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FAMILY ASCARIDIDAE
Characteristics:
1. Three lips are present. One dorsal and two subventral. Each lips bears two papillae.
2. In between the lips there maybe small intermediate lips which are called interlabia.
3. Teeth or cutting plates are absent
4. Pharynx and oesophageal bulb are absent.
5. Usually two spicules are present. Bursa is absent.
6. Eggs are typically round or subglobular and outer wall is pitted.
Genus Ascaris
Ascaris suum
Host- pig
Site/location- small intestine
Developmental stages:
Eggs are expelled out. Larvae developed within the egg shell. Second larvae (L2) within the
egg shell are the infective stage. The host gets the infection by ingestion of the infective eggs.
Hatching occurs in the intestine and the larvae reach the liver either by active penetration by active
penetration via peritoneal cavity or hepatoportal circulation. The larvae are transported to heart
and then to lungs where these are found as 3rd larvae. Then the larvae follow the tracheal
migration. The larvae are coughed up and develop to the fourth stage in the intestine and to adult
stage subsequently.
Pathogenesis:
1. Migration of larvae causes hemorrhagic lesions in liver and creates a “milk spot”
appearance.
2. The migrating larvae also creates lesions in the bronchiole and a;veoli which results in
pneumonia
Genus Toxocara
Species:
T. canis- dangerous to the puppies
T. cati
T. vitolorum- causes calf-hood mortality in buffaloes in India
Routes of Infection
a. oral infection or ingestion of infective stage of eggs
i. tracheal route of migration
-when the pups are below 3 months of age, the tracheal route of migration occurs.
After ingestion of the eggs, hatching occurs due to the presence of different biochemical factors
present in the host. The larvae come out and start migration. The larvae penetrate into the wall of
the intestine and enter into the general circulation. By the hepatoportal circulation the larvae are
transported to the liver and it penetrates the liver capsule. Then the larvae are transported to the
lungs through the pulmonary circulation. The larvae are found first in the lower lung and then
migrate to the upper part of the respiratory tract. They reach the bronchi, trachea etc. In the lung
one moulting occurs and the second stage larvae are transformed into the third stage larvae. Then
the larvae reached the GI tract when the cough containing the larvae is ingested and the larvae
developed furthewr to become adult.
Family Oxyuridae:
-whip like, non-bursate nematodes.
Genus Oxyuris
Oxyuris equi ( pin worm or seat worm of horses)
-causes rat tailed appearance due to the irritation caused by the crawling female
worms in the anus
Life cycle: The female parasites crawl out of the anus and lay a number of eggs. Wirthin the
eggs the larvae develop. The eggs fall on the ground and the host gets the infection by ingestion of
the eggs along with the feed materials. After ingestion the eggs hatch and the larvae come out of
the egg. The larvae develop to the adult stage in the intestine.
( egg>larva1 in the egg shell>larva2 in the egg shell (infective stage)>larva3>larva4>adult)
Family Heterakidae
Genus Ascaridia
Ascaridia galli
-non-bursate nematode of birds. The incidence or the manifestation of this parasitic
infection does not apparently cause significant damage to the host
Life cycle: The eggs are expelled out of the host. In the environment the eggs develop to reach to
the infective stage (L2). The birds get the infection when the eggs are ingested along with the food
material. After ingestion the eggs hatch and the larvae come out and develop to adulthood. No
migration occurs.
Genus Heterakis
Heterakis gallinarum
Life cycle: The eggs are expelled out the host .The eggs develop in the environment. Within the
eggs, the second stage larvae are formed. The second stage larva within the shell is the infect5ive
stage. The birds gert the infection when the eggs are ingested. After ingestion the eggs hatch and
the larvae come out. The larvae develop very simply in the GI tract. No migration of the larvae
occurs
Genus Subulura
Subulura brumpti- pinmorm of fowl
Family Strongyloididae
Strongyloides spp discovered by Bavay in the year 1876
Species:
S.papillosus- Si of sheep, goat, cattle
S. cati- cat
S. westeri- pigs and horse
S. ransomi- pigs
S. stercoralis human being
Salient morphology:
1. parasites show free living and parasitic life cycle
2. oesophagus is rhabditiform in free living generation and filariform in parasitic generation.
3. the adult parasite has noticeably long esophagus
4. the female worm is parthenogenetic
Life cycle:
a. Homogonic
The eggs are expelled out of the host and hatch and larvae come out. The larvae
reach the infective stage. The final host get the infection by skin penetration or orally. The larvae
get entry into the blood circulation and transported to the lungs. They penetrate the lung alveoli
and gradually ascend up the respiratory tract. Then they dome down to the esophagus and finally
reach the intestine.
b. Heterogonic
The eggs are expelled out of the host and hatch in the environment and larvae cone
out. The larvae directly develop to adult male and female. This free living male and female
parasite copulate and the gravid female lay the eggs. The eggs hatch in the environment
and the larvae come out. The larvae directly develop to become infective stage parasite.
The final host gets the infection by skin penetration performed by the infective larvae or by
ingestion. Further development occurs as that mentioned in homogenic life cycle.
Pathogenesis:
The larvae cause formation of lesion when they penetrate the skin. Foot rot condition is
commonly found in the sheep and goat. The cutaneous lesion is called larva migrans. The lesions
are circular or straight in appearance as linear urticarial lesion called as larva currens
Control:
Biological control is applied by nematode-trapping fungi, Arthrobotrys oligospora
Family Trichuridae:
Salient morphology:
1. appearance is like a whip
2. parasite is slender anteriorly and thick posteriorly
3. the anterior part contains the GI tract and the posterior part contains the reproductive
system
4. The posterior end of the male parasite is slightly spiral and one spicule is protruded out of
the hind end. The spicule remains within a spicule sheath.
*guys kindly check your manual regarding this, sorry coz I consulted a picture regarding the parts of trichuris and it showed that the posterior
part bears the whip.. *
Life cycle:
The host gets the infection by ingestion of infective stage of eggs. After hatching the larva
comes out and then the parasite reaches the adult stage after several moultings.
Capillaridae:
-known as hair worms
-egg barrel shaped and with sides nearly parallel and with polar plugs.
Family Trichinellidae:
Morphology
1. spicules are absent
2. the parasite is posteriorly thicker than the anterior part.
3. the male parasite has two lateral flaps on either side at the posterior end.
Life cycle: The seat of predilection site of the parasite is small intestine. After copulation the male
parasite dies and the female parasite burrows the wall of the intestine. During this activity the
larvae get entrance in the blood circulation via lymphatic vessel and the larvae enter the different
musculatures like subcostal muscles, tongue and diaphragm. The affected muscle is called nurse
cell. The larvae get maturity in the striated muscle fiber and the larvae remain there forming a coil.
The whole coil remains encapsulated. The final host gets infection by ingestion of the muscles
containing the larva. ( egg>L1 the infective stage>L2>L3>L4 >adult)
PICTURES