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MCQs Revision Biochemistry for

NEET Jan - 2018


DR. SMILY PRUTHI PAHWA
(MD-BIOCHEMISTRY)
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By Tulip academy of Medical sciences
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Q1 . Kinks in alpha structure are formed by
which amino acid :
1) Glycine
2) Lysine
3) Methionine
4) Glutamate
Q1 . Kinks in alpha structure are formed by
which amino acid :
1) Glycine
2) Lysine
3) Methionine
4) Glutamate
Q2. A patient’s lipid profile was done. His TGs
levels were found to be 200 mg/dl, Total
cholesterol was 300 mg/dl & HDL = 40 mg/dl.
Calculate LDL:
a. 220
b. 260
c. 60
d. 100
FRIEDWALD’S EQUATION
• Total cholesterol = HDL + LDL + VLDL
FRIEDWALD’S EQUATION
• Total cholesterol = HDL + LDL + VLDL
• LDL = Total cholesterol – HDL – TG/5
= 300 – 40 – (200/5)
= 300 -40 -40
= 300-80
= 220 mg/dl
• In mmol/L  divide by 2.2
• LDL chol = total chol – HDL chol – TG/2.2
Q2. A patient’s lipid profile was done. His TGs
levels were found to be 200 mg/dl, Total
cholesterol was 300 mg/dl & HDL = 40 mg/dl.
Calculate LDL:
a. 220
b. 260
c. 60
d. 100
Q3. Lipoprotein lipase is activated by:
a. Apo C-I
b. Apo C-II
c. Apo C-III
d. Apo A-I
Q3. Lipoprotein lipase is activated by:
a. Apo C-I
b. Apo C-II
c. Apo C-III
d. Apo A-I
Role of Apoproteins in Lipoproteins
1. Structural Role
2. Enzyme Cofactors :
Apo C-II  activate Lipoprotein Lipase
Apo C-III  inhibits Lipoprotein Lipase
Apo A-I  activate LCAT
Apo A-II  inhibits LCAT
3. Ligands for Receptors
Ligands for Receptors :
• Apo E  ligand for remnants
• Apo B100 & Apo E  LDL-Receptors
• Apo A1  HDL-Receptors
Q4. Tom cat urine smell is seen in:
a) Hawkinsinuria
b) Multiple Carboxylase defect
c) G6PD
d) Phenylketonuria
Q4. Tom cat urine smell is seen in:
a) Hawkinsinuria
b) Multiple Carboxylase defect
c) G6PD
d) Phenylketonuria
• Tom cat urine odour - Multiple Carboxylase
defect
• Mousy/Musty odour - Phenylketonuria
• Burnt sugar like odour - MSUD
• Swimming pool odour - Hawkinsinuria
Q5. Odd chain fatty acids can form glucose by
which pathway ?
a) Propionyl CoA
b) Glycerol
c) Acetyl CoA entering TCA cycle
d) Lactate
Q5. Odd chain fatty acids can form glucose by
which pathway ?
a) Propionyl CoA
b) Glycerol
c) Acetyl CoA entering TCA cycle
d) Lactate
Q6. Muscle cannot Q7. Muscle cannot
maintain blood glucose make use of glycogen
because of deficiency of : because of deficiency
a) Glucose-6- of :
phosphatase a) Glucose-6-
b) Glycogen phosphatase
phosphorylase b) Glycogen
c) Hexokinase phosphorylase
d) Phospho-gluco- c) Hexokinase
mutase
d) Phospho-gluco-
mutase
Q6. Muscle cannot Q7. Muscle cannot
maintain blood glucose make use of glycogen
because of deficiency of : because of deficiency
a) Glucose-6- of :
phosphatase a) Glucose-6-
b) Glycogen phosphatase
phosphorylase b) Glycogen
c) Hexokinase phosphorylase
d) Phospho-gluco- c) Hexokinase
mutase
d) Phospho-gluco-
mutase
Q8. Malate shuttle is important in :
a) Glycogenesis
b) Glycolysis
c) Gluconeogenesis
d) Glycogenolysis
Q8. Malate shuttle is important in :
a) Glycogenesis
b) Glycolysis
c) Gluconeogenesis
d) Glycogenolysis
Q9. During prolonged starvation, rate of
gluconeogenesis depends on :
a) Increased alanine levels in liver
b) Decreased cGMP levels in liver
c) ADP in liver
d) Decreased essential fatty acids in liver
Q9. During prolonged starvation, rate of
gluconeogenesis depends on :
a) Increased alanine levels in liver
b) Decreased cGMP levels in liver
c) ADP in liver
d) Decreased essential fatty acids in liver
Q10. Which of the following is a Pallindrome ?
a) GGCC
b) GTCC
c) TAAT
d) TTCG
Q10. Which of the following is a Pallindrome ?
a) GGCC
b) GTCC
c) TAAT
d) TTCG
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