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  • H's and T's of ACLS

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    5H5T pada Cardiac arrest

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    H’s and T’s of ACLS

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    5H5T pada Cardiac arrest

    Copyright:

    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
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    5K views1 page

    H's and T's of ACLS

    Uploaded by

    gabbyneng
    5H5T pada Cardiac arrest

    Copyright:

    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
    Flag for inappropriate content
    of 1

    Hs and Ts of ACLS

    H
    Hypovolemia Hypoxia Hydrogen Ion Hypo/Hyper- Hypothermia
    (acidosis) kalemia
    Loss of fluid volume in the Deprivation of an adequate Obtain an arterial blood gas Both a high and low K+ can cause If a patient has been
    circulatory system. oxygen supply can be a to determine respiratory cardiac arrest. exposed to the cold,
    significant contributing acidosis. warming measures should
    Look for obvious blood loss. cause of cardiac arrest. Signs of high K+ include taller, be taken.
    Provide adequate peaked T-waves, and widening of
    Most important intervention is Ensure that the airway is ventilations. the QRS complex. Core temp. should be
    to obtain IV access and open. raised above 86 F and 30 C
    administer IV fluids. Use sodium bicarbonate to Signs of low K+ include flattened as soon as possible.
    Ensure adequate ventilation, prevent metabolic acidosis if T-waves, prominent U-waves and
    Use a fluid challenge to and bilateral breath sounds. necessary. possibly widened QRS complex. The patient may not
    determine if the arrest is respond to drug or
    related to hypovolemia Ensure oxygen supply is Never give undiluted intravenous electrical therapy while
    connected properly. potassium. hypothermic.

    T
    Toxins Tamponade Tension Thrombosis Thrombosis
    Pneumothorax (heart: acute, massive MI) (lungs: massive PE)

    Accidental overdose : Some of Fluid build-up in the Tension pneumothorax Causes acute myocardial Can rapidly lead to
    the most common include: pericardium results in shifts in the intrathroacic infarction. respiratory collapse and
    tricyclics, digoxin, ineffective pumping of the structure and can rapidly sudden death.
    betablockers, and calcium blood which can lead to lead to cardiovascular ECG signs: 12 lead ECG with ST-
    channel blockers). pulseless arrest. collapse and death. segment changes, T-wave ECG signs of PE: Narrow
    inversions, and/or Q waves. QRS Complex and rapid
    Cocaine is the most common ECG symptoms: Narrow QRS ECG signs: Narrow QRS heart rate.
    street drug that increases complex and rapid heart complexes and slow heart Physical signs: elevated cardiac
    incidence of pulseless arrest. rate. rate. markers on lab tests, and chest Physical signs: No pulse
    pain/pressure. felt with CPR. distended
    Physical signs include Physical signs: jugular vein Physical signs: JVD, tracheal neck veins, positive d-
    bradycardia, pupil symptoms, distention (JVD), no pulse or deviation, unequal breath Treatments: use of fibrinolytic dimer test, prior positive
    and other neurological difficulty palpating a pulse, sounds, difficulty with therapy, PCI (percutaneous test for DVT or PE.
    changes. and muffled heart sounds. ventilation, and no pulse felt coronary intervention).
    with CPR. Treatment: surgical
    Poison control can be utilized Perform: pericardiocentesis The most common PCI procedure intervention (pulmonary
    to obtain information about to reverse. Treatment: Needle is coronary angioplasty with or thrombectomy) and
    toxins and reversing agents. decompression. without stent placement. fibrinolytic therapy.

    2016 Jeffery Media Productions

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