Professional Documents
Culture Documents
COLLEGE OF NURSING
Cebu City, Philippines
Contents i
Introduction 1
Objectives 3
Nursing Assessment
Client’s Profile 4
Physical Assessment 5
Gordon’s Functional Health Patterns 6
Laboratory Findings 8
Anatomy and Physiology of the Female Reproductive System 10
Pathogenesis of Ectopic Pregnancy 12
With ectopic pregnancy, fertilization occurs as usual in the distal third of the uterine tube.
Immediately after the union of the ovum and the spermatozoon, the zygote begins to
divide and grow normally. Unfortunately, because an obstruction is present, such as
adhesion of the uterine tube from a previous infection (chronic salpingitis or pelvic
inflammatory disease), congenital malformations, scars from tubal surgery, or a uterine
tumor pressing the proximal end of the tube, the zygote cannot travel the length of the
tube. It lodges at the strictured site along the uterine tube and implants there instead of in
the uterus.
Assessment
With ectopic pregnancy, there are no unusual symptoms at the time of implantation. The
corpus luteum of the ovary continues to function as if the implantation were in the uterus.
No menstrual flow occurs. A woman may experience the nausea and vomiting of early
pregnancy, and pregnancy test for human chorionic gonadotrophin (hCG) will be positive.
A woman usually expediencies a sharp, stabbing pain in one of her lower abdominal
quadrants at the time of the rupture, followed by scan vaginal spotting. With placental
dislodgement, progesterone secretion stops and the uterine decidua begins to slough,
causing additional bleeding. The amount of bleeding evident with a ruptured ectopic
pregnancy often does not reveal the actual amount present, however, because the
products o conception from the ruptured tube and the accompanying blood may be
expelled into the pelvic cavity rather than into the uterus. Therefore, this blood does not
reach the vagina to become evident. If internal bleeding progresses to acute hemorrhage,
a woman may experience lightheadedness and rapid pulse, signs of shock.
When helping determine the possibility of an ectopic pregnancy, ask a woman whether
she has pain or vaginal bleeding. Any woman with sharp abdominal pain and vaginal
spotting needs to be evaluated by her health care provider to rule out the possibility of
ectopic pregnancy. Occasionally, a woman will move suddenly and move and pull one of
her round ligaments, the anterior uterine supports. This can cause a sharp, but
momentarily and innocent, lower quadrant pain. However, it would be rare for this
phenomenon to be reported in connection with vaginal spotting.
By the time a woman with a ruptured ectopic pregnancy arrives at the hospital of
physician’s office, she may already be in severe shock, as evidenced by rapid, thready
pulse, rapid respirations, and falling blood pressure. Leukocytosis may be present, not
from infection but from trauma. Temperature is usually normal. A transvaginal sonogram
will demonstrate the ruptured tube and blood collecting in the peritoneum. Either a falling
hCG or serum progesterone suggests that pregnancy has ended. If the diagnosis of ectopic
pregnancy is in doubt, a physician may insert a needle through the postvaginal fornix into
the cul-de-sac under sterile conditions to see whether blood can be aspirated. A
laparoscopy or culdoscopy can be used to visualize the uterine tube if the symptoms alone
do not reveal a clear picture of what has happened. However, sonography alone usually
reveals a clear-cut diagnostic picture.
If a woman waits before seeking help, gradually her abdomen becomes rigid from
peritoneal irritation. Her umbilicus may develop a bluish tinge (Cullen’s Sign). A woman
may have continuing extensive or dull vaginal and abdominal pain; movement on the
cervix on pelvic examination my cause excruciating pain. There may be pain in her
shoulders from blood in the peritoneal cavity causing irritation to the phrenic nerve. A
tender mass is usually palpable in Douglas’ cul-de-sac on vaginal examination.
Therapeutic Management
Although some ectopic pregnancies spontaneously end and then reabsorbed, requiring no
treatment, it is difficult to predict when this will happen, so when an ectopic pregnancy is
revealed by an early sonogram, some action is taken. If an ectopic pregnancy can be
diagnosed before the tube has ruptured, it can be treated medically by oral administration
of methotrexate and leucovorin. Methotrexate, a folic acid antagonist chemotherapeutic
agent that attacks and destroys fast-growing cells. Because trophoblast and zygote growth
is rapid, the drug is drawn to the site of ectopic pregnancy. Women are treated until a
negative hCG titer is achieved. A hestrosalpingogram or sonogram is usually performed
after the chemotherapy to assess whether the tube is fully patent. Mifepristone, an
abortifacient, is also effective at causing sloughing of the tubal implantation site. The
advantage of these therapies is that the tube is left intact, with no surgical scarring that
could cause second ectopic implantation.
The therapy for ruptured ectopic pregnancy is laparoscopy to ligate the bleeding vessels
and to remove or repair the damaged uterine tube. A rough suture line on the uterine tube
may lead to another tubal pregnancy, so either the tube will be removed or suturing on
the tube is done with microsurgical technique.
If a tube is removed, a woman is theoretically only 50% fertile, because every other
month, when she ovulates next to the removed tube, sperm cannot reach the ovum on
that side. However, this is not reliable contraceptive measure. Research in rabbits has
shown that translocation of ova can occur –that is, an ovum released from the right ovary
can pass through the pelvic cavity to the opposite (left) uterine tube and become fertilized
and vice versa.(salphigictomy-removal of the fallopian tube.)
As with miscarriage, women with Rh-negative blood should receive Rh (D) immune
globulin (RhIG) after an ectopic pregnancy fro isoimmunization protection in future
childbearing.
II. OBJECTIVES
Generally, later than three weeks of orientation and exposure at the Perpetual
Succour Hospital –Station 3B, the proponents should contribute to the practice of
managing ectopic pregnancy cases in any clinical setting by utilizing the acceptable
notions, skills, and outlooks that they will be achieving from this study.
2) pool all data for printing and binding and finish the study before March 5,
2010, Friday, the scheduled date of presentation.
3) submit the final hard and soft copies of the output to the clinical instructor.
6) defend the case study in front the panelists by answering the relevant
questions thrown by them.
10)gather again as a group for pointers and reactions from each member and
from the clinical instructor after the case presentation.
III. NURSING ASSESSMENT
Client in Context
Client F.B.M., 25 years old, female, 5 weeks age of gestation; admitted to Perpetual
Succour Hospital for the first time on March 02, 2010 at 8:58 A.M., accompanied by
her husband; in for complaints of right, lower abdominal pain; pre-operative diagnosis –
ectopic pregnancy; operative diagnosis –ruptured ectopic pregnancy, right uterine tube;
undergone major operation on March 02, 2010 at 10:00 A.M. –exploratory laparostomy,
right salpingo-oophorectomy; under the services of Dr. Lyn Alana Busa of the Department
of Obstetrics; with hospital number 219923.
Biographical Data
Client not aware of pregnancy, LMP on January 22, 2010; experienced vaginal spotting
with minimal bleeding on February 24, 2010, regarded as usual menstruation, drank beer;
experienced abdominal pain on afternoon of February 27, 2010, 3 days PTA, started at
RLQ, squeezing in quality, tolerable, radiated downwards to right thigh, no other
associated symptoms such as fever, nausea and vomiting; no medications taken, no
consultation; pain persisted and increased in quality on March 02, 2010, thus prompted
admission; ER blotter: T 36.4°C, HR 92 bpm, RR 24 cpm, BP 90/60 mmHg.
None
Time of Time
Medication
Dose/Frequency Last Medication Name Dose/Frequency of Last
Name
Dose Dose
None
PHYSICAL ASSESSMENT
General appearance
Client F.M, 25 years old married and resident of holyname mabolo cebu city. She
was seen grimacing once in awhile. She can now move minimally with assistant and was
able to turn sides occasionally. She was still pale and weak.
Vital signs
Temperature: 36.8°C
Pulse: 79 bpm
Respiration: 20 cpm
Blood Pressure: 90/60 mmHg
Height: 5 feet
Weight: 47kg
Integumentary
Skin is fair colored, warm, soft, and smooth, with moles at the right lower face, left upper
face behind the left nares and freckles around the left upper forehead; trauma in the right
dorsal part of the hand; hairs is thick, long, wavy, without parasites nor flakes on the
scalp; no clubbing present, negative capillary test (3 sec)
HEENT
Head/face normocephalic; no tenderness or masses; facial features symmetrical. Vision
was not assessed, extraocular muscles intact, visual fields normal by confrontation, cornea
and iris are intact, sclera is white, conjunctivae clear and pale pink, PERRLA, positive
constriction and convergence. External ear canals clear without redness, swelling, lesions,
and tympanic membrane intact, gray. Nares patent, no sinus tenderness present; nasal
mucosa pink, cilia noted; septum intact, no deviation. Lips dry; oral mucosa and gingivae
pink and moist without lesions; 32 ivory colored teeth, dental cary noted at the upper left
canine; tonsils are not assessed; tongue is smooth pink, symmetrical, no lesions.
Neck and Axillae
Positive swallow reflex
Thorax
Breasts symmetrical; light brown areolas and nipples with no masses or discharges;
normal spinal curvatures
Abdomen
Sutures seen in the abdomen, Wasn’t able to auscultate abdomen due to abdominal
binder present and the client felt the pain when binder was loose
Musculoskeletal System and Extremities
Full ROM of lower extremities (patient was sitting with her legs dangling), upper
extremities are not fully movable because of the IVF at the right arm and the left arm is
still in trauma; skin is warm, hairs are visible in both legs; wasn’t able to assess gait, heel-
to-toe walk and the likes because client is still lethargic and still needs assistance in
moving.
Neuro-sensory
NO DATA
Genitalia-Rectum
Menarche at 13 years old, regular for 3 days, consumes 1 napkin in a day; positive
dysmenorrheal;
GORDON’S FUNCTIONAL HEALTH PATTERNS
Nutritional –Metabolic
Patient eats 3 meals a day. For breakfast, she eats fish, rice and drinks milk. For
lunch she eats 1cup of rice, fish and drinks orange juice and for dinner she usually
eats vegetables, a cup of rice and milk. Patient eats snack between meals. When
she was admitted she said that her eating pattern is not the same before, she can
only eat 2-2 ½ cup of rice for the 3meals compared to 3-4 cups of rice for the 3
meals before she was admitted
Elimination
Prior to admission and during admission, patient’s elimination pattern is still the
same. She urinates 4-5x a day with approximately 240- 250 ml per void. She
defecates 4-5x a week. She said that she is constipated. Patient said that she
noticed if she eats apple in the morning she can defecate an hour or two after.
Activity –Exercise
She wakes up early every morning. Before going to work she strolls outside their
apartment as her exercise. She spends 30mins- 1hr walking. At work, she usually rest
during her break. She takes a nap every break time. Now that she is admitted her
activity is limited because she needs rest due to her surgery.
Sleep –Rest
Patient usually wakes up at 6-7 in the morning and sleep at around 10:30 in the
evening. She can only take a nap sometimes. So far she doesn’t talk while sleeping
but ‘’hagok’’ if she’s very stress from work. She also mentioned that previously she
treat her insomnia by means of taking ‘’4G’’ but as of now she takes ferrous sulfate
to treat her insomnia. During her admission, patient sleeping pattern was different
because patient doesn’t have enough sleep due to some noise in the ward.
Cognitive –Perceptual
The client can understand well. She responds calmly to the interviewers. She has no
difficulties in all her senses. When she was admitted, she said she was exhausted.
Role –Relationship
Patient aware that her responsibilities in the family is to be a good, loving, caring,
understanding wife to her husband and to their future children. As a wife, she said
that she takes care of her husband’s needs like cooking him for breakfast, preparing
his food for work. She is very close to her husband, she even ask advices from her
husband. She is not very close to her siblings because it’s been long time since
they’ve seen each other. In work, she believes that she’s almost responsible to all.
She defines roles and responsibilities in life as a law and is to be followed
accordingly. The client felt sadness after knowing that her baby has already gone.
Her husband is always at her side to comfort her
.
Value –Belief
Self-perception –Self-concept
Patient describes herself as emotional, hard working and of course loving wife to her
husband. She’s emotional, because according to her, she’s very sensitive
(emotionally); hardworking, because she really focuses on her work; lastly, she’s
loving wife, because she still have time for her husband although she’s workaholic.
Coping –Stress
Patient stated that, ‘’A problem is part of our lives. It molds us to become stronger.’’
For her, problem is like a challenge that if without it; a person won’t fully enjoy and
feel life’s accomplishments and satisfaction. She also mentioned during assessment
that problems gives stress and makes a person very depress unless that certain
person knows how to handle it. Her ways in coping up with problems/stressors are
to always pray and ask God’s guidance; Work on it in order to solve it whether by
herself or with the help of others.
Sexuality –Reproductive
Patient stated that she had her first menstruation at the age of 13. Her
menstruation is regular, usually lasts for 3 days, and she consumes at least 1
sanitary pad per day. She rated her sexual satisfaction as 9/10. .. … …. ….. …… ……
IV. LABORATORY FINDINGS
MACROSCOPIC
Color (Urine)
Appearance
Glucose
Protein
pH
Specific gravity
Bilirubin Umol/ L
Urobilinogen Mg/ dl
Urine ketone
Nitrite
Leukocytes
Blood
/hpf
MICROSCOPIC /hpf
RBC/ hpf
WBC/ hpf
Epithelial cells
Mucus threads
Amorphous material
Bacteria
LEGEND
NEG= Negative BLOOD PROTEIN
POS= Positive + = 0.03mg/dl
+ = 30mg/dl
TNTC= Too numerous to count ++ = 0.2mg/dl
++ = 100mg/dl
OCC= Occational +++ = 1.0mg/dl
+++ = 300mg/dl
++++ =
2000mg/dl
BILIRUBIN GLUCOSE
UROBILINOGEN
+ = 1mg/ dl + =50mg/dl
+ = 2mg/dl
++ = 2mg/ dl ++ = 150mg/dl
++ = 4mg/dl
+++ = 4mg/ dl +++ = 500mg/dl
+++ = 8mg/dl
++++ = 1000mg/dl
++++ = 12mg/dl
The system consists of external and internal genitalia, which develop and function
according to hormonal influences that affect fertility and childbearing. It also consists of
urinary structures.
External genitilia include the mons pubis, clitoris, vestibule, labia majora, labia minora,
vaginal introitus, hymen, Bartholin’s gland, Skene’s gland, and the urethral meatus.
Internal genitalia include the vagina, cervix, uterus, adjacent structures (adnexa), ovaries,
and uterine tubes. Internal urinary structures include the ureters, bladder, and urethra.
V. PATHOGENESIS
HOST AGENT
ENVIRONMENT
-Female, 25 y/o Unknown -rides on
motor-
-unaware of pregnancy cycle with
hus-
band
Fertilization
Recovery
Uterine deciduas sloughs off scant vaginal spotting Pain (RLQ)
Bleeding
Additional bleeding
Hemoperitoneum
(1500 cc)
- shoulder
pain
Hypovolemia
- tachycardia, thready pulse
- tachypnea
- hyptotension
Coma
Death
NURSING
NEED/NUR SCIEN
OBJECTIVES/
SING TIFIC OBJECTIV EVALUA EVALUA
NURSING RATIONALE
DIAGNOSI ANALY ES TION TION
INTERVENTIO
S/CUES SIS
NS
doctor.
There are
good
treatments
that can
help you
enjoy life
again. The
sooner you
get
treatment,
the sooner
you will
feel better.
CLASSIFIC
ATION
INDICATION NURSING
AND CONTRAINDIC SIDE
DRUG S AND RESPONSIB
MECHANI ATIONS EFFECTS
DOSAGE ILITIES
SM OF
ACTIONS
mefenamic Mefenamic 500mg/tab GI ulceration of Gi Instruct
acid acid is a inflammation. disturbances patient to
(Dolfenal) Q6 RTC/ prn Kidney or liver and
nonsteroid for pain avoid
al anti- impairment. hemorrhage, alcohol
inflammato Relief of mild blood (includes
ry drug to dyscrasias. wine, beer,
(NSAID) moderately Drowsiness, and liquor)
which is an severe dizziness, when taking
anthranilic somatic and headache, this
acid neuritic pain; visual medicine
derivative. headache, disturbances. since it can
It exhibits migraine,trau Skin cause
anti- matic pain, reactions and increases in
inflammato post-partum nephropathy. stomach
Tramadol ry, pain, postop Resp irritation.
(TDL) analgesic pain, dental depression,
and pain and in especially in
antipyretic pain and presence of Avoid
activity by fever cyanosis and aspirin,
inhibiting following excessive
aspirin-
prostaglan various bronchial
containing
din inflammatory secretion, and
conditions; after op on products,
synthesis Nausea,
dysmenorrhe biliary tract. other pain
in body vomiting,
parecoxib al, Acute medicines,
tissues. fatigue,
(Dynastat) menorrhagia alcoholism, other blood
Unlike accompanied head injuries, headache,
most other constipation, thinners
by spasm of conditions in
nonsteroid drowsiness, (warfarin,
hypogastric which
al anti- intracranial confusion, ticlopidine,
pain
inflammato pressure is skin clopidogrel),
ry drugs, raised. Attack reactions, garlic,
mefenamic of dry mouth, ginseng,
acid 50mg Q6 prn bronchospasm. facial
ginkgo, and
appears to for Heart failure flushing,
compete sweating, vitamin E
painModerat secondary to
with vertigo, while taking.
e to severe chronic lung
prostaglan disease. bradychardia Talk with
acute and
dins for chronic pain, , palpitation, healthcare
binding at painful orthostatic provider
the diagnostic Hypersensitivit hypotension,
prostaglan procedures y to parecoxib hypothermia,
din and surgery or to any other restleness,
receptor ingredient of changes in
site and Dynastat. modod,
thus, Patients who miosis.
potentially have Rarely,
affect demonstrated muscle
prostaglan allergic-type weakness,ap
dins that reactions to petite
Short term
have changes,
treatment of sulfonamides,
already difficulty in
acute pain & acetylsalicylic
been passing
cefazolin post-op pain. acid (aspirin) or ®assess type,
formed. urine, biliary
(Stancef) May be used nonsteroidal location and
anti- spasm. intensity of pain
pre-op to
Binds to prevent or inflammatory before 2-3 hr
mu-opoid drugs (NSAIDS) Body as a after
reduce post- administration.
receptors. op pain; can including other Whole: Back
Inhibits reduce cyclooxygenas pain. ®assess BP
reuptake of e-2 (COX-2) Central and and RR. Respi
opioid depression has
serotonin requirements specific Peripheral
not occurred
and when used inhibitors; Nervous with
norepineph concomitantl asthma and System: recommended
rine in the y. urticaria Dizziness. doses.
ranitidine(E
ntac) CNS. GI System: ®advise patient
Therapeuti Alveolar to change
osteitis (dry position slowly
c effect: socket), to minimize
decreased constipation orthostatic
pain and hypotension.
flatulence. ®do not
Platelet, confuse
Bleeding and tramadol from
Clotting: toradol.
Ecchymosis.
Parecoxib Psychiatric:
is a Agitation and
prodrug of insomnia.
valdecoxib. History of Skin and
The shock by Appendages:
mechanism cefazolin. Increased
of action of sweating and
valdecoxib pruritus.
is by Events
inhibition Occurring
of ≥0.5% and
cyclooxyge <1%:
nase-2 Application
(COX-2)- Hypersensitivit Site: Injection
mediated 500mg y; some site pain.
prostaglan IVTT products that Autonomic
din Q8H contain alcohol Nervous
synthesis. and should be System: Dry
Cyclooxyge Infections of avoided in mouth.
nase is the resp, GIT patient with Body as a
responsible & GUT, otic & known Whole:
for bone; skin, intolerance; Asthenia and
generation soft tissue & some products peripheral
of post-op that contain edema.
prostaglan infections; aspartame and Hearing and
dins. Two bacteremia, patient with Vestibular:
isoforms, septicemia, phenylketonuri Earache.
COX-1 and endocarditis a. Heart Rate
COX-2, & other and Rhythm:
have been infections Bradycardia.
identified. due to Metabolic
COX-2 is susceptible and
the isoform organisms; Nutritional:
of the surgical Hyperglycem
enzyme prophylaxis ia.
that has Musculoskele
been tal System:
shown to Arthralgia.
be induced Treatment of Respiratory
by pro- peptic ulcer System:
inflammato disease, Pharyngitis.
ry stimuli GERD, Skin and
and has selected Appendages:
been cases of Rash and
postulated persistent skin Give the
to be dyspepsia, postoperativ medication
primarily pathological e around the
responsible hypersecreto complication clock at
for the ry states eg s. evenly
synthesis Zollinger- Urinary spaced
of Ellison System: times and to
prostanoid syndrome, Oliguria. finish the
mediators stress medication
of pain, ulceration & completely
inflammati in patient at at directed,
on and risk of acid Shock; even if
fever. At aspiration hypersensitiv feeling
therapeutic during ity reactions; better.
doses, general hematologic Check for
valdecoxib anesthesia. eg signs of
is a COX-2 granulocytop super
selective enia, infection
inhibitor of eosinophilia
(vaginal
both or
peripheral thrombocyto itching/
and central penia; discharges)
prostaglan hepatic, and allergy.
dins and renal
does not impairment;
inhibit GIT disease
COX-1, eg colitis;
thereby CNS signs
sparing including
COX-1- convulsions;
dependent alteration in
physiologic bacterial
al flora; vit Assess
processes deficiencies patient for
in tissues, & others eg epigastric or
particularly headache, abdominal
the dizziness or pain and
stomach, malaise frank or
intestine occult blood
and in the stool,
platelets. emesis, of
COX-2 is Confusion, gastric
also dizziness, aspirate.
thought to drowsiness, Administer
be involved hallucination, with meals
in headache. or
ovulation, Arrythmiasis, immediately
implantatio constipation
afterward
n and and nausea.
closure of and at
the ductus bedtime to
arteriosus prolong
and CNS effect.
functions
(fever
induction,
pain
perception
and
cognitive
function).
Bind to
bacterial
cell wall
membrane
causing
cell death.
Therapeuti
c effect:
bactericidal
action
against
susceptible
bacteria.
Inhibits the
action of
histamine
at the h2-
receptor
site located
primarily in
gastric
parietal
cells,
resulting in
inhibition
of gastric
acid
secretion.
X. BIBLIOGRAPHY
Dillon, Patricia M. 2007. Nursing Health Assessment, ed. 2. Bangkok, Thailand:
iGroup Press Co., Ltd.
Pillitteri, Adele. 2007. Maternal and Child Health Nursing: Care for the Childbearing
and Childbearing Family, ed. 5. Philippines: Lippincott Williams and Wilkins.
XI. APPENDIX