Professional Documents
Culture Documents
Several pathogenic processes are involved in the development of diabetes. These range from
autoimmune destruction of the -cells of the pancreas with consequent insulin deficiency to
abnormalities that result in resistance to insulin action. The basis of the abnormalities in carbohydrate,
fat, and protein metabolism in diabetes is deficient action of insulin on target tissues. Deficient insulin
action results from inadequate insulin secretion and/or diminished tissue responses to insulin at one or
more points in the complex pathways of hormone action. Impairment of insulin secretion and defects in
insulin action frequently coexist in the same patient, and it is often unclear which abnormality, if either
alone, is the primary cause of the hyperglycemia.
Symptoms of marked hyperglycemia include polyuria, polydipsia, weight loss, sometimes with
polyphagia, and blurred vision. Impairment of growth and susceptibility to certain infections may also
accompany chronic hyperglycemia. Acute, life-threatening consequences of uncontrolled diabetes are
hyperglycemia with ketoacidosis or the nonketotic hyperosmolar syndrome.
"Diabetes is a major cause of blindness, kidney failure, heart attacks, stroke and lower limb
amputation" -WHO.
There are two(2) Types of diabetes, Type 1 diabetes (previously known as insulin-dependent,
juvenile or childhood-onset) is characterized by deficient insulin production and requires daily
administration of insulin. The cause of type 1 diabetes is not known and it is not preventable with
current knowledge. Symptoms include excessive excretion of urine(polyuria), thirst(polydipsia),
constant hunger, weight loss, vision changes, and fatigue. These symptoms may occur suddenly.
Type 2 diabetes (formerly called non-insulin-dependent, or adult-onset) results from the bodys
ineffective use of insulin. Type 2 diabetes comprises the majority of people with diabetes around the
world, and is largely the result of excess body weight and physical inactivity.
Symptoms may be similar to those of type 1 diabetes, but are often less marked. As a result, the
disease may be diagnosed several years after onset, once complications have already arisen.
Until recently, this type of diabetes was seen only in adults but it is now also occurring increasingly
frequently in children.(World Health Organization)
Globally, The number of people with diabetes has risen from 108 million in 1980 to 422
million in 2014. In 2015, an estimated 1.6 million deaths were directly caused by diabetes. Another
2.2 million deaths were attributable to high blood glucose in 2012. Almost half of all deaths
attributable to high blood glucose occur before the age of 70 years. WHO projects that diabetes will
be the seventh leading cause of death in 2030 (World Health Organization)
In the Philippines, At least six million Filipinos all over the country have been diagnosed to
have diabetes (Philippine Daily Inquirer. Aug 13 2016) Dr. Augusto Litonjua, president of the
Philippine Center for Diabetes Education Foundation, warned that this figure could double to 12
million or even more by 2040 because of undiagnosed diabetes cases.
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Introduction
"We are losing the war againts diabetes because diabetes keeps increasing in prevalence around
the world." -Dr. Litonjua
Diabetes Mellitus indeed is a very deadly and scary disease which could be 1 of the leading
causes of death in the near future if we don't act today, but even diabetes is very deadly we can prevent
through our lifestyle, Lifestyle plays a very important role in maintaining our health, even the simplest of
exercise can reduce the risk of developing diabetes, Diet which is also very vital in preventing the
occurence of diabetes mellitus, As said by Dr. Litonjua we should avoid the "Ks" which is
Katakawan(Gluttony), Katamaran(laziness), Katabaan(Obesity) to increase our defense against this
deadly disease.
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Personal Information
PERSONAL DATA
Nationality: Filipino
Birthday: 1/ 25/1960
Age: 57
Final Diagnosis: Diabetic Foot (R), Sever anemia, Acute Renal Failure 2 infection of CKD 2 DM
nephropathy, S/P BKA
Mr. E.L is a 57 year old male admitted on Aug. 13, 2017 with a past medical history of DM who present
himself today complaining of non-healing wound. The wound has been gradually worsening over the
past months. on Aug. 14 Mr. E.L undergone the procedure insertion of Intra Jugular Catheter. August 15
Mr. E.L undergone his first Hemodialysis . August 18 Mr. E.L undergone another Hemodialysis. August
19 Mr. E.L undergone BKA or Below Knee Amputation to control the necrosis of the skin. August 20
Client undergone another Hemodialysis. August 22 Mr. E.L was ordered of MGH.
Mr. E.L was hospitalized on 2011 due to DM foot(L) but was able to save the foot and didn't
undergone any amputation. Mr. E.L recalled that his parents doesn't have any history of the disease and
verbalized that it was his lifestyle that caused the said illness as he was very fond of eating sweet foods
prepared by his wife. Mr. E.L is the Eldest among 9 siblings and could not remember any immunizations
he was able to take. He verbalized that he experience colds, lbm , measles, chicken pox before when he
was young. His wife is monitoring his blood glucose in their house as they have their own glucometer.
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Physical Assesment
Integument
Skin: The clients skin is yellowish in color, both Right and Left lower leg skin is dark, dry and shiny.
Hair: The hair of the client is thin, hair is no evenly distributed and most part of the scalp is bald with
Nails: The client has a light brown nails and has the shape of convex curve and slightly longer than the
normal height due to failure of cutting which is advised by the AP to avoid injury. When nails pressed
between the fingers (Blanch Test), the nails return to usual color in more than 3 seconds.
Head
Face: The face of the client appeared smooth but saggy and has uniform consistency and with no
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Physical Assesment
EENT
Eyelids: There were no presence of discharges, no discoloration and lids close symmetrically with
Eyes: The pupils of the eyes are black and equal in size. The iris is flat and round. PERRLA (pupils equally
Ears: The Auricles are symmetrical and has the same color with his facial skin. The client is able to hear
clearly
Nose: The nose appeared symmetric, straight and uniform in color. There was no presence of discharge
Mouth: The lips of the client are uniformly pink; moist, symmetric and have a smooth texture.
Teeth and Gums: enamels light yellow in color, no retraction of gums, pinkish in color of gums. The
buccal mucosa of the client appeared as uniformly pink; moist, soft, glistening and with elastic texture.
The tongue of the client is centrally positioned. It is yellowish in color, moist and slightly rough. There is
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Physical Assesment
Neck: The neck muscles are equal in size. The client showed coordinated, smooth head movement with
no discomfort. The lymph nodes of the client are not palpable. The trachea is placed in the midline of
the neck. The thyroid gland is not visible on inspection and the glands ascend during swallowing but are
not visible.
Lungs / Chest: The chest wall is intact with no tenderness and masses. Theres a full and symmetric
expansion and the thumbs separate 2-3 cm during deep inspiration when assessing for the respiratory
excursion. The client manifested crackles during respiration. Heart: There were no visible pulsations on
Abdomen: The abdomen of the client has a saggy skin and is uniform in color. The abdomen has a
symmetric contour. There were symmetric movements caused associated with clients respiration.
The extremities are symmetrical in size and length. After Aug. 19 2017, S/P BKA on (R) Leg the
Muscles: The muscles are not palpable with the absence of tremors. (R) Lower leg is exhibiting necrosis
with dark, dry and shiny in appearance. After Aug. 19 2017, S/P BKA on (R) Leg exhibited tremors when
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Anatomy and Physiology
When a bone is broken, the periosteum and blood vessels in the cortex, marrow, and surrounding soft
tissues are disrupted. Bleeding occurs from the damaged ends of the bone and from the neighboring
soft tissue. A clot (hematoma) forms within the medullary canal, between the fractured ends of the
bone, and beneath the periosteum. Bone tissue immediately adjacent to the fracture dies. This necrotic
tissue along with any debris in thefracture area stimulates an intense inflammatory response
leukocytes andmast cells. [for information on the pathophysiology of the immune response,
vascular tissue invades the fracture area from surrounding soft tissue and themarrow cavity, and blood
flow to the entire bone is increased. Bone-forming cells in the periosteum, endosteum, and marrow are
activated to produce subperiosteal procallusalong the outer surface of the shaft and over the broken
ends of the bone. Osteoblastswithin the procallus synthesize collagen and matrix, which becomes
mineralized to formcallus (woven bone). As the repair process continues, remodeling occurs, during
whichunnecessary callus is resorbed and trabeculae are formed along lines of stress. Except for the liver,
bone is unique among all body tissues in that it will form new bone, not scar tissue, when it heals after a
fracture
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Bibliography
https://s-media-cache-ak0.pinimg.com/736x/32/b6/fc/32b6fc55087bf90d696220df05fe574b--teaching-
https://www.fadavis.com/product/nursing-pharmacology-davis-drug-guide-deglin-vallerand-sanoski-
https://www.rnpedia.com/nursing-notes/medical-surgical-nursing-notes/complete-blood-count-cbc/
Books
Judith Hopfer Deglin PharmD, April Hazard Vallerand PhD, RN, FAAN
Cynthia A. Sanoski BS, PharmD, FCCP, BCPS. Davis Drug Guide 12th Edition (August 9, 2017)
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