Professional Documents
Culture Documents
CARDIOVASCULAR SYSTEM
CARDIOVASCULAR SYSTEM
HEART ANATOMY and PHYSIOLOGY: A. Heart wall 1. pericardium a. fibrous pericardium b. serous pericardium 2. epicardium 3. myocardium 4. endocardium
CARDIOVASCULAR SYSTEM
B. Chambers 1. Atria
2. Ventricles a. right b. left a. right b. left
CARDIOVASCULAR SYSTEM
c. Function: - permit unidirectional flow of blood from specific atrium to specific ventricle during ventricular diastole - prevent reflux during ventricular systole - valve leaflets open during ventricular diastole and close during ventricular systole; valve closure produces the first heart sounds (S1)
CARDIOVASCULAR SYSTEM
2. Semilunar valves a. Pulmonary valve b. Aortic valve c. Function: - permit unidirectional flow of blood from specific ventricle to arterial vessel during ventricular systole - prevent reflux during ventricular diastole - valves open when ventricles contract and close during ventricular diastole; valve closure produces the second heart sound (S2)
CARDIOVASCULAR SYSTEM
D. Conduction System 1. Sinoatrial (SA) node 2. Internodal Tracts 3. Atrioventricular (AV) node 4. Bundle of His - right bundle branch - left bundle branch 5. Purkinje fibers * Electrical activity of heart can be visualized by ECG
CARDIOVASCULAR SYSTEM
E. Coronary Circulation 1. Arteries a. right coronary artery b. left coronary artery 2. Veins a. coronary sinus veins b. thebesian veins
CARDIOVASCULAR SYSTEM
VASCULAR SYSTEM Function: a. supply tissues with blood b. remove wastes c. carry unoxygenated blood back to the heart
CARDIOVASCULAR SYSTEM
TYPES OF BLOOD VESSELS A. Arteries B. Arterioles C. Capillaries: the following exchanges occur: - oxygen and carbon dioxide - solutes between the blood and tissues - fluid volume transfer between the plasma and interstitial spaces D. Venules E. Veins
CARDIOVASCULAR SYSTEM
ASSESSMENT HEALTH HISTORY A. Presenting problem 1. Nonspecific symptoms may include - fatigue - shortness of breath - cough - palpitations - headache - weight loss/gain - syncope - difficulty sleeping - dizziness - anorexia
CARDIOVASCULAR SYSTEM
2. Specific signs and symptoms a. chest pain b. dyspnea (shortness of breath) c. orthopnea / paroxysmal nocturnal dyspnea d. palpitations: precipitating factors e. edema f. cyanosis B. Lifestyle: occupation, hobbies, financial status, stressors, exercise, smoking, living conditions
CARDIOVASCULAR SYSTEM
C. Use of medications: OTC drugs, contraceptives, cardiac drugs D. Personality profile: Type A, manic-depressive, anxieties E. Nutrition: dietary habits, cholesterol, salt intake, alcohol consumption F. Past Medical History G. Family history: heart disease (congenital, acute, chronic); risk factors (DM, hypertension, obesity)
CARDIOVASCULAR SYSTEM
PHYSICAL EXAMINATION A. Skin and mucous membranes: - color/texture, temperature, hair distribution on extremities, atrophy or edema, petechiae B. Peripheral pulses: - palpate and rate all arterial pulses (temporal, carotid, brachial, radial, femoral, popliteal, dorsalis pedis and posterior tibial) on scale of: 0=absent, 1=palpable, 2=normal, 3=full, 4=full and bounding
CARDIOVASCULAR SYSTEM
C. Assess for arterial insufficiency and venous impairment D. Measure and record blood pressure E. Inspect and palpate the neck vessels: a. jugular veins: note location, characteristics, jugular venous pressure b. carotid arteries: location and characteristics F. Auscultate heartsounds - normal (S1, S2) - abnormal (S3, S4)
CARDIOVASCULAR SYSTEM
LABORATORY / DIAGNOSTIC TESTS A. Blood Chemistry and electrolyte analysis 1. Cardiac enzymes: in MI a. Troponin T: detected 3-12 hours after chest pain b. Troponin I: detected 3-12 hrs c. creatine phosphokinase (CPK MB): 6-12Hrs d. Aspartate aminotransferase (AST) (SGOT): 24 Hrs after chest pain e. Lactic dehydrogenase (LDH): 36 Hrs
CARDIOVASCULAR SYSTEM
2. Electrolytes a. Sodium (Na) 135-148meq/L - hyponatremia: fluid excess - hypernatremia: fluid deficit b. Potassium (K) 3.5-5 meq/L - inc. or dec. levels can cause dysrhythmias c. Magnesium (Mg) 1.3-2.1 meq/L - dec. levels can cause dysrhythmias
CARDIOVASCULAR SYSTEM
d. Calcium (Ca) 4.5-5.3 meq/L: - nec. For blood clotting and neuromuscular activity - dec. levels cause tetany, inc. levels causes muscle atony - dec. and inc. levels cause dysrhythmias 3. Serum Lipids a. Total Cholesterol 150-200mg/dl: - high levels predispose to atherosclerotic HD
CARDIOVASCULAR SYSTEM
b. High density lipids (HDL) 30-85 mg/dl - low levels predispose to CVD c. Low density lipids (LDL) 50-140 mg/dl: - high levels predispose to atherosclerotic plaque formation d. Triglycerides 10-150 mg/dl: - high levels increase risk of atherosclerotic heart disease
CARDIOVASCULAR SYSTEM
B. Hematologic Studies 1. CBC 2. Coagulation time: 5-15mins; inc. levels indicate bleeding tendency, used to monitor heparin tx. 3. Prothrombin time (PT) 9.5-12sec.; INR 1.0, used to monitor warfarin tx. 4. Activated partial thromboplastin time (APTT) 20-45sec; used to monitor heparin therapy 5. Erythrocyte sedimentation rate(ESR) <20mm/hr; inc. level indicate inflamm. process
CARDIOVASCULAR SYSTEM
C. Urine Studies (routine U/A) D. Electrocardiogram (ECG) 1. Noninvasive ECG a graphic record of the electrical activity of the heart 2. Portable recorder (Holter monitor) provides continuous recording of ECG for up to 24 hrs. E. Exercise ECG (stress test): the ECG is recorded during prescribed exercise; may show heart disease when resting ECG does not F. Echocardiogram: noninvasive recording of the cardiac structures using ultrasound
CARDIOVASCULAR SYSTEM
G. Cardiac catheterization: invasive, but often definitive test for diagnosis of cardiac disease. 1. A catheter is inserted into the right or left side of the heart to obtain information 2. Purpose: to measure intracardiac pressures and oxygen levels in various parts of the heart; with injection of a dye, it allows visualization of the heart chambers, blood vessels and blood flow (angiography)
CARDIOVASCULAR SYSTEM
3. Nursing care: prior to the test - informed consent - any allergies esp. to iodine - keep client on NPO for 8-12 hrs - record height, weight, V/S - inform client that a feeling of warmth and fluttering sensation as catheter is inserted
CARDIOVASCULAR SYSTEM
4. Nursing care: post test - assess circulation to the extremity used for catheter insertion - check peripheral pulses, color, sensation of affected extremity - if protocol requires, keep affected ext. straight for approx. 8 hrs. - observe catheter insertion site for swelling, bleeding - assess V/S and report for sig. changes
CARDIOVASCULAR SYSTEM
H. Coronary arteriography 1. visualization of coronary arteries by injection of radiopaque contrast dye and recording on a movie film. 2. Purpose: evaluation of heart disease and angina, location of areas of infarction and extent of lesions, ruling out coronary artery disease in clients with MI. 3. Nursing care: same as cardiac catheterization
ANALYSIS
Nursing diagnosis for the client with CVD include A. Fluid volume excess B. Decreased cardiac output C. Altered peripheral tissue perfusion D. Impairment of skin integrity E. Risk for activity intolerance F. Pain G. Ineffective coping H. Fear I. Anxiety
B. Medical management, assessment findings and nursing interventions Angina pectoris and MI
ANGINA PECTORIS
A. Gen. info: 1. transient, paroxysmal chest pain produced by insufficient blood flow to the myocardium resulting in myocardial ischemia 2. Risk factors: - CAD - DM - hypertension - aortic insufficiency - severe anemia - atherosclerosis - thromboangiitis obliterans
ANGINA PECTORIS
3. Precipitating factors: - physical exertion - sexual activity - strong emotions - cigarette smoking - consumption of a heavy meal - extremely cold weather
B. Medical mgt: 1. Drug therapy: nitrates, beta adrenergic blocking agents, and/or calcium blocking agents, lipid reducing drugs if cholesterol is elevated
ANGINA PECTORIS
2. Lifestyle modification 3. Surgery: coronary bypass surgery
C. Assessment Findings: 1. Pain: substernal with possible radiation to the neck, jaw, back and arms, relieved by REST 2. Palpitations, tachycardia, dyspnea, diaphoresis 3. el. serum lipid levels
ANGINA PECTORIS
4. Diagnostic tests: - ECG may reveal ST segment depression and Twave inversion during chest pain - Stress test may reveal an abnormal ECG during exercise
D. Nursing interventions: 1. administer oxygen 2. give prompt pain relief with nitrates or narcotic analgesics as ordered.
ANGINA PECTORIS
3. Monitor V/S, status of cardiopulmonary function, monitor ECG 4. place patient in semi-high Fowlers position 5. provide emotional support, health teachings and discharge instructions. 6. Instruct client to notify physician immediately if pain occurs and persists, despite rest and medication administration.
MYOCARDiAL INFARCTiON
A. General information: 1. The death of myocardial cells from inadequate oxygenation, often caused by a sudden complete blockage of a coronary artery; characterized by localized formation of necrosis (tissue destruction) with subsequent healing by scar formation and fibrosis. 2. Risk factors: - atherosclerotic CAD - DM - thrombus formation - hypertension
MYOCARDiAL INFARCTiON
B. Assessment findings: 1. Pain same as in angina, crushing, viselike with sudden onset; UNRELIEVED by rest or nitrates 2. nausea/vomiting, dyspnea 3. skin: cool, clammy, ashen 4. elevated temperature 5. initial increase in BP and pulse, with gradual drop in BP 6. Restlessness
MYOCARDiAL INFARCTiON
7. Occasional findings: rales or crackles; presence of S4; pericardial friction rub; split S1, S2 8. Diagnostic tests: a. elevated WBC, cardiac enzymes (troponin, CPK-MB, LDH, SGOT) b. ECG changes (specific changes dependent on location of myocardial damage and phase of the MI; inverted T wave and ST segment changes seen with myocardial ischemia c. inc. ESR, el. serum cholesterol
MYOCARDiAL INFARCTiON
C. Nursing interventions: 1. establish a patent IV line 2. provide pain relief; morphine sulfate IV (poor peripheral perfusion, false + for enzymes) 3. Administer O2 as ordered to relieve dyspnea and prevent arrhythmias 4. Provide bed rest with semi fowlers position 5. Monitor ECG and hemodynamic procedures 6. Administer anti-arrhythmias as ordered.
MYOCARDiAL INFARCTiON
7. Monitor I & O, report if UO <30 ml/hr 8. Maintain full liquid diet with gradual increase to soft, low salt 9. Maintain quiet environment 10. Administer stool softeners as ordered 11. Relieve anxiety associated with CCU environment 12. Administer anticoagulants, thrombolytics (tpa or streptokinase) as ordered and monitor for S/E
MYOCARDiAL INFARCTiON
13. Provide client teaching and discharge instruction concerning - effects of MI, healing process and treatment regimen - Medication regimen: name, purpose, schedule, dosage, S/E - Risk factors with necessary lifestyle modification - Dietary restrictions: low salt, low cholesterol, avoidance of caffeine - Resumption of sexual activity as ordered (usually 46weeks)
MYOCARDiAL INFARCTiON
- Need to report the ff. symptoms: * increased persistent chest pain * pain, dyspnea, weakness, fatigue * persistence palpitations, light headedness - Enrollment of client in a cardiac rehabilitation program
DYSRHYTHMIAS
An arrhythmia is a disruption in the normal events of the cardiac cycle. It may take a variety of forms. Treatment varies on the type dysrhythmias
SINUS TACHYCARDIA A. General Information: 1. A heart rate of over 100 beats/min, originating in the SA node
DYSRHYTHMIAS
2. May be caused by: - fever - anemia - apprehension - hyperthyroidism - physical activity - myocardial ischemia - caffeine - drugs (epi., theo)
B. Assessment findings: 1. Rate: 100-160 beats /min 2. Rhythm: regular
DYSRHYTHMIAS
3. P wave: precedes each QRS complex with normal contour 4. P-R interval: normal (0.08 sec) 5. QRS complex: normal (0.06 sec)
C. Treatment; - correction of underlying cause, elimination of stimulants, sedatives, propranolol (Inderal)
DYSRHYTHMIAS
SINUS BRADYCARDIA A. General Information: 1. A slowed heart rate initiated by SA node 2. Caused by: - excessive vagal or decreased sympathetic tone - MI - IC tumors - meningitis - myxedema - cardiac fibrosis - normal variation of the heart rate in well trained athletes
DYSRHYTHMIAS
B. Assessment findings: 1. Rate: <60 beats/min 2. Rhythm: regular 3. P wave: precedes each QRS with a normal contour 4. P-R interval: normal 5. QRS complex: normal C. Treatment: usually not needed - if cardiac output is inadequate: atropine and isoproterenol; pacemaker
DYSRHYTHMIAS
ATRIAL FIBRILLATION A. General information 1. An arrhythmia in which ectopic foci cause rapid, irregular contractions of the heart 2. seen in clients with - rheumatic mitral stenosis - thyrotoxicosis - cardiomyopathy - pericarditis - hypertensive heart disease - CHD
DYSRHYTHMIAS
B. Assessment findings: 1. Rate: atrial: 350-600 beats/min ventricular: varies bet. 100-160 beats /min 2. Rhythm: atrial and ventricular regularly irregular 3. P wave: no definite P wave; rapid undulations called fibrillatory waves 4. P-R interval: not measurable 5. QRS complex: generally normal
DYSRHYTHMIAS
C. Treatment: digitalis preparations, propanolol, verapamil in conjunction with digitalis; direct current cardioversion PREMATURE VENTRICULAR CONTRACTIONS A. General Information: 1. Irritable impulses originate in the ventricles 2. Caused by: - electrolyte imbalance (hypokalemia) - digitalis drug therapy
DYSRHYTHMIAS
Contd: (causes) - stimulants( caffeine, epinephrine, isoproterenol) - hypoxia - CHF
B. Assessment findings: 1. Rate: varies according to no. of PVCs 2. Rhythm: irregular because of PVCs 3. P wave: normal; however, often lost in QRS complex
DYSRHYTHMIAS
4. P-R interval: often not measurable 5. QRS complex: greater then 0.12secs, wide
C. Treatment: 1. IV push of Lidocaine (50-100mg) followed by IV drip of lidocaine at rate of 1-4 mg/min 2. Procainamide, quinidine 3. Treatment of underlying cause
DYSRHYTHMIAS
VENTRICULAR TACHYCARDIA A. General information: 1. 3 or more consecutive PVCs; occurs from repetitive firing of an ectopic focus in the ventricles 2. caused by: - MI - CAD - digitalis intoxication - hypokalemia
DYSRHYTHMIAS
B. Assessment findings: 1. Rate: atrial: 60-100 beats/min ventricular: 110-250 beats/min 2. Rhythm: atrial(regular), ventricular (occly. irregular) 3. P wave: often lost in QRS complex 4. P-R interval usually not measurable 5. QRS complex: greater than 0.12 secs, wide
DYSRHYTHMIAS
C. Treatment: 1. IV push of lidocaine (50-100mg), then IV drip of lidocaine 1-4 mg/min 2. Procainamide via IV infusion of 2-6 mg/min 3. direct current cardioversion 4. bretylium, propanolol
CARDIAC ARREST
A. General Info: - sudden, unexpected cessation of breathing and adequate circulation of blood by the heart
B. Medical management: 1. Cardiopulmonary resuscitation (CPR) 2. Drug therapy: a. lidocaine, procainamide, verapamil b. Dopamine, isoproterenol, Norepinephrine
CARDIAC ARREST
c. Epinephrine to enhance myocardial automaticity,
excitability, conductivity, and contractility the heart, thus increasing the heart rate
d. Atropine sulfate to reduce vagus nerves control over e. Sodium bicarbonate: administered during first few
moments of a cardiac arrest to correct respiratory and metabolic acidosis
CARDIAC ARREST
C. Assessment findings: - unresponsiveness, cessation of respiration, pallor, cyanosis, absence of heart rate/ BP/pulses, dilation of pupils, ventricular fibrillation
D. Nursing interventions: 1. Begin precordial thump and if successful, administer lidocaine 2. If unsuccessful, defibrillation - CPR 3. Assist with administration of and monitor effects of emergency drugs
CARDIOPULMONARY RESUSCITATION
A. General info: process of externally supporting the circulation and respiration of a person who has had a cardiac arrest B. Nursing interventions: unwitnessed cardiac arrest 1. Assess LOC a. Shake victims shoulder and shout b. if no response, summon for help 2. Position victim supine on a firm surface
CPR
3. Open airway a. Use head tilt, chin lift maneuver b. Place ear nose and mouth - look to see if chest is moving - listen for escape of air - feel for movement of air against face c. If no respiration, proceed to #4 4. Ventilate twice, allowing for deflation between breaths
CPR
5. Assess circulation: if not present, proceed to #6 6. Initiate external cardiac compressions a. Proper placement of hands: lower half of the sternum b. Depth of compressions: 1 - 2 in. for adults c. One rescuer: 15 compressions (80-100/min) with 2 ventilations d. Two rescuers: 5 compressions (80-100/min) with 1 ventilation
ENDOCARDITIS
B. Medical management: 1. Drug therapy: a. antibiotics specific to sensitivity or organism cultured b. PenG and streptomycin if org. not known c. antipyretics 2. Cardiac surgery to replace valve
ENDOCARDITIS
C. Assessment findings: 1. Fever, malaise, fatigue, dyspnea and cough acute upper quadrant pain, joint pain 2. petechiae, murmurs, edema, splenomegaly, hemiplegia and confusion, hematuria 3. elevated WBC & ESR, decreased Hgb & Hct. 4. Diagnostic tests: positive blood culture for causative organism
ENDOCARDITIS
D. Nursing interventions: 1. antibiotics as ordered 2. control temperature 3. assess for vascular complications and pulm. embolism 4. Provide client teaching and discharge planning - types of procedures, antibiotic therapy - S/S to report: persistent fever, fatigue, chills, anorexia, joint pains - avoidance of individuals with known infections
MYOCARDITIS
A. General Info: an acute or chronic inflammation of the myocardium as a result of pericarditis, systemic infection or allergic response. B. Assessment: - fever, pericardial friction rub, gallop rhythm - murmur, signs of heart failure, fatigue, dyspnea - tachycardia, chest pain
MYOCARDITIS
C. Implementation: 1. Assist client to assume a position of comfort 2. Administer analgesics, salicylates, NSAIDS 3. Administer O2, provide adequate rest periods 4. Limit activities, to dec. workload of heart 5. Treat underlying cause 6. Administer meds. as ordered: - antibiotics, diuretics, ACE inhibitors, digitalis 7. Monitor complications: thrombus, heart failure, cardiomyopathy
PERICARDITIS
A. General Info: 1. An inflammation of the visceral and parietal pericardium 2. caused by bacterial, viral, or fungal infection; collagen diseases; trauma; acute MI, neoplasms, uremia, radiation, drugs (procainamide, hydralazine, Doxorubicin HCL)
PERICARDITIS
B. Medical management: 1. Determination and elimination/control of underlying cause 2. Drug therapy a. Medication for pain relief b. Corticosteroids, *salicylates (aspirin), indomethacin, to reduce inflammation 3. Specific antibiotic therapy against the causative organism may be indicated
PERICARDITIS
C. Assessment findings: 1. chest pain with deep inspiration (relieved by sitting up), cough, hemoptysis, malaise 2. tachycardia, fever, pericardial friction rub, cyanosis or pallor, jugular vein distension 3. Elevated WBC and ESR, normal or inc. SGOT 4. Diagnostic test: a. chest x-ray may show increased heart size b. ECG: ST elevation, T wave inversion
PERICARDITIS
D. Nursing Interventions: 1. Ensure comfort, bed rest with semi- or high Fowlers position 2. Monitor hemodynamic parameters 3. Administer medications as ordered and monitor effects 4. Provide client teaching and discharge planning: - S/S of pericarditis indicative of recurrence (chest pain intensified by lying down and relieved when sitting up; medication regimen
b. In premature infants, PDA sometimes can be closed using prostaglandin synthetase inhibitors (Indomethacin) w/c stimulate closure of the ductus
arteriosus
enlarged R side of the heart and the inc. pulmonary circulation demonstrates the separation of the R atrial septum and the inc. oxygen saturation in the R atrium
reveals RVH and possible PA dilatation from the inc. blood flow
size of the VSD and the degree of PS. 1. Acute episodes of cyanosis (tet spells) and transient cerebral ischemia. Tet spells are char. By irritability, pallor, and blackouts or convulsions.
1. Pathophysiology
a. this defect results in two separate circulatory patterns; the right heart manages systemic circulation and the left manages pulmonary circulation b. to sustain life, the child must have an associated defect.
2. Assessment findings:
a. Clinical manifestations vary, depending on associated defects
C. TRUNCUS ARTERIOSUS
- failure of normal septation and division of the embryonic bulbar trunk into the PA and aorta, resulting in a single vessel that overrides both ventricles
CYANOTIC CHD
1. Pathophysiology
a. blood ejected from the ventricles enters the common artery and flows either the lungs or aortic arch. b. pressure in both ventricles is high and blood flow to the lungs is markedly increased.
2. Assessment findings:
a. neonates with this defect appear normal; however, as pulmonary vascular resistance decreases after birth, severe pulmonary edema and CHF commonly develop
CYANOTIC CHD
2. marked cyanosis, especially on exertion; S/S of CHF; LVH, dyspnea, marked activity intolerance, and retarded growth 3. loud systolic murmur best heard at the lower left sternal border and radiating throughout the chest
2. Assessment findings:
a. Clinical manifestations 1. the child may be asymptomatic or may experience the classic difference in BP and pulse quality between the upper and lower ext. the BP is elevated in the UE and dec. in the LE while the pulse is bounding in the UE and dec. or absent in the LE. Thus femoral pulse are weak or absent
1. Pathophysiology a. RV pressure increases leading to RVH and eventually RV failure may occur
2. Assessment findings: a. Clinical manifestations 1. may be asymptomatic or may have mild cyanosis or CHF
Types: a. Essential (primary, idiopathic): marked by loss of elastic tissue and arteriosclerotic changes in the aorta and larger vessels coupled with decreased caliber of the arterioles b. Benign: a moderate rise in BP marked by a gradual onset and prolonged course
HYPERTENSION
c. Malignant: characterized by a rapid onset and short dramatic course with a DBP of >150mmHg d. Secondary: elevation of the BP as a result of another disease such as renal parenchymal disease, Cushings disease, pheochromocytoma, primary aldosteronism, coarctation of the aorta
A. Essential hypertension usually occurs between ages 3550; more common in men over 35, women over 45; African-American men affected twice as often as white men/women
HYPERTENSION
Risk Factors: - (+) family history, obesity, stress, cigarette smoking, hypercholesterolemia, inc. sodium intake B. Medical management: 1. Diet and weight reduction (restricted sodium, kcal, cholesterol) 2. Lifestyle changes: alcohol moderation, exercise regimen, cessation of smoking 3. Antihypertensive drug therapy
HYPERTENSION
C. Assessment findings: 1. Pain similar to anginal pain; pain in calves of legs after ambulation or exercise (intermittent claudication); severe occipital headaches, particularly in the morning; polyuria; nocturia; fatigue; dizziness; epistaxis; dyspnea on exertion 2. BP consistently above 140/90, retinal hges and exudates, edema of extremities 3. Rise in SBP from supine to standing position (indicative of essential hypertension) 4. Diagnostic tests: elevated serum uric acid, sodium, cholesterol levels
HYPERTENSION
D. Nursing interventions: 1. Record baseline BP in 3 positions (lying, sitting, standing) and in both arms 2. Continuously assess BP and report any variables that relate to changes in BP (positioning, restlessness) 3. Administer antihypertensive agents as ordered; monitor closely and assess for S/E 4. Monitor intake and hourly output 5. Provide client teaching and discharge planning: - risk factors, dietary instructions, compliance of antihypertensive medications, routine follow up w/ MD
ARTERIOSCLEROSIS OBLITERANS
- a chronic occlusive arterial disease that may affect the abdominal aorta or the LE. The obstruction to blood flow with resultant ischemia usually affects the femoral, popliteal, aortic and iliac arteries - occurs most often in men ages 50-60 - caused by atherosclerosis - Risk Factors: cigarette smoking, hyperlipidemia, hypertension, DM
ARTERIOSCLEROSIS OBLITERANS
B. Medical management: 1. Drug therapy a. Vasodilators: papaverine, Isoxsuprine Hcl (Vasodilan), Nylidrin Hcl (Arlidin), nicotinyl alcohol (Roniacol) cyclandelate (Cyclospasmol), tolazoline Hcl (priscoline) to improve arterial circulation; effectiveness questionable b. Analgesics to relieve ischemic pain c. Anticoagulants to prevent thrombus formation d. Lipid reducing drug: cholestyramine, colesti[pol Hcl, dextrothyroxine sodium, clofibrate, gemfibrozil (Lopid), niacin, lovastatin (Mevacor), atorvastatin
ARTERIOSCLEROSIS OBLITERANS
2. Surgery: bypass grafting, endarterectomy, balloon catheter dilation, lumbar sympathectomy (to increase blood flow), amputation may be necessary C. Assessment findings: 1. Pain both intermittent claudication and rest pain, numbness or tingling of the toes 2. Pallor after 1-2 mins. Of elevating feet, and dependent hyperemia/rubor; diminished or absent dorsalis pedis, posterior tibial and femoral pulses; shiny, taut skin with hair loss on lower legs
ARTERIOSCLEROSIS OBLITERANS
3. Diagnostic tests: a. Oscillometry may reveal decrease pulse volume b. Doppler U/S reveals decreased blood flow through affected vessels c. Angiography reveals location and extent of obstructive process
4. Elevated serum triglycerides; sodium D. Nursing Interventions: 1. Encourage slow, progressive physical activity
ARTERIOSCLEROSIS OBLITERANS
2. Administer medications as ordered 3. Assist with Buerger-Allen exercises qid a. client lies with legs elevated above heart for 2-3 mins b. client sits on edge of bed with legs and feet dependent and exercises feet and toes upward and downward, inward and outward for 3 mins c. client lies flat with legs at heart level for 5 mins 4. Assess for sensory function; protect client from injury 5. Provide client teaching and discharge planning: stop cigarette smoking, diet, drug compliance, exercise
THROMBOANGIITIS OBLITERANS
(BUERGERS DISEASE)
- Acute inflammatory disorder affecting medium/smaller arteries and veins of the LE. Occurs as focal, obstructive process; results in occlusion of a vessel with subsequent development of collateral circulation - Most often affects men ages 25-40; disease is idiopathic; high incidence among smokers
A. Medical management: same as arteriosclerosis obliterans but only cessation of smoking is effective treatment
THROMBOANGIITIS OBLITERANS
(BUERGERS DISEASE)
B. Assessment findings: 1. Intermittent claudication, sensitivity to cold (skin of extremity may at first be white, changing to blue then red) 2. Decreased or absent peripheral pulses (post. tibial and dorsalis pedis), ulceration and gangrene (advanced) 3. Diagnostic tests: same as arteriosclerosis obliterans except no elevation in serum triglycerides
C. Nursing Interventions: 1. Prepare client for surgery
THROMBOANGIITIS OBLITERANS
(BUERGERS DISEASE)
2. Provide client teaching and discharge planning - drug regimen, avoidance of trauma to the affected extremity, need to maintain warmth esp. during cold weathers, importance of stopping smoking
RAYNAUDS PHENOMENON
- intermittent episode of arterial spasms, most frequently involving the fingers; most often affects women between the teenage years and age 40; cause unknown - Predisposing factors: collagen diseases (SLE, RA), trauma (from typing, playing piano)
A. Medical management: vasodilators, catecholaminedepleting antihypertensive drugs (reserpine, guanethidine monosulfate)
RAYNAUDS PHENOMENON
B. Assessment findings: 1. coldness, numbness, tingling in one or more digits; pain (usually pptd. By exposure to cold, emotional upsets, tobacco use) 2. intermittent color changes (pallor, cyanosis, rumor); small ulcerations and gangrene tips of digits
C. Nursing interventions 1. provide client teaching concerning: - importance of stopping smoking; need to maintain warmth; need to use gloves in handling cold objects; drug regimen
ANEURYSM
- a sac formed by dilation of an artery secondary to weakness and stretching of an arterial wall. The dilation may involve one or all layers of the arterial wall.
Classification
1. Fusiform: uniform spindle shape involving the entire circumference of the artery 2. Saccular: outpouching on one side only, affecting part of the arterial circumference
ANEURYSM
3. Dissecting: separation of the arterial wall layers to form a cavity that fills with blood 4. False: the vessel wall is disrupted, blood escapes into surrounding area but is held in place by surrounding tissue
A. General info: 1. an aneurysm, usually fusiform or dissecting, in the descending, ascending, or transverse section of the thoracic aorta 2. usually occurs in men ages 50-70; caused by arteriosclerosis, infection, syphilis, hypertension
ANEURYSM
B. Medical management: 1. control of underlying hypertension 2. Surgery: resection of the aneurysm and replacement with a Teflon/Dacron graft; client will need extracorporeal circulation
C. Assessment findings: 1. Often asymptomatic; deep, diffuse chest pain; hoarseness; dysphagia; dyspnea 2. Pallor, diaphoresis, distended neck veins
ANEURYSM
3. Diagnostic tests: a. Aortography shows exact location of the aneurysm b. X-rays: chest film reveals abnormal widening of aorta; abdominal film may show calcification within walls of aneurysm
4. Nursing interventions: same as in Cardiac surgery
THROMBOPHLEBITIS
A. General info: 1. Inflammation of the vessel wall with formation of a clot (thrombus); may affect superficial or deep veins 2. Most frequent veins affected are the saphenous, femoral, and popliteal. 3. Can result in damage to the surrounding tissues, ischemia and necrosis 4. Risk Factors: obesity, CHF, prolonged immobility, MI, pregnancy, oral contraceptives, trauma, sepsis, cigarette smoking, dehydration, severe anemias, venous cannulation, complication of surgery
THROMBOPHLEBITIS
B. Medical management: 1. Anticoagulation therapy: a. Heparin: blocks conversion of prothrombin to thrombin and reduces formation of thrombus - S/E: spontaneous bleeding, injection site reactions, ecchymoses, tissue irritation and sloughing, reversible transient alopecia, cyanosis, pain in arms or legs, thrombocytopenia b. Warfarin (coumadin): blocks prothrombin synthesis by interfering with vit. K synthesis - S/E: GI: anorexia, nausea/vomiting, diarrhea, stomatitis
THROMBOPHLEBITIS
- hypersensitivity: dermatitis, urticaria, pruritus, fever - other: transient hair loss, burning sensation of feet, bleeding complications. 2. Surgery a. Vein ligation and stripping b. venous thrombectomy: removal of a clot in the iliofemoral region c. plication of the inf. vena cava: insertion of an umbrella-like prosthesis into the lumen of the vena cava to filter incoming clots
THROMBOPHLEBITIS
C. Assessment findings: 1. Pain in the affected extremity 2. Superficial vein: tenderness, redness, induration along course of the vein 3. Deep vein: swelling, venous distension of limb, tenderness over involoved vein, (+) Homans sign 4. Elevated WBC and ESR 5. Diagnostic tests: a. venography (phlebography): inc. uptake of radioactive material
THROMBOPHLEBITIS
b. Doppler ultrasonography: impairment of blood flow ahead of thrombus c. Venous pressure measurements: high in affected limb until collateral circulation is developed
D. Nursing interventions 1. Provide bed rest, elevating involved extremity 2. Apply continuous warm, moist soaks to dec. lymphatic congestion 3. Administer anticoagulants as ordered
THROMBOPHLEBITIS
a. Heparin 1. monitor PTT, use infusion pump to administer IV heparin 2. assess for bleeding tendencies (hematuria; hematemesis; bleeding gums; epistaxis, melena) 3. have antidote ( protamine sulfate) available
b. Warfarin (Coumadin) 1. assess PT daily, advise client to withhold dose and notify physician immediately if bleeding or signs of bleeding occurs 2. instruct client to use a soft toothbrush and to floss gently, prepare antidote: Vit. K
THROMBOPHLEBITIS
4. monitor for chest pain or SOB (possible pulmonary embolism) 5. Provide client teaching and discharge planning: a. need to avoid standing, sitting for long periods; constrictive clothing; crossing legs at the knees; smoking; oral contraceptives b. importance of adequate hydration c. use of elastic stockings when ambulatory d. importance of planned rest with elevation of feet e. importance of weight reduction and exercise
VARICOSE VEINS
A. General info: 1. Dilated veins that occur most often in the lower extremities and trunk. As the vessel dilates, the valves become stretched and incompetent with resultant venous pooling/edema 2. most common between ages 30-50 3. predisposing factor: congenital weakness of the veins, thrombophlebitis, pregnancy, obesity, heart disease
B. Medical management: vein ligation (involves ligating the
saphenous vein where it joins the femoral vein and stripping the saphenous vein system from groin to ankle)
VARICOSE VEINS
C. Assessment findings: 1. Pain after prolonged standing (relieved by elevation) 2. Swollen, dilated, tortuous skin veins 3. Diagnostic tests: a. Trendelenburg test: varicose veins distend very quickly (less than 35 secs) b. Doppler U/S: decreased or no blood flow heard after calf or thigh compression
D. Nursing interventions: 1. Elevate legs above heart level
VARICOSE VEINS
2. Apply knee length elastic stockings 3. Provide adequate rest 4. Prepare client for vein ligation, if necessary a. Provide routine pre-op care b. keep affected extremity elevated above the level of the heart to prevent edema c. apply elastic bandages and stockings, which should be removed every 8hrs for short periods. d. assist out of bed within 24hrs, ensuring that elastic stockings are applied. e. assess for increased bleeding