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THE SEVEN HABITS OF HIGHLY EFFECTIVE

PEOPLE
by Stephen R. Covey
Habit 1: Be Proactive
Ability to control ones environment, rather that have it
control you.
Taking initiative doesnt mean being aggressive, it
means recognizing your responsibility to make things
happen
Habit 2: Begin with the end in mind
The habit of personal leadership
Lead oneself towards what you consider your aims
Based on imagination the ability to envision, to see
the potential, to create with our minds what we cannot at
present see with our eyes
Habit 3: Put First Things First
The habit of personal management
Create a clear, mutual understanding of what needs to
be accomplished, focusing on what, not how;
results
not methods
Spend time. Be patient. Visualize the desired result

Habit 4: Think Win-Win


The habit of interpersonal relationship
Achievements are largely dependent on co- operative
efforts with others
Agreements or solutions are mutually beneficial &
satisfying
Habit 5: Seek first to understand and then to be
understood
The habit of communication
Diagnose before you prescribe
Simple, effective & essential in developing &
maintaining positive relationships in all aspects of life
Habit 6: Synergize
The habit of creative cooperation
the whole is greater than the sum of its parts
See good & potential in other persons contribution
Habit 7: Sharpen the saw
The habit self-renewal, of continuous improvement
Circles & embodies all other habits

1 drop = 1/20 mL
1 teaspoonful = 5 mL
1 tablespoonful = 15 mL
1 wineglassful = 60 mL = 2 ounces
1 glassful = 250 mL = 8 ounces
1 grain = 60 mg
Paracetamol Drops
1 pint = 500 mL
wt: move 1 decimal
1 quart = 1000 mL
point to
the left
1 ounce = 30 mL
AgeWt
1 Kg = 2.2 lbs
110kg
1 lb = 0.45359 Kg
212
314
416
518
620

BODY TEMPERATURE
Subnormal
Normal
Subfebrile
Fever
High fever
Hyperpyrexia
AGE
Preterm
Term
0-3 mo
3-6 mo
6-12 mo
1-3 yrs
3-6 yrs
6-12 yrs
12-17 yrs

<36.6C
37.4C
35.7 38.0C
38.0C
>39.5C
>42.0C

HR (bpm)
120-170
120-160
100-150
90-120
80-120
70-110
65-110
60-95
55-85

*BP cuff should cover 2/3 of arm


small cuff: falsely high BP
large cuff: falsely low BP

BP (mmHg)
55-75/35-45
65-85/45-55
65-85/45-55
70-90/50-65
80-100/55-65
90-105/55-70
95-110/60-75
100-120/60-75
110-135/65-85

RR (cpm)
40-70
30-60
35-55
30-45
25-40
20-30
20-25
14-22
12-18

ABG
pH:
pCO2:
pO2:

7.35-7.45
35-45
80-100

HCO3: 22-26mEq/L
B.E.:
+/- 2mEq/L
O2 sat: 97%

Normal Laboratory Values

RBC

NB
4.8-7.1

Infant
3.8-5.5

WBC
9-30,000 6-17,500
Neutrophils
61%
61%
Lymphocytes
31%
32%
Hgb (gm %)
14-24
11-20
Hct (%)

44-64

47
Platelets
140-300
(thou/mm3)
Reticulocyte 2.6-6.5
Count (%)
Bleeding time 1-5 min
Clotting time 5-8 min
Prothrombin 12-20
time (sec)

Child
3.8-5.

Adolescent
M: 4.6-6.2
F: 4.2-5.4

5-10,000

6-10,000

60%
30%
11-16

60%
30%
M: 14-18
F: 12-16
M: 40-54
F: 37-

35-49

31-46

200-423

150-450

150-450

0.5-3.1

0-2

0-2

1-6
5-8
12-14

1-6
5-8
12-14

1-6
5-8
12-14

BMI

Underweight
Normal
Overweight
at risk
Obese I
Obese II

Asian
<18.5
18.5 22.9
23.0
23 24.9
25 29.9
30

Caucasian
<18.5
18.5 24.9
25 29.9
30 39.9
>40

ANTHROPOMETRIC MEASUREMENTS

Expected Body Weight upto 1 month of age


Term

[{age in days) 10] x 20 + BW (gms)

Preterm

[(age in days) 14] x 15 + BW (gms)

GCS SCORING

APGAR

EXPANDED PROGRAM ON
IMMUNIZATION

ACUTE DIARRHEA (at least 3x BM in 24 hrs)


4 Major Mechanisms
1. Poorly absorbed osmotically active substances in
lumen
2. Intestinal ion secretion (increased) or decreased
absorption
3. Outpouring into the lumen of blood, mucus
4. Derangement of intestinal motility
Rotaviral AGE (vomiting first then diarrhea)
ingestion of rotavirus rotavirus in intestinal villi
destruction of villi
(secretory diarrhea absorption, secretion) AGE
Assessment of dehydration (skin pinch test)
(+) if > 2 seconds
no dehydration if skin tenting goes back immediately

Etiology of AGE
Bacteria
Aeromonas
Bacillus cereus
Campylobacter jejuni
Clostridium perfringens
Clostridium difficile
Escherichia coli
Plesiomonas shigelbides
Salmonella
Shigella
Staphylococcus aureus
Vibrio cholerae 01 & 0139
Vibrio parahaemolyticus
Yersinia enterocolitica

Parasites
Balantidium coli
Blastocyctis hominis
Cryptosporidium
Giardia lamblia

Viruses
Astroviruses
Caloviruses
Norovirus
Enteric adenoviruses
Rotavirus
Cytomegalovirus
Herpes simplex virus

Types of Dehydration

Classifcation of the Severity of


Dehydration

DIARRHEA TREATMENT PLAN A


4 Rules of Home Treatment
1. Give extra fluid (as much as the child will take)
> Breastfeed frequently & longer at each feeding
> if the child is exclusively breastfed, give one or more
of the following in addition to breastmilk:
- ORS solution
- food based fluid (e.g. soup, rice, water)
- clean water
How much fluid to be given in addition to the usual fluid
intake:
upto 2 years:
50-100 mL after each loose stool
2 years or more: 100-200 mL
> give frequent small sips from a cup
> if the child vomits, wait for 10 min then resume
> continue giving extra fluids until diarrhea stops
2. Give Zinc supplements
upto 6 mo: tab or 10mg per day for 10-14 days
6 months or more: 1 tab or 20mg OD x 10-14 days
3. Continue feeding
4. Know when to return

DIARRHEA TREATMENT PLAN B


Recommended amount of ORS over 4 hour period
Age upto 4 mo 4 mo
Wt

<6kg

in mL 200-400

12mo 12mo

2 yrs
2 yrs
5 yrs

6-9.9kg

10-11.9kg

12-19kg

400-700

700-900

900-1400

* Use childs age only when weight is not known


* Approximate amount of ORS (mL)

CHILDS WT (kg) x 25
> if the child wants more ORS than shown, give more
> give frequent small sips from a cup
> if the child vomits, wait for 10 min then resume
> continue breastfeeding whenever the child wants
* After 4 hours
> reassess the child & classify dehydration status
> select the appropriate plan to continue treatment
> begin feeding the child while at the clinic

DIARRHEA TREATMENT PLAN C


Treat severe dehydration QUICKLY!
> start IV fluid immediately
> if the child can drink, give ORS by mouth while the IV
drip is being set up
> give 100mL/kg Lactated Ringers solution
Age

First give
30mL/kg in:

Infants
(<12mo)

1 hour*

Children
30 min*
(12mo-5yrs)

Then give
70mL/kg in:
5 hours

2 hours

* Repeat once if radial pulse is very weak or not detectable


> reassess the child every 15-30 min. if dehydration is not
improving, give IV fluid more rapidly
> also give ORS (~5mL/kg/hr) as soon as the child can
drink [usually after 3-4 hours in infants; 1-2 hours in
children]
> reassess after 6 hrs (infant) & 3 hrs (child)

NOTE: Computed Value is in mL/day


Ex) 25kg child
Answer: 1500 + [100] = 1600cc/day

HOLIDAY-SEGAR METHOD (MAINTENANCE)

LUDANS METHOD (HYDRATION THERAPY)

IV-FLUID COMPOSITIONS (Commonly Used for Infants and Children):

Child Age 2months up to 5years

ARI PROTOCOL (PROGRAM FOR THE CONTROL OF ARI)

Young Infants <2months

ETIOLOGY OF PNEUMONIA
> Bacterial
- Streptococcus pneumoniae
- Group B streptococci (neonates)
- Group A streptococci
- Mycoplasma pnemoniae (adolescents)
- Chlamydia trachomatis (infants)
- Mixed anearobes (aspiration pneumonia)
- Gram negative enteric (nosocomial pneumonia)
> Viral
- Respiratory syncitial virus
- Parainfluenza type 1-3 (Croup)
- Influenza types A, B
- Adenovirus
- Metapneumovirus
> Fungal
- Histoplasma capsulatum (bird, bat contact)
- Cryptococcus neoformans (bird contact)
- Aspergillus sp. (immunosuppressed)
- Mucormycosis (immunosuppressed)
- Coccidioides immitis
- Blastomyces dermatitides
- Pneumocystis carinii (immunosuppressed, HIV,
steroids)

ATYPICAL PNEUMONIA: extrpulmonary manifestations,


low grade fever, patchy diffuse infiltrates, poor response
to Penicillin, negative sputum gram stain
Etiologic Agents Grouped by Age
> Neonates (<1mo)
- GBS
- E. coli
- other gram (-) bacilli
- Streptococcus pneumoniae
- Haemophilus influenzae (Type B)
> 1-3 mo
* Febrile pneumonia
- RSV
- Other respiratory viruses
- Streptococcus pneumoniae
- Haemophilus influenzae (Type B)
* Afebrile pneumonia
- Chlamydia trachomatis
- Mycoplasma homilis
- CMV

> 3-12 mo
- RSV
- Other respiratory viruses
- Streptococcus pneumoniae
- Haemophilus influenzae (Type B)
- C. trachomatis
- M. pneumoniae
- Group A Streptococcus
> 2-5 yrs
- RSV
- Other respiratory viruses
- Streptococcus pneumoniae
- Haemophilus influenzae (Type B)
- C. trachomatis
- M. pneumoniae
- Group A Streptococcus
- Staph aureus
2-5 yrs
- Streptococcus pneumoniae
- Haemophilus influenzae (Type B)
- C. trachomatis
- M. pneumoniae
- Group A Streptococcus
- Staph aureus

Dengue Pathophysiology

DENGUE
> Mode of transmission: mosquito bite (man as reservior)
> Vector: Aedes aegypti
> Factors affecting transmission:
- breeding sites, high human population density,
mobile viremic human beings
> Age incidence peaks at 4-6 yrs
> Incubation period: 4-6 days
> Serotypes:
- Type 2 most common
- Types 1& 3
- Type 4 least common but most severe
> Main pathophysiologic changes:
a. increase in vascular permeability

extravasation of plasma
- hemoconcentration
- 3rd spacing of fluids
b. abnormal hemostasis
- vasculopathy
- thrombocytopenia
- coagulopathy

Dengue Fever Syndrome (DFS)


Biphasic fever (2-7 days) with 2 or more of the ff:
1. headache
2. myalgia or arthralgia
3. retroorbital pain
4. hemorrhagic manifestations
[petechiae, purpura, (+) torniquet test]
5. leukopenia
Dengue Hemorrhagic Fever (DHF)
1. fever, persistently high grade (2-7 days)
2. hemorrhagic manifestations
- (+) torniquet test
- petechiae, ecchymoses, purpura
- bleeding from mucusa, GIT, puncture sites
- melena, hematemesis
3. Thrombocytopenia (< 100,000/mm3)
4. Hemoconcentration
- hematocrit >40% or rise of >20% from baseline
- a drop in >20% Hct (from baseline) following
volume replacement
- signs of plasma leakage
[pleural effusion, ascites, hypoproteinemia]

Dengue Shock Syndrome


Manifestations of DHF plus signs of circulatory failure
1. rapid & weak pulse
2. narrow pulse pressure (<20mmHg)
3. hypotension for age
4. cold, clammy skin & irritability / restlessness
DANGER SIGNS OF DHF
1. abdominal pain (intense & sustained)
2. persistent vomiting
3. abrupt change from fever to hypothermia with
sweating
4. restlessness or somnolence
Grading of Dengue Hemorrhagic Fever

Torniquet Test: SBP + DBP = mean BP for 5 minutes


2
if 20 petechial rash per sq. inch on antecubital fossa
(+) test
Hermans Rash:
> usually appears after fever lysed
> initially appears on the lower extremities
> not a common finding among dengue patients
> an island of white in an ocean of red

Recommended Guidelines for Transfusion:


Transfuse:
- PC < 100,000 with signs of bleeding
- PC < 20,000 even if asymptomatic
- use FFP if without overt bleeding
- FWB in cases with overt bleeding or signs of
hypovolemia
> if PT & PTT are abnormal: FFP
> if PTT only: cryprecipitate
Leukopenia in dengue: probable etiology is Pseudomonas
therefore: give Meropenem or Ceftazidime

MANAGEMENT OF DENGUE
A. Vital Signs and Laboratory Monitoring (Vital Signs and Laboratory Monitoring)
Monitor BP, Pulse Rate
We have to watch out for Shock (Hypotension)

Management of
Hemorrhage

URINARY TRACT INFECTION


Suggestive UTI:
- Pyuria: WBC 5/HPF or 10mm3
- Absence of pyuria doesnt rule out UTI
- Pyuria can be present w/o UTI
Presumptive UTI:
- (-) urine culture
- lower colony counts may be due to:
* overhydration
* recent bladder emptying
* previous antibiotic intake
Proven or Confirmed UTI:
- (+) urine culture 100,000 cfu/mL urine of a single
organism
- multiple organisms in culture may indicate a
contaminated sample

ACUTE GLOMERULONEPHRITIS (PSAGN)


Antecedent Infection (2-3 weeks)

Ag-Ab complexes + complement (ASO)

Ag-Ab deposition in GBM

Ab binding to
Glomerular Ab
C3

Proliferation deposition
Hematuria

Decreased Glomerular surface area


Decreased GFR
Activation of RAAS

Oliguria;
Normal or
increased
Creatinine

Na+ & H2O retention


Fluid overload
Circulatory congestion EDEMA

Edema,
HPN

CHF;
Pulmonary
edema

ACUTE GLOMERULONEPHRITIS
Complications of AGN
- CHF 2 to fluid overload
- HPN encephalopathy
- ARF due to GFR
STAGES of AGN
- Oliguric phase [7-10days] complications sets in
- Diuretic phase [7-10days] recovery starts
- Convalescent phase [7-10days] ptts usually sent
home
Prognosis
- Gross hematuria 2-3 weeks
- Proteinuria 3-6 weeks
- C3: 8-12 weeks
- microscopic hematuria: 6-12 mo
> Hyperkalemia may be seen due to Na+ retention
> Ca++ decreases in PSAGN
> in ASO titer
- normal within 2 weeks
- peaks after 2 weeks
- more pronounced in pharyngeal infection than in
cutaneous

RHEUMATIC FEVER

JONES CRITERIA:
A. Major Manifestations
Carditis (50-60%)
Polyarthritis (70%)
Chorea (15-20%)
Erythema Marginatum (3%)
Subcutaneous Nodules (1%)

B. Minor Manifestations
Arthralgia
Fever
Laboratory Findings of:
Elevated Acute Phase Reactants (ESR / CRP)
Prolonged PR interval

C. PLUS Supporting Evidence of Antecedent Group-A


Strep Infection
(+) Throat Culture or Rapid Strep-Ag Test
Elevated or Rising Strep-AB Test

TREATMENT OF RHEUMATIC FEVER

A. Antibiotic Therapy
10 days of Oral Penicillin or Erythromycin
IM Injection of Benzethine Penicillin

**NOTE: Sumapen = Oral Penicillin!


B. Anti-Inflammatory Therapy
1. Aspirin (if Arthritis, NOT Carditis)
Acute: 100mg/kg/day in 4 doses x 3-5days
Then, 75mg/kg/day in 4 doses x 4 weeks
2. Prednisone
2mg/kg/day in 4 doses x 2-3weeks
Then, 5mg/24hrs every 2-3 days

C. Duration of Chemoprophylaxis

B. Secondary Prevention

A. Primary Prevention
10 days of Oral Penicillin or Erythromycin
IM Injection of Benzethine Penicillin

PREVENTON

KAWASAKI DISEASE
CDC-CRITERIA FOR DIAGNOSIS: ADOPTED FROM
KAWASAKI (ALL SHOULD BE PRESENT)
A) HIGH Grade Fever (>38.5 Rectally) PRESENT for AT
LEAST 5-days without other Explanation
High Grade Fever of at least 5 days
DOES NOT Respond to any kind of Antibiotic!
B) Presence of 4 of the 5 Criteria:
1) Bilateral CONGESTION of the Ocular
Conjunctiva (seen in 94%)
2) Changes of the Lips and Oral Cavity (At least
ONE)
3) Changes of the Extremities (At least ONE)
4) Polymorphous Exanthem (92%)
5) Cervical Adenopathy = Non-Suppurative
Cervical Adenopathy (should be >1.5cm) in 42%

KAWASAKI DISEASE
HARADA Criteria
- used to determine whether IVIg should be given
- assessed within 9 days from onset of illness
1.
2.
3.
4.
5.
6.
7.

WBC > 12,000


PC <350,000
CRP > 3+
Hct <35%
Albumin <3.5 g/dL
Age 12 months
Gender: male

IVIg is given if 4 of 7 are fulfilled


If < 4 with continuing acute symptoms,
risk score must be reassessed daily

TREATMENT: Currently Recommended Protocol:


A. IV-Immunoglobulin
2g/kg Regimen Infusion EQUALLY Effective in
Prevention of Aneurysms and Superior to 4-day
Regimen with respect to Amelioration of
Inflammation as measured by days of Fever, ESR,
CRP, Platelet Count, Hgb, and Albumin.
NOTE: There is a TIME FRAME of 10 days
B. Aspirin
HIGH Dose ASA (80-100mg/kg/day divided q 6h) should be
given Initially in Conjunction with IV-IG
THEN
Reduced to Low Dose Aspirin (3-5mg/kg/day)
AND
Continued until Cardiac Evaluation COMPLETED
(approximately 1-2 months AFTER Onset of Disease)

SEIZURES
> Seizures: sudden event caused by abrupt, uncontrolled,
hypersynchronous discharges of neurons
> Epilepsy: tendency for recurrent seizures that are
unprovoked by an immediate cause
> Status epilepticus: >30min or back-to-back w/o return
to baseline
> Etiology:
- V ascular
- I nfections
- T raumatic
- A utoimmune
- M etabolic
- I diopathic
- N eoplastic
- S tructural
- S yndrome

:
:
:
:
:
:
:
:
:

AVM, stroke, hemorrhage


meningitis, encephalitis
SLE, vasculitis, ADEM
electrolyte imbalance
idiopathic epilepsy
space occupying lesion
cortical malformation, prior stroke
genetic disorder

TYPES OF SEIZURES
A. Partial Seizures (Focal / Local)
Simple Partial
Complex Partial (Partial Seizure + Impaired
Consciousness)
Partial Seizures evolving to Tonic-Clonic
Convulsion)
B. Generalized Seizures
Absence (Petit mal)
Myoclonic
Clonic
Tonic
Tonic-Clonic
Atonic
SIMPLE FEBRILE SEIZURE vs. COMPLEX FEBRILE
SEIZURE
Febrile Seizure: A seizure in association with a febrile
illness in the absence of a CNS infection or acute
electrolyte imbalance in children older than 1 month of
age without prior afebrile seizures

CLASSIFICATION BY CAUSE
A. Acute Symptomatic (shortly after an acute insult)
Infection
Hypoglycemia, low sodium, low calcium
Head trauma
Toxic ingestion
B. Remote Symptomatic
Pre-existing brain abnormality or insult
Brain injury (head trauma, low oxygen)
Meningitis
Stroke
Tumor
Developmental brain abnormality
C. Idiopathic
No history of preceding insult
Likely genetic component

SIMPLE FEBRILE SEIZURE


A. Criteria for an SFS
< 15 minutes
Generalized-tonic-clonic
Fever > 100.4 rectal to 101 F (38 to 38.4 C)
No recurrence in 24 hours
No post-ictal neuro abnormalities (e.g. Todds
paresis)
Most common 6 months to 5 years
Normal development
No CNS infection or prior afebrile seizures
B. Risk Factors
Febrile seizure in 1st/2nd degree relative
Neonatal nursery stay of >30 days
Developmental delay
Height of temperature
C. Risk Factors for Epilepsy (2 to 10% will go on to have
epilepsy)
Developmental delay
Complex FS (possibly > 1 complex feature)
5% > 30 mins => _ of all childhood status
Family History of Epilepsy
Duration of fever

BRONCHIAL ASTHMA (GINA GUIDELINES)


Controlled

Partly
controlled

Uncontrolled

Day
symptoms

none

> 2x per wk

Limitation of
activities

none

any

Nocturnal Sx
(awakening)

none

any

3 or more
symptoms of
Partly
Controlled
Asthma in any
week

Need for
reliever

< 2x per wk

> 2x per wk

Lung function

normal

< 80%

Exacerbation

none

> 1x per yr

1x / week

Tuberculosis (Treatment)
I.

Pulmonary TB
A.

Fully susceptible M. tuberculosis, no history of previous


anti-TB drugs, low local persistence of primary
resistance to Isoniazid (H)

2HRZ OD then 4HR OD or 3x/wk DOT

B.

Microbial susceptibility unknown or initial drug


resistance suspected (e.g. cavitary), previous anti-TB
use, close contact w/ resistant source case or living in
high areas w/ high pulmonary resistance to H.

I.

2HRZ + E/S OD, then 4 HR + E/S OD or 3x/wekk


DOT

Extrapulmonary TB
Same in PTB

For severe life threatening disease (e.g. miliary, meningitis,


bone, etc)
2HRZ + E/S OD, then 10HR + E/S OD or 3x/wk DOT

H.E.A.D.S.S.S.
Home Environment
With whom does the adolescent live?
Any recent changes in the living situation?
How are things among siblings?
Are parents employed?
Are there things in the family he/she wants to
change?
Employment and Education
Currently at school? Favorite subjects?
Patient performing academically?
Have been truant/expelled from school?
Problems with classmates/teachers?
Currently employed?
Future education/employment goals?
Activities
What he/she does in spare time?
Patient does for fun?
Whom does patient spend spare time?
Hobbies, interests, close friends?

H.E.A.D.S.S.S.
Drugs
Used tobacco/alcohol/steroids?
Illicit drugs? Frequency? Amount? Affected daily
activiities?
Still using? Friends using/selling?
Sexual activities
Sexual orientation?
GF/BF? Typical date?
Sexually active? When started? # of persons?
Contraceptives? Pregnancies? STDs?
Suicide/Depression
Ever sad/tearful/unmotivated/hopeless?
Thought of hurting self/others?
Suicide plans?
Safety
Use seatbelts/helmets?
Enter into high risk situations?
Member of frat/sorority/orgs?
Firearm at home?

Respiratory Distress Syndrome


(Hyaline Membrane Disease)
Male, preterm, low BW, maternal DM, & perinatal
asphyxia
Corticosteroids:

most successful method to induce fetal lung maturation


Administered 24-48 hours before delivery decrease
incidence of RDS
Most effective before 34 weeks AOG

Microscopically: diffuse atelectasis, eosinophilic


membrane
Pathophysiology:
1. Impaired/delayed surfactant synthesis & secretion
2. V/Q (ventilation/perfusion) imbalance due to
deficiency of surfactant and decreased lung
compliance
3. Hypoxemia and systemic hypoperfusion
4. Respiratory and metabolic acidosis
5. Pulmonary vasoconstriction
6. Impaired endothelial &epithelial integrity
7. Proteinous exudate
8. RDS

Respiratory Distress Syndrome


(Hyaline Membrane Disease)
Clinical Features:
1. Tachypnea, nasal flaring, subcostal and intercostal
retractions, cyanosis, grunting
2. Pallor from anemia, peripheral vasoconstriction
3. Onset within 6 hours of life
Peak severity days 2-3
Recovery 72 hours
Retractions:
Due to (-) intrapleural pressure produced by interaction
b/w contraction of diaphragm & other respiratory muscles
and mechanical properties of the lungs & chest wall
Nasal flaring:
Due to contraction of alae nasi muscles leading to marked
reduction in nasal resistance
Grunting:
Expiration through partially closed vocal cords
Initial expiration: glottis closedlungs w/ gasinc.
transpulmo P w/o airflow
Last part of expiration: gas expelled against partially closed
cords
Cyanosis:
Central tongue & mnucosa (imp. Indicator of impaired
gas exchange); depends on total amount of desaturated
Hgb

F.R.I.C.H.M.O.N.D.
Fluids
Respiration
Infection
Cardiac
Hematologic
Metabolic
Output & Input [cc/kg/h] N: 1-2
Neuro
Diet

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