Tulip Academy is a complete help package for the medical students keeps them close and latest in the medical field. Students can get every latest exam news, NEET PG registration, mock test, test series biochemistry video lectures and exam notifications exam board's release time to time.
Tulip Academy is a complete help package for the medical students keeps them close and latest in the medical field. Students can get every latest exam news, NEET PG registration, mock test, test series biochemistry video lectures and exam notifications exam board's release time to time.
Tulip Academy is a complete help package for the medical students keeps them close and latest in the medical field. Students can get every latest exam news, NEET PG registration, mock test, test series biochemistry video lectures and exam notifications exam board's release time to time.
DR. SMILY PRUTHI PAHWA (MD-BIOCHEMISTRY) See video on Youtube………. By Tulip academy of Medical sciences Or See video in mobile app – Search : tulip academy of medical sciences Q1 . Kinks in alpha structure are formed by which amino acid : 1) Glycine 2) Lysine 3) Methionine 4) Glutamate Q1 . Kinks in alpha structure are formed by which amino acid : 1) Glycine 2) Lysine 3) Methionine 4) Glutamate Q2. A patient’s lipid profile was done. His TGs levels were found to be 200 mg/dl, Total cholesterol was 300 mg/dl & HDL = 40 mg/dl. Calculate LDL: a. 220 b. 260 c. 60 d. 100 FRIEDWALD’S EQUATION • Total cholesterol = HDL + LDL + VLDL FRIEDWALD’S EQUATION • Total cholesterol = HDL + LDL + VLDL • LDL = Total cholesterol – HDL – TG/5 = 300 – 40 – (200/5) = 300 -40 -40 = 300-80 = 220 mg/dl • In mmol/L divide by 2.2 • LDL chol = total chol – HDL chol – TG/2.2 Q2. A patient’s lipid profile was done. His TGs levels were found to be 200 mg/dl, Total cholesterol was 300 mg/dl & HDL = 40 mg/dl. Calculate LDL: a. 220 b. 260 c. 60 d. 100 Q3. Lipoprotein lipase is activated by: a. Apo C-I b. Apo C-II c. Apo C-III d. Apo A-I Q3. Lipoprotein lipase is activated by: a. Apo C-I b. Apo C-II c. Apo C-III d. Apo A-I Role of Apoproteins in Lipoproteins 1. Structural Role 2. Enzyme Cofactors : Apo C-II activate Lipoprotein Lipase Apo C-III inhibits Lipoprotein Lipase Apo A-I activate LCAT Apo A-II inhibits LCAT 3. Ligands for Receptors Ligands for Receptors : • Apo E ligand for remnants • Apo B100 & Apo E LDL-Receptors • Apo A1 HDL-Receptors Q4. Tom cat urine smell is seen in: a) Hawkinsinuria b) Multiple Carboxylase defect c) G6PD d) Phenylketonuria Q4. Tom cat urine smell is seen in: a) Hawkinsinuria b) Multiple Carboxylase defect c) G6PD d) Phenylketonuria • Tom cat urine odour - Multiple Carboxylase defect • Mousy/Musty odour - Phenylketonuria • Burnt sugar like odour - MSUD • Swimming pool odour - Hawkinsinuria Q5. Odd chain fatty acids can form glucose by which pathway ? a) Propionyl CoA b) Glycerol c) Acetyl CoA entering TCA cycle d) Lactate Q5. Odd chain fatty acids can form glucose by which pathway ? a) Propionyl CoA b) Glycerol c) Acetyl CoA entering TCA cycle d) Lactate Q6. Muscle cannot Q7. Muscle cannot maintain blood glucose make use of glycogen because of deficiency of : because of deficiency a) Glucose-6- of : phosphatase a) Glucose-6- b) Glycogen phosphatase phosphorylase b) Glycogen c) Hexokinase phosphorylase d) Phospho-gluco- c) Hexokinase mutase d) Phospho-gluco- mutase Q6. Muscle cannot Q7. Muscle cannot maintain blood glucose make use of glycogen because of deficiency of : because of deficiency a) Glucose-6- of : phosphatase a) Glucose-6- b) Glycogen phosphatase phosphorylase b) Glycogen c) Hexokinase phosphorylase d) Phospho-gluco- c) Hexokinase mutase d) Phospho-gluco- mutase Q8. Malate shuttle is important in : a) Glycogenesis b) Glycolysis c) Gluconeogenesis d) Glycogenolysis Q8. Malate shuttle is important in : a) Glycogenesis b) Glycolysis c) Gluconeogenesis d) Glycogenolysis Q9. During prolonged starvation, rate of gluconeogenesis depends on : a) Increased alanine levels in liver b) Decreased cGMP levels in liver c) ADP in liver d) Decreased essential fatty acids in liver Q9. During prolonged starvation, rate of gluconeogenesis depends on : a) Increased alanine levels in liver b) Decreased cGMP levels in liver c) ADP in liver d) Decreased essential fatty acids in liver Q10. Which of the following is a Pallindrome ? a) GGCC b) GTCC c) TAAT d) TTCG Q10. Which of the following is a Pallindrome ? a) GGCC b) GTCC c) TAAT d) TTCG Tulip Academy Of Medical Sciences