Professional Documents
Culture Documents
© ACS
Shock
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© ACS
Objectives
Define shock
Recognize the shock state
Determine the cause
Apply treatment principles
Apply principles of fluid management
Monitor patient’s response
Employ options for vascular access
Recognize complications of vascular access
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Cardiac Physiology
CO = SV x HR
Venous Vascular
dp / dt
Capacitance Tone
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Pathophysiology
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Etiology of Shock
Hemorrhagic Nonhemorrhagic
Most common Tension
• Selected Neurogenic
Hemorrhagic Shock
Loss of circulating blood volume
Normal blood volume
• Adult 7% of ideal weight
• Child: 9 % of ideal weight
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Classification of Hemorrhage
Class I-IV
Not absolute
Only a clinical guide
Subsequent treatment determined by
patient response
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Class I Hemorrhage
750 mL BVL
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Class II Hemorrhage
750 – 1500 mL BVL
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Class IV Hemorrhage
≥ 2000 mL BVL
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Compounds
intravascular loss
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Recognize shock
Stop the bleeding !
Replenish intravascular volume
Restore organ perfusion
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CNS status
Skin perfusion
Urinary output
Pulse oximetry
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Resuscitation Evaluation
Hourly Urinary Output
Inadequate output suggests
inadequate resuscitation
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Therapeutic Decisions
Patient response determines
subsequent therapy
Hemodynamically “normal” vs
hemodynamically “stable”
Recognize need to resuscitate in
operating room
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Therapeutic Decisions
Rapid Response
< 20% blood loss
Continue to monitor
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Therapeutic Decisions
Transient Response
20% - 40% blood loss
Therapeutic Decisions
Minimal to No Response
> 40% blood loss
Immediate operation
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Volume Replacement
Warmed fluids
Crossmatched PRBCs
Type – specific
Type O, Rh negative
Autotransfusion
Coagulopathy
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Pitfalls
Equating Bp Athletes
with cardiac Pregnancy
output Medications
Hypothermia
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Avoiding Complications
Continued hemorrhage
Fluid overload
• CVP
• Pulmonary artery catheter
Other problems
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Continuous reevaluation
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Questions
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Summary
Restore organ perfusion
Early recognition of the shock state
Oxygenate and ventilate
Stop the bleeding
Restore volume
Continuous monitoring of response
Anticipate pitfalls