DAFTAR PUSTAKA

Definisi aterosklerosis:

8. Brown CT. Penyakit Aterosklerotik Koroner. Dalam: Price SA, Wilson LM.
Patofisiologi Konsep Klinis Proses-Proses Penyakit. Edisi Ke - 6. Pendit Bu,
Hartanto H, Wulansari P, Mahanani DA, Alih Bahasa. Hartanto H, Susi N,
Wulansari P, Mahanani DA, Editor Edisi Bahasa Indonesia. Jakarta: EGC; 2005. Hal
576-612.

22. Marks DB, Marks AD, Smith CM. Metabolisme Kolesterol Dan Lipoprotein
Darah. Dalam: Biokimia Kedokteran Dasar. Pendit BU, Alih Bahasa. Suyono J,
Sadikin V, Mandera LI, Editor Edisi Bahasa Indonesia. Jakarta: EGC; 2000. Hal.
513-532.

Etiologi:
1. Mayo Clinic Staff. 2005. Arteriosclerosis/Artherosclerosis. Available from: URL:
http://www.mayoclinic.com/health/arteriosclerosis-atherosclerosis/DS00525 (accessed: 2006, 24
April)

Patogenesis:
18. Ross R. Atherosclerosis-an inflammatory disease. The New England
Journal of Medicine; 1999; 340:115 – 26
19. Ross R. The pathogenesis of atherosclerosis-an update. The New England
Journal of Medicine; 1986; 314: 488 – 500
20. Kusmana D, Hanafi M: Patofisiologi penyakit jantung koroner. Dalam:
Ismudiati L editor. Buku Ajar Kardiologi. Jakarta: FK UI; 2003:159 – 165
24. Tugasworo D, Patogenesis aterosklerosis editor oleh Purwaningsih E.
Badan Penerbit UNDIP, Semarang; 2010: 15 – 24

Tanda dan gejala :

11. Olford, James L. 2005. Atherosclerosis. Available from: URL: HYPERLINK:

http://www.emedicine.com/med/topic182.htm (accessed: 2006, April 20)

Faktor resiko
5. Dzau VJ. Tissue angiotensin and pathobiology of vascular disease; a
unifying hypothesis. Hipertension. 2001: 37: 1047- 52
6. Heart-Health Screenings. American Heart Association [Internet]. 2011.
[cited 2011 Jan 8]. Available from:
http://www.heart.org/HEARTORG/Conditions/Heart-Health-
Screenings_UCM_428687_Article.jsp#.TxegnNUqt0c

Pemeriksaan n treatment:
1. Mayo Clinic Staff. 2005. Arteriosclerosis/Artherosclerosis. Available from: URL:
http://www.mayoclinic.com/health/arteriosclerosis-atherosclerosis/DS00525 (accessed: 2006, 24
April)

5. University of Maryland Medicine. 2003. Atherosclerosis. Available from: URL;

HYPERLINK: http://www.umm.edu/heartinfo/Atherosclerosis.htm (accessed 2006, April 20)

hiperbarik
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Critical Care Nurse 2002; 22; 52-8.
8. Perhimpunan Kesehatan Hiperbarik Indonesia. Pengantar Ilmu Kesehatan
Penyelaman. Jakarta: PKHI; 2000 p. 11-22
9. Lipsky BA, Berendt AR. Hiperbaric Oxygen Therapy for Diabetic Foot Wounds.
Diabetic Journal 2010; 33; 1143-45.
10. Sugiharto. Pengaruh Teakanan Udara Tinggi terhadap Kesehatan tubuh. Kemas
2007; 2; 112-20.
11. Widiyanti P. Basic Mecanism of hyperbaric Oxygen in Infectious Disease.
Indonesian Journal of Tropical and Infectious Disease 2011; 2; 49-54.

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Hyperbaric Oxygen Therapy Committee Report (12th ed.). Durham, NC:
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3. Bennett, M. H.; Kertesz, T.; Perleth, M.; Yeung, P.; Lehm, J. P. (17 October
2012). Hyperbaric oxygen for idiopathic sudden sensorineural hearing loss
and tinnitus (Review). Cochrane Database of Systematic Reviews. 10:
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4. Jain, KK. "Indications, Contraindications, and Complications of HBO Therapy".
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5. Stubbs JM, Johnson EG, Thom SR (2005). Trends Of Treating Patients, That
Have Received Bleomycin Therapy In The Past, With Hyperbaric Oxygen
Treatment (Hbot) And A Survey Of Considered Absolute Contraindications To
Hbot. Undersea Hyperb Med abstract. 32 (supplement).

6. Takenaka S, Arimura T, Higashi M, Nagayama T, Ito E (Aug 1980).

Experimental study of bleomycin therapy in combination with hyperbaric
oxygenation. Nihon Gan Chiryo Gakkai shi. 15 (5): 864–75.

7. Rijadi S, Mahdi H, Sasongko, Siswanto, Hinarya D, Suharsono, Soepriyoto,

Setiawan W, Hanjaya M, Guntoro, Susanto A. 2016. Buku ajar ilmu kesehatan
penyelaman dan hiperbarik. Lakesla. pp. 337-345.

8. Marx, JA, ed. (2002). "chapter 194". Rosen’s Emergency Medicine: Concepts
and Clinical Practice (5th ed.). Mosby
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levels of spontaneously hypertensive rats. Clin Exp Hypertens. 32(5):318
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oxygen treatment of hemodynamically unstable patient. Diving Hyperb. Med.
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KESIMPULAN

2. aterosklerosis secara histopatologik dibagi menjadi beberapa tahap: 1) lesi

awal ( fatty streak, dengan mikrotrombi), 2) lesi lanjut (fibrosis, plak ateroma-
aterosklerotik) dan 3) lesi komplikata (ulserasi, kalsifikasi, perdarahan,
ganggren, aneurisma serta infark).
3. Tujuan dari pengobatan adalah untuk memperlambat atau bahkan mengembalikan
keadaan normal. Pengobatan dapat meliputi membuat perubahan gaya hidup jangka
panjang, minum obat, dan menjalani operasi.
4. Pada manusia normal oksigen hiperbarik menyebabkan penurunan curah
jantung sebesar 10-20 % yang disebabkan oleh karena teerjadinya bradikardi
dan penurunan isi sekuncup, tekanan darah pada umumnya tidak mengalami
perubahan selama pemberian oksigen hiperbarik. Bahkan oksigen hiperbarik
mampu meningkatkan oksigen pada jaringan sehingga mengurangi hipoksia
pada kasus iskemia, selain meningkatkan oksigen di tingkat jaringan juga
meningkatkan oksigen di tingkat seluler serta meningkatkan kelarutannya
dalam plasma darah sehigga mampu mencapai target organ atau target sel
yang sebelumnya kurang efektif untuk dicapai dengan terapi obat.
5. Namun terapi oksigen ini juga memiliki efek negatif yaitu jika plak sudah
rapuh dan mudah ruptur hal ini memicu suatu reaksi inflamasi dan dapat
memperparah kerusakan vaskular dan jaringan.