily restore
oxygenation while a more denitive airway is pursued (52). However, TTJV should be used only by those who are skilled and comfortable with the technique.
Despite all of the opinions and reviews written on this subject,
few clinicians actually have experience applying technique in children. The cricothyroid membrane is small and may be difficult to
palpate in infants and smaller children. In such instances, it is appropriate to place an intravenous catheter through the trachea in the
region of the cricothyroid membrane. Oxygenation and lifesustaining levels of ventilation can be sustained for an hour or more
in dogs with nearly complete upper airway obstruction. Gas exchange was provided by low-ow oxygen in spontaneously breathing animals and by positive pressure ventilation with a self-lling
bag in paralyzed animals (53).
Needle cricothyrotomy in children is a temporary measure and
is used to preserve oxygenation until a denitive airway can be obtained. Although oxygenation may be preserved at life-sustaining
levels with this technique, ventilation is often marginally adequate
for a nite time period, and careful attention must be paid to the
rise and fall of the chest and oxygenation. Complete, or high-grade
proximal airway obstruction, can result in inadequate exhalation
and air trapping, leading to barotrauma and pneumothoraces.
Complications from errant placement of the needle and catheter
include pneumothorax, subcutaneous emphysema resulting in the
loss of favorable conditions for subsequent attempts at correct
catheter placement, mediastinal emphysema, esophageal injury,
and bleeding. In addition, ventilation with a self-lling resuscitation bag may be difficult because of the high resistance imposed
by the small intravenous catheter in the airway. It may be necessary to disable the pop-off valve on the self-lling bag to optimize
ventilation. Finally, TTJV requires the use of specialized equipment and may cause injury if used improperly (54). Careful monitoring of the rise and fall of the chest, oxygen saturations, and
carbon dioxide tensions on blood gases is mandatory. Tracheostomy (with surgical consultation in ED or operating room) or
translaryngeal endotracheal intubation from above can then be attempted after oxygenation is ensured by needle cricothyrotomy.
Commercial kits are available for needle cricothyrotomy with subsequent passage of a guidewire into the trachea, passage of serial
dilators, and placement of a cricothyrotomy tube. The wider bore
cricothyrotomy tube is much more able to facilitate ventilation
than the needle catheter; however, such kits exist only for children
older than 10 years.
The Unanticipated Difficult Airway. Despite attempts to
screen for and predict airway difficulties, the difficult airway may
be rst recognized during the laryngoscopy phase of RSI. The next
step is to immediately determine whether the patient can be ventilated with bag and mask. If the patient can be ventilated, the techniques available include optimizing the position and allowing the
most skilled individual present to repeat laryngoscopy. If the patient cannot be intubated, alternative techniques of airway management include beroptic intubation, Bullard laryngoscopy, light
wand, retrograde techniques, and placement of LMA with blind or
beroptic placement of an endotracheal tube through the LMA.
Other options are needle cricothyrotomy with bag or transtracheal
jet ventilation or the placement of a cricothyrotomy tube via the
Seldinger technique (patient age and size permitting).
If the patient cannot be ventilated, urgent action is needed to es-
117
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April 2002
TABLE 6
Suggested sizes for laryngeal mask airways in children
Mask size
1
2
2.5
3
4
Patient size
Weight (kg)
Infant
Child
Child
Small adult
Normal and large adult
6.5
6.520
2030
30
2 to 4
10
15
20
30
B
FIG. 2. A, Laryngeal mask airways (LMA) of different sizes. B,
Schematic drawing of proper placement of LMA: side view of LMA (A),
insertion of LMA with head in sniffing position while pressing device
along hard palate (B), advancement of LMA against posterior pharynx until seated (C), correctly seated LMA (D). Reprinted with permission from
Morgan GE Jr, Mikhail MS. Airway management. In: Clinical anesthesiology, ed 2. Stamford, CT: Appleton-Lange, 1996; 56.
LMA, it is more likely that the patient can be ventilated to some degree but cannot be intubated by the translaryngeal route under direct visualization. This is common in patients with cervical spine
trauma, cervical spine fusion, limited mouth opening, and other
anatomic reasons contributing to the clinicians inability to align
the oral, pharyngeal, and laryngeal axes. In this situation, translaryngeal tracheal intubation is possible via the blind or indirect visualization techniques.
Indirect visualization techniques allow the clinician to see the
larynx indirectly using flexible fiberoptic bronchoscopy or a
Bullard laryngoscope. The Bullard laryngoscope is available in
adult and pediatric sizes (Fig. 4) and consists essentially of a
fiberoptic apparatus mounted on a handheld, L-shaped frame. It is
inserted into the oropharynx and allows the airway structures to
be directly visualized while the preloaded endotracheal tube is
passed off a wire stylet through the glottis under indirect visualization. This can be done when the patient is awake (with topical
anesthesia) or asleep with either spontaneous or positive pressure
mask ventilation (asleep and paralyzed). Flexible fiberoptic intubation entails the use of a variety of sizes (outer diameter and
length) of fiberoptic bronchoscopes over which an endotracheal
tube has been loaded. The bronchoscope can be introduced into
the airway via the oral or nasal routes and, likewise, can be done
when the patient is awake, asleep and breathing spontaneously, or
asleep and paralyzed. This can be accomplished via intermittent
attempts between mask ventilation with periods of apnea or it can
be performed through specialized facemasks that allow for continuous ventilation while flexible fiberoptic bronchoscopy is performed.
Blind techniques are methods in which the endotracheal tube is
placed into the trachea without direct or indirect visualization.
These techniques essentially include blind nasal techniques as well
as light wand intubations. Blind nasal intubation is easier in a spontaneously breathing patient. This technique involves the introduction of an endotracheal tube into the nasopharynx of the patient and
the advancement of the tube as the breath sounds in the endotracheal tube become louder. Commercially available endotracheal
tubes that incorporate a guiding ring and cable in the wall of the endotracheal tube make it easy to direct the tube in an anterior direction for easier insertion. This technique is not advisable in patients
119
with potential basilar skull fracture. Techniques to facilitate sedation with spontaneous ventilation include light benzodiazepineopioid sedation with topical airway anesthesia, inhalation anesthesia, or ketamine-benzodiazepine methods. Blind nasal intubation is
not likely to be successful in small children because of the anterior
location of the glottis and the small laryngeal aperture that is obscured by the epiglottis. This technique is not recommended for
routine use in children younger than 10 years (55).
Light wand techniques involve passing an endotracheal tube
from an illuminated intubating stylet into the trachea without direct
or indirect visualization. The light wand has a very bright light on
its tip that is blindly passed through the glottic aperture. Placement
is attempted when a very bright light is visible through the skin
overlying the thyroid and cricoid cartilage. The main pitfall of this
technique is that, even with esophageal placement of the stylet, a
very bright light can sometimes still be seen through the anterior
neck. It is important to practice this technique to be able to reliably
distinguish between correct and incorrect stylet positions so as to
pass the endotracheal tube without displacing the stylet. This technique works better in anesthetized patients.
The nal methods for translaryngeal intubation are collectively
referred to as retrograde techniques. This term encompasses a wide
variety of permutations of maneuvers but in its distilled form refers
to the placement of a guidewire into the airway via the cricothyroid
membrane or trachea. The wire is passed proximally through the
laryngeal aperture and retrieved from the oropharynx. The wire is
passed through the Murphy eye of the endotracheal tube, and the
endotracheal tube is advanced into the trachea over the guidewire.
Alternatively, the guidewire can be passed through the working port
of a bronchoscope over which an endotracheal tube has already been
loaded. The bronchoscope and endotracheal tube combination is advanced over the guidewire and placed in the trachea under indirect
visualization. These techniques work best with generous topical airway anesthesia or after the induction of anesthesia.
CONCLUSIONS
Proper airway management requires practice and judgment in
addition to an appreciation of the anatomic, pharmacologic, and
physiologic differences that separate infants and children from
adults. It is prudent to become procient at techniques for airway
management beyond simple direct laryngoscopy and endotracheal
intubation, even though most cases will require nothing more. The
clinician should strive to be familiar with many techniques, but
should become very adept at only a few.
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