*Due to acute spinal cord *Hemodynamic phenomenon- * Loss of vasomotor tone & Loss of sympathetic injury nervous system tone > inpaired cellular *Absence all voluntary metabolism
and reflex neurologic *Critical features-
Hypotension (due to massive vasodilation activity below level of Bradycardia- due to unopposed paraynmpathetic injury stimulation Decreased reflexes Poikilothermia; *Unable to regulate temperature- Loss of sensation Occurs Within 30 min cord injury level T 5 or above; last up to 6 Flaccid paralysis below weeks; also due to effect some drugs that effect injury vasomotor center of medulla as opioids, benzodiazedines Lasts days to months Management (*Determine underlying cause)
(Transient) Airway support Fluids as needed- Typically 0.9 NS , rate depends upon *Spinal shock & need Atropine for bradycardia neurogenic shock can in Vasopressors as phenylelphrine (Neo-synephrine) for same patient-BUT not BP support same disorder (some sources may group both together)