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Multiple pulp polyps associated with deciduous teeth

Article · January 2015

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KHURSHID MATTOO et al.
DATE OF PUBLICATION: MARCH 19, 2015 VOL 1 ISSUE 2 JAN-MAR 2015

MULTIPLE PULP POLYPS ASSOCIATED WITH DECIDUOUS TEETH

ADITYA KAPOOR1,KHURSHID MATTOO2, LAKSHYA YADAV3

1Department of Prosthodontics, Subharti dental college, Subharti University, Meerut, (India)

2Department of Prosthodontics, College of dentistry, Jazan University, (KSA)

3 Department of Prosthodontics, King George dental college, KGMC University, Lucknow, (India)

Corresponding author: Khurshid Mattoo, College of dental sciences, Jazan University (KSA)

Mobile – +966595086078; Email address – drkamattoo@rediffmail.com

Work attributed to: Subharti dental college and hospital, Subharti University, India.

ABSTRACT

Chronic hyperplastic pulpits occur in molar teeth of children and young adults and is characterized by a clinically visible over‐
growth of granulomatous tissue into the surrounding tooth cavity. Here, we report rare occurrence of three pulp polyps in the
same individual at the same time two in the lower deciduous molars and one in the maxillary left first deciduous molar.

KEYWORDS: pulp, hyperplasia, deciduous, granulation tissue, inflammation, repair

1. INTRODUCTION

Pulp polyp also called as chronic hyperplastic pulpitis is a


vascular condition of the pulp that usually takes place in an
open carious lesion that is devoid of intra pulpal pressure
thereby asserting itself as a mild non painful pulpal condi‐
tion.1,2 Mechanical irritation compounded with bacterial
invasion results in chronic inflammation producing a
hyperplastic response from the pulp filling the dentinal de‐
fect. Being a type of irreversible pulpitis it is usually

asymptomatic in nature. Only when the tissue overgrows
the size of the tooth occlusal pressure may cause bleeding Figure 1:(A) Mandibular left molar showing deep red tissue
or tenderness. The diagnosis is based on clinical picture proliferation within the open cavity of the tooth (B) Intra
which is usually a reddish mass filling the pulp chamber oral periapical view of the same tooth (C) Maxillary deci‐
origin of which is not from surrounding periodontal tissues duous first molar overshadowed by pulp polyp (D) IOPA of
and is responsive to pulp vitality tests.3,4 Often also re‐ the same tooth showing underlying permanent premolars
ferred as a pyogenic granuloma of the pulp tissue it is
seldom seen in permanent teeth of older adults because 2. CLINICAL REPORT
adults lack rampant caries occurrence and do not have
A young boy aged 15 years, was referred to the department
wide apices of roots to bring an ample blood supply.
of Prosthodontics for making of primary impressions due to
patient’s extreme sensitiveness to gagging reflex. Medical
history was irrelevant, but dental history disclosed multiple
broken and carious teeth with numerous overgrowths of
tissues within the tooth. Extra oral examination was non‐
contributory whereas intra oral examination showed mul‐
International Journal of Research in Medical Sciences and Technology- www.ijset.in 8
KHURSHID MATTOO et al.
DATE OF PUBLICATION: MARCH 19, 2015 VOL 1 ISSUE 2 JAN-MAR 2015

tiple proximal and occlusal caries with open cavities. Max‐ of infiltrate from acute to predominant chronic inflamma‐
illary left sided deciduous first molar and mandibular tory cells.
second molars on either side showed pink tissue where
teeth would be (Fig 1 A and C). On careful palpation the
4. CONCLUSION
tissue originated from within the pulp in all three teeth and Chronic hyperplastic pulpitis is a unique pulpal disorder
obscured remaining tooth in two teeth. The patient had that needs to be differentiated from other disorders of the
observed the growth about three months back and then pulp and gingiva. Occurrences of three cases in the same
there was a gradual enlargement of the tissue. The lesions patient are rarely reported and this case suggests that mul‐
varied from half centimeter to one centimeter in length tiple pulp polyps do occur.
with about 2 to 3 mm in height between all lesions. All
three lesions had a pedunculated stalk arising from the REFERENCES
floor of the pulp chamber with one of the lesion having mild
1. Smulson MH, Sieraski SM. Histophysiology and diseases
surface ulcerations. All three teeth showed reduced res‐
of in dental pulp. In: Weine FS, ed. Endodontic Therapy,
ponses to the vitality tests. Periapical radiographs showed
4th edn. St. Louis, USA: CV Mosby, 1989; pp. 142‐5.
underlying permanent teeth with deep carious lesion of the
involved teeth (Fig 1 C and D). The diagnosis was made on 2. Peterson LJ. Principle of management of impacted
clinical grounds and the treatment plan decided was ex‐ teeth. In: Peterson LJ, Indresano AT, Marciani RD, Roser
traction of the deciduous teeth with placement of a space SM, editors. Principles of Oral and maxillofacial Sur‐
maintainer till underlying permanent teeth would erupt. gery. Vol. 104. Philadelphia: J.B. Lippincott Company;
1992.
3. DISCUSSION
3. Calskan MK, Oztop F, Calskan G. Histological evaluation
The response that pulp displays in cases of chronic hyper‐
of teeth with hyperplastic pulpitis caused by trauma or
plastic pulpits is a classic example of balance between tis‐
caries: case reports. Int Endod J. 2003; 36:64–70.
sue response, stimulus and body defense mechanism.5, 6
Any one among them dominating will eventually cause a 4. Ness GM, Peterson LG. Impacted teeth. In: Miloro M,
different response from the pulp. In this case the lesion de‐ Ghali GE, Larsen PE, Waite PD, editors.Peterson's prin‐
veloped when carious pulp exposure created a large, but ciple of Oral and Maxillofacial surgery. London: BC
open cavity establishing the pathway for drainage of the Decker Inc.; 2004. p. 139.
inflammatory exudate. Due to this draining mechanism, the
acute inflammation subsides and with adequate host re‐ 5. Piskin B, Aktener BO, Karakisi H. Neural changes in
sponse the chronic inflammatory tissue proliferates.7‐9 ulcerative and hyperplastic pulpitis: a transmission
Pulpal proliferation indicates fairly good natural defense electron microscopic study. Int Endod J, Jul 1993;
mechanism with rich blood supply,10,11 although it can be 26(4):234‐40. .
argued that same would not occur if the pulp chamber
6. Walton RE, Pashley DH, Dowden WE. Pulp pathosis. In:
would be closed one and the cavity would have not been
Ingle JI, Taintor FC, eds. Endodontics, 3rd edn. Phila‐
large. This case is unique in that the patient had multiple
delphia, USA:Lea & Febiger,1995, pp. 398‐ 402.
teeth involved (three in number) and it would be inter‐
esting to know that despite three lesions in different areas 7. Neville BW, Damm D, Allen CM, Bouquot JE. Oral and
of the mouth the patient did not have any major problems Maxillofacial Pathology. Philadelphia: W.B. Saunders
although the patient disclosed that he had difficulty in Company; 1995. pp. 97–8.
chewing hard food sometimes as it would cause discomfort.
Despite pulp being a delicate tissue, which usually creates a 8. Stabholz A, Shekter M, Schwartz Z. Condensing osteitis
painful experience if inflamed, the presence of three chron‐ and chronic hyperplastic pulpitis in the same pulpally
ic lesions reiterates that unless pain is not experienced, the involved tooth. Quint Int 1982; 2, 137‐8.
patient does not seek treatment thus stressing pain as a
9. Trowbridge HO. Histology of pulpal inflammation. In:
useful indicator of any disease. Multiple lesions in the oral
Hargreaves KM, Goodis HE, 3 rd ed. Seltzer’s and
cavity since last three or four months without any history of
Bender’s Dental Pulp, Quintessence, 2002; pp.227‐245
pain could also be a clue to the lesion following a prepro‐
grammed pattern that histologically is seen by the change 10. Stanley HR. Pulp capping: conserving the dental
International Journal of Research in Medical Sciences and Technology- www.ijset.in 9
KHURSHID MATTOO et al.
DATE OF PUBLICATION: MARCH 19, 2015 VOL 1 ISSUE 2 JAN-MAR 2015

pulp‐can it be done; is it worth it? Oral Surg Oral Med


and Oral Pathol 1989; 68, 628‐39.

11. Ingle JI, Simmon JHS, Walton RE, Pashley DH, Bakland
LK, Heithersay GS, et al. Pulpal pathology: its ethiology
and prevention. In: Ingle JI, Bakland LK, edi‐
tors. Endodontics. London: BC Decker Inc; 2002. pp.
157–9.

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