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  • Pathophysiology of Chronic Renal Failure (Condensed) Part 2

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    deborah malnegro
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    The document summarizes the progression and effects of end-stage renal disease. As kidney function declines below 10%, nitrogenous wastes accumulate in the blood, impairing platelets and immune function. This leads to anemia, bleeding tendencies, edema, hypertension, heart failure, and damage to multiple organ systems. Without treatment through dialysis or transplantation, the final stage is multiple organ failure and death.

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    Pathophysiology of Chronic Renal Failure (condensed) part 2

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    The document summarizes the progression and effects of end-stage renal disease. As kidney function declines below 10%, nitrogenous wastes accumulate in the blood, impairing platelets and immune function. This leads to anemia, bleeding tendencies, edema, hypertension, heart failure, and damage to multiple organ systems. Without treatment through dialysis or transplantation, the final stage is multiple organ failure and death.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    4K views1 page

    Pathophysiology of Chronic Renal Failure (Condensed) Part 2

    Uploaded by

    deborah malnegro
    The document summarizes the progression and effects of end-stage renal disease. As kidney function declines below 10%, nitrogenous wastes accumulate in the blood, impairing platelets and immune function. This leads to anemia, bleeding tendencies, edema, hypertension, heart failure, and damage to multiple organ systems. Without treatment through dialysis or transplantation, the final stage is multiple organ failure and death.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    of 1

    Nitrogenous Decreased Continuous Toxins irritate Toxins impair Urea

    Malfunction of wastes impairs Erythropoietin decline in renal pericardial sac immune deposits
    RAAS platelets Production function system on the skin

    > 90 % of Pericarditis
    Bleeding Decreased Uremic
    Na & H2O tendencies kidney Immune frost
    retention damage system
    ANEMIA Cardiac
    Tamponade

    Decreased Increased Edema Risk for


    -fatigue - Reduction in renal
    Urine blood superinfection
    - weakness capillaries
    Output pressure -Scarring of Glomeruli
    Toxins
    Pulmonary - Atrophy & Fibrosis of ┼ affect
    Oliguria Edema, Renal tubules
    Sepsis CNS
    Peripheral
    Hypertension
    Edema

    Stage IV Uremic
    END-STAGE RENAL ┼ Encephalopathy
    Heart Failure
    DISEASE
    (ESRD)
    GFR <10% -changes in
    mentation/
    psychiatric
    Continuous Multisystem symptoms
    Affectation -irritability
    -fatigue
    -insomnia
    Multiple Organ Failure
    ┼

    ┼ DEATH

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