Professional Documents
Culture Documents
Outline
Definition
Types
Risk factors
Treatment
Nursing assessment and intervention
determined by:
Peripheral vascular
resistance
The ability of the
vessels to stretch.
Viscosity (Thickness)
The amount of
circulating blood volume
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Components of B/P
Pressure of blood
against the walls of
the arteries
The elasticity of the
artery walls
The volume and
thickness of the blood
Diastolic
Force between heart
pumps
Pressure as the heart
begins to fill with blood
Normal >85
What is Hypertension
Is the result of persistent
high arterial blood
pressure which may cause
damage to the vessels
and arteries of the
Heart
Brain
Kidneys
Eyes
Indication
Width (cm)
Length (cm)
Child/Small adult
10-12
18-24
Standard Adult
12-13
23-35
Large Adult
12-16
35-40
20
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Gently open the valve for a slow controlled release of air from the cuff.
The Korotkoff sounds are quite faint, but distinctive, when recognized. Listen carefully for the
first Bump, note the associated dial reading. This is the real Systolic pressure (measures the
pressure in the arteries when the heart beats).
The last sound that listen is the Diastolic pressure (measures the pressure in the arteries
between beats).
Open the air valve fully, to rapidly deflate the cuff. Release the patient from the equipment.
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CATEGORY
SYSTOLIC (MMHG)
DIASTOLIC (MMHG)
Optimal
<120
<80
Normal
120-129
80-84
High normal
130-139
85-89
Stage 1 Hypertension
140-159
90-99
Stage 2 Hypertension
160-179
100-109
Stage 3 Hypertension
180
110
CAUTION: The systolic and diastolic pressures are measured in millimetre of mercury (mmHg).
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Family history
More common in men
than women.
Women are more likely to
develop
hypertension
after menopause.
Age
Gender
Overweight or obesity
Weight
Tobacco (chemicals in
tobacco can damage the
lining of the artery walls)
Alcohol (the regular
consumption
of
3-4
alcoholic drinks per day,
increases the risk of
hypertension and reduce
the
action
of
antihypertensive
therapy.)
Stress
Unhealthy Diet
Addictions
Sedentary life
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Primary Hypertension
Chronic elevation of blood pressure from an unknown cause.
90%-95% of all cases
Secondary Hypertension
Signify high blood pressure from an identified cause (e.g.
kidney disease)
5%-10% of all cases
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Headache
Bloody nose
Kidney
Brain
Blurred vision
Dizziness
Fatigue, activity intolerance
Palpitations
Blood spots in the eyes
Heart
Target
organ
diseases
Eyes
Facial flushing
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Complications
Hypertensive Heart Disease
Coronary Artery Disease
Hypertension is a major risk factor for CAD
Left Ventricular Hypertrophy (LVH)
Increased resistance in the arteries
Stiffness and narrowing of vessels
Left heart works harder pumping against
higher pressure
Increases myocardial work and 02
consumption
Heart Failure
Heart can no longer pump enough blood to meet
the metabolic needs of the body
Contractility depressed
Stroke volume and cardiac output decreases
C/O
SOB on exertion, paroxysmal nocturnal dyspnea and
fatigue
Complications Cont
Cerebrovascular Disease (CVA)
Most common cause Atherosclerosis
Portions of plaque or a blood clot (forms on
plaque) breaks off
Thromboembolism
Travels to intracerebral vessels
Stops the flow of blood to parts of the brain
Aneurysms burst R/T increased pressure
Hemorrhage
Brain tissue damage
C/O
Intermittent claudication
Nephrosclerosis
End stage renal disease
Renal dysfunction
Ischemia
Narrowed intrarenal vessel
Atrophy of tubules
Destruction of glomeruli
Death of nephron
Earliest symptom
nocturia
Retinal Damage
Red flag
Damage to retinal vessels may indicate vessel
damage in the heart, brain, and kidney
C/O
Blurred vision
Retinal hemorrhage
Loss of vision
Medical background:
Food habits (alcohol use, salt and fat intake, weight gain/loss)
Elimination (nocturia)
Activity (fatigue, activity intolerance, dyspnoea on exertion, palpitation, angina, chest
pain, intermittent claudication, muscle cramps, )
Addictions
Cognitive/perception (blurred vision, paresthesia)
Coping/stress (stressful life events, noncompliance)
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FHP 3 Elimination
Nocturia
FHP 4 Activity/Exercise
Fatigue, Dyspnea on
exertion, palpitation,
angina, chest pain,
intermittent claudication,
muscle cramps, smoking
history, sedentary lifestyle
FHP 6
Cognitive/perception
Blurred vision
paresthesia
FHP 9 Sexual/Repro
Impotence
FHP 10 Coping/stress
Stressful life events
Noncompliance
knowledge deficit
financial
Objective Data
Cardiovascular
Persisted elevated B/P
Orthostatic change in B/P
or pulse
Retinal changes
Abnormal heart sounds
Diminished or absent
peripheral pulses
Carotid, renal, ischial or
femoral bruits
edema
Musculoskeletal
Truncal obesity
Abnormal waist-hip
ratio
Neurologic
Mental status
changes,
Localized edema
ECG
Serum electrolytes
Potassium
Hyperaldosteronism
Blood Glucose
Serum cholesterol and
triglycerides
Uric acid
Left Ventricular
hypertrophy
EEG
Ischemic heart disease
Medications
Diuretics
Suppresses renal tubular re-absorption of
sodium
Diuril
Loop diuretics
Bumex, Lasix, Demadex
Potassium supplement
Potassium sparing diuretic
Aldactone
Beta Blockers
Blocks sympathetic stimulation, decreases renin secretions,
decreases cardiac output.
Tenormin, Lopressor, Corgard, Inderal
Alpha Inhibitors
Decreases peripheral vascular resistance,
Vasodilator
Catapres
Central Inhibitors
Decreases cardiac output, peripheral resistance, and heart
rate
Aldomet, Tenex
Peripheral Inhibitors
Relaxes smooth muscle, decreases peripheral
resistance, decreases heart rate, and B/P
Resperine
Vasodilators
Relaxation of arteriolar smooth muscle,
vasodilatation, decreases cardiac output,
decreases peripheral resistance
Nitroglycerin
Calcium Channel
Blockers
Inhibits calcium into
smooth muscle cells,
vasodilatation,
decreases peripheral
resistance, increases
cardiac output
Amicor
AngiotensionConverting
Enzyme Inhibitors
Decreases
peripheral
vascular
resistance
Anapril 20 mg
Expected Outcomes
Patient will achieve and maintain desired B/P
Patient will understand, accept, and
implement the therapeutic plan for B/P
Patient will experience minimal or no side
effects from therapy
Patient will exhibit a confident ability to
manage and cope with hypertension.
MAIN GOALS:
Maintain
or
enhance
cardiovascular
functioning.
Prevent complications.
Provide information about disease process,
prognosis, and therapy.
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DIET:
reduce salt and sodium intake;
diet rich in fruits, vegetables, proteins, potassium
and calcium;
Teaching
Therapy
Adherence
STRESS
MANAGEMENT
use
non-pharmacologic
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Health
Promotion
Hypertensions pathology
Therapy
Adherence
Drug therapy
Inform about support groups and
Community support
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Explain
Health
Promotion
the
importance
about
therapy adherence.
Teaching
In case of Non-adherence:
Understand the patients reasons;
Adjust clinical treatment according
to the patients cultural beliefs and
individual attitudes
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